@NOAA ‘s desperate new paper: Is there no global warming ‘hiatus’ after all?
By Patrick J. Michaels, Richard S. Lindzen, and Paul C. Knappenberger | Watts Up With That? | June 4, 2015
A new paper published today by Science, from Thomas Karl and several co-authors[1], that removes the “hiatus” in global warming prompts many serious scientific questions.
The main claim[2] by the authors that they have uncovered a significant recent warming trend is dubious. The significance level they report on their findings (.10) is hardly normative, and the use of it should prompt members of the scientific community to question the reasoning behind the use of such a lax standard.
In addition, the authors’ treatment of buoy sea-surface temperature (SST) data was guaranteed to create a warming trend. The data were adjusted upward by 0.12°C to make them “homogeneous” with the longer-running temperature records taken from engine intake channels in marine vessels.
As has been acknowledged by numerous scientists, the engine intake data are clearly contaminated by heat conduction from the structure, and as such, never intended for scientific use. On the other hand, environmental monitoring is the specific purpose of the buoys. Adjusting good data upward to match bad data seems questionable, and the fact that the buoy network becomes increasingly dense in the last two decades means that this adjustment must put a warming trend in the data.
The extension of high-latitude arctic land data over the Arctic Ocean is also questionable. Much of the Arctic Ocean is ice-covered even in high summer, meaning the surface temperature must remain near freezing. Extending land data out into the ocean will obviously induce substantially exaggerated temperatures.
Additionally, there exist multiple measures of bulk lower atmosphere temperature independent from surface measurements which indicate the existence of a “hiatus”[3]. If the Karl et al., result were in fact robust, it could only mean that the disparity between surface and midtropospheric temperatures is even larger that previously noted.
Getting the vertical distribution of temperature wrong invalidates virtually every forecast of sensible weather made by a climate model, as much of that weather (including rainfall) is determined in large part by the vertical structure of the atmosphere.
Instead, it would seem more logical to seriously question the Karl et al. result in light of the fact that, compared to those bulk temperatures, it is an outlier, showing a recent warming trend that is not in line with these other global records.
And finally, even presuming all the adjustments applied by the authors ultimately prove to be accurate, the temperature trend reported during the “hiatus” period (1998-2014), remains significantly below (using Karl et al.’s measure of significance) the mean trend projected by the collection of climate models used in the most recent report from the United Nation’s Intergovernmental Panel on Climate Change (IPCC).
It is important to recognize that the central issue of human-caused climate change is not a question of whether it is warming or not, but rather a question of how much. And to this relevant question, the answer has been, and remains, that the warming is taking place at a much slower rate than is being projected.
The distribution of trends of the projected global average surface temperature for the period 1998-2014 from 108 climate model runs used in the latest report of the U.N.’s Intergovernmental Panel on Climate Change (IPCC)(blue bars). The models were run with historical climate forcings through 2005 and extended to 2014 with the RCP4.5 emissions scenario. The surface temperature trend over the same period, as reported by Karl et al. (2015, is included in red. It falls at the 2.4th percentile of the model distribution and indicates a value that is (statistically) significantly below the model mean projection.
[1] Karl, T. R., et al., Possible artifacts of data biases in the recent global surface warming hiatus. Scienceexpress, embargoed until 1400 EDT June 4, 2015.
[2] “It is also noteworthy that the new global trends are statistically significant and positive at the 0.10 significance level for 1998-2012…”
[3] Both the UAH and RSS satellite records are now in their 21st year without a significant trend, for example
[NOTE: An earlier version of this posting accidentally omitted the last two paragraphs before the graphic, they have been restored, and the error is mine – Anthony]
Editors of World’s Most Prestigious Medical Journals: “Much of the Scientific Literature, Perhaps HALF, May Simply Be Untrue”
Zero Hedge | June 4, 2015
Lancet and the New England Journal of Medicine are the two most prestigious medical journals in the world.
It is therefore striking that their chief editors have both publicly written that corruption is undermining science.
The editor in chief of Lancet, Richard Horton, wrote last month:
Much of the scientific literature, perhaps half, may simply be untrue. Afflicted by studies with small sample sizes, tiny effects, invalid exploratory analyses, and flagrant conflicts of interest, together with an obsession for pursuing fashionable trends of dubious importance, science has taken a turn towards darkness. As one participant put it, “poor methods get results”. The Academy of Medical Sciences, Medical Research Council, and Biotechnology and Biological Sciences Research Council have now put their reputational weight behind an investigation into these questionable research practices. The apparent endemicity [i.e. pervasiveness within the scientific culture] of bad research behaviour is alarming. In their quest for telling a compelling story, scientists too often sculpt data to fit their preferred theory of the world. Or they retrofit hypotheses to fit their data. Journal editors deserve their fair share of criticism too. We aid and abet the worst behaviours. Our acquiescence to the impact factor fuels an unhealthy competition to win a place in a select few journals. Our love of “significance” pollutes the literature with many a statistical fairy-tale. We reject important confirmations. Journals are not the only miscreants. Universities are in a perpetual struggle for money and talent, endpoints that foster reductive metrics, such as high-impact publication. National assessment procedures, such as the Research Excellence Framework, incentivise bad practices. And individual scientists, including their most senior leaders, do little to alter a research culture that occasionally veers close to misconduct.
***
Part of the problem is that no-one is incentivised to be right.
Similarly, the editor in chief of the New England Journal of Medicine, Dr. Marcia Angell, wrote in 2009:
It is simply no longer possible to believe much of the clinical research that is published, or to rely on the judgment of trusted physicians or authoritative medical guidelines. I take no pleasure in this conclusion, which I reached slowly and reluctantly over my two decades as an editor of The New England Journal of Medicine.
In her must-read essay, Dr. Angell skewers drug companies, university medical departments, and medical groups which set the criteria for diagnosis and treatment as being rotten with corruption and conflicts of interest.
And we’ve previously documented that the government sometimes uses raw power to cover up corruption in the medical and scientific fields.
Postscript: Corruption is not limited to the medical or scientific fields. Instead, corruption has become systemic throughout every profession … and is so pervasive that it is destroying the very fabric of America.
Why Didn’t my Doctor Tell Me Chemo Kills?
By F. William Engdahl – New Eastern Outlook – 03.06.2015
In my daily research I came across a report so alarming I put aside planned writing in order to bring this to the attention of those who care about life. It has to do with one of the main treatments for cancer used in modern medicine—chemotherapy. New research has documented that chemotherapy, far from ridding anyone of cancer actually feeds the growth and spread of cancer.
Sometimes it almost seems like the drugs industry works overtime to find new ways to hurt, cripple or even kill us. Scientist Peter Nelson of the Fred Hutchinson Cancer Research Center in Seattle in a write-up of a study of why cancer cells were so easy to kill in the lab but not inside our bodies, found that healthy cells damaged by chemotherapy secreted more of a protein called WNT16B which boosts cancer cell survival. “The increase in WNT16B was completely unexpected,” Nelson told AFP.
He added that,“WNT16B, when secreted, would interact with nearby tumor cells and cause them to grow, invade, and importantly, resist subsequent therapy.” That would explain why in cancer treatment, tumors often respond well initially, followed by rapid regrowth and then resistance to further chemotherapy.
The study was conducted by a team of scientists from different cancer research centers, universities as well as from the Lawrence Berkeley National Laboratories. It was published online in August 2012 in the journal Nature Medicine. Among their alarming conclusions was that, “The expression of WNT16B in the prostate tumor micro-environment attenuated the effects of cytotoxic chemotherapy in vivo, promoting tumor cell survival and disease progression.”
Mustard Gas Toxin
While their study results were alarming enough, more alarming is the complete absence of aggressive action to reexamine the entire field of cancer treatment. Chemo’s origins go back to World War I research into the human effects of exposure to mustard gas. Scientists discovered that the gas was a potent suppressor of blood cell production. During World War II researchers at Yale University School of Medicine in further study of nitrogen mustards, reasoned that an agent that damaged the rapidly growing white blood cells might have a similar effect on cancer. Left out was how to target only cancer cells and not healthy cells. In December 1942, the scientists gave several patients with advanced lymphomas (cancers of the lymphatic system and lymph nodes), a chemotherapeutic drug intravenously. Their improvement was called remarkable. The media concentrated on the remarkable improvement and did not bother to note that soon after treatment all were dead.
The chemotherapy revolution in cancer treatment was off and running. In the 1950’s the first chemo drug used commercially was mustine or Chlormethine. Mustine under the code-name HN2 is a chemical warfare agent. Adverse effect include: “Hypersensitivity reactions, including anaphylaxis… Nausea, vomiting and depression of formed elements in the circulating blood… Jaundice, alopecia, vertigo, tinnitus and diminished hearing.”
The research and development of mustine as a possible anti-cancer chemotherapy was led by Cornelius P. Rhoads, director of Memorial Sloan-Kettering Cancer Center, in wartime secrecy and published in 1946 after the war. Rhoads came to Memorial Sloan-Kettering from the Rockefeller Institute for Medical Research.
There during the 1930’s as part of the Rockefeller family’s obsession with eugenics, Rhoads spent six months in Puerto Rico, a stateless island often used covertly for human experimentation with new drugs.
In Puerto Rico in 1931 Rhoads wrote a letter to a friend in Boston where he stated, “Porto (sic) Ricans are beyond doubt the dirtiest, laziest, most degenerate and thievish race of men ever inhabiting this sphere. What the island needs is not public health work but a tidal wave or something to totally exterminate the population. I have done my best to further the process of extermination by killing off eight and transplanting cancer into several more.”
Rockefeller family spin doctor, Ivy Lee, launched a major damage control campaign over the scandal and managed to get Rhoads on the cover of Time as a “life-saving” hero.
Deadly consequences
The subsequent use of toxic chemotherapies on perhaps millions of cancer patients since then have hardly been encouraging. Published side effects of today’s chemo drugs, the largest share of which are made by Roche, are horrendous. They include “depression of the immune system, often by paralysing the bone marrow and leading to a decrease of white blood cells, red blood cells, and platelets. Anemia and thrombocytopenia… sepsis, or as localized outbreaks, such as Herpes simplex, shingles, or other members of the Herpesviridea.”
It gets worse. Because of the chemo resulting in immune system suppression, patients often get typhlitis, a life-threatening gastrointestinal complication of chemotherapy. Typhlitis is an intestinal infection which may manifest itself through symptoms including nausea, vomiting, diarrhea, a distended abdomen, fever, chills, or abdominal pain and tenderness. Typhlitis is a medical emergency. It has a very poor prognosis and is often fatal. It can cause infertility failure in men and ovarian failure in women. All that in addition to the well-known hair-loss, dry skin, damaged fingernails, a dry mouth (xerostomia), water retention, and sexual impotence.
In 2004 the Department of Radiation Oncology, Northern Sydney Cancer Centre, Australia, conducted a long-term investigation into the contribution of chemotherapy to 5-year survival in 22 major adult malignancies. The results were shocking: The overall contribution of curative and adjuvant cytotoxic chemotherapy to 5-year survival in adults was estimated to be 2.3% in Australia and 2.1% in the USA. The study came to the following conclusion: “..it is clear that cytotoxic chemotherapy only makes a minor contribution to cancer survival. To justify the continued funding and availability of drugs used in cytotoxic chemotherapy, a rigorous evaluation of the cost-effectiveness and impact on quality of life is urgently required.”
Chemo is massively toxic and kill any rapidly dividing cell, tumor or normal. The three best-selling cancer drugs worldwide in 2013 were all made by Roche—Rituxan, Herceptin and Avastin. For all three top chemo drugs sales totaled more than $21 billion.
And the Fred Hutchinson Cancer Research Center now documents how chemotherapy drugs act as carcinogens—they cause cancer which is why, depending on the patient’s immune strength and dosage, within five years a staggering number die after the chemo that was to have saved them.
I was in Beijing several years ago on a speaking tour and had severe back pain after the long flight. My Chinese publisher organized a treatment from a doctor trained in Traditional Chinese Medicine (TCM). She was also the grand-daughter of the chief TCM doctor of the Last Emperor who she said was still alive and chipper at 93 and passing his wisdom on to her and her brother. She told me at the Beijing medical university where she studied, the students were told, “One third of patients die of the psychological shock of being told by a doctor that they have cancer. Another third die from the negative effects of chemotherapy and radiation. The last third simply die.”
It would be useful for all doctors in active practice perhaps to rethink the principal ethical mandate of all physicians since the time of Hippocrates– “nil nocere” – do no harm. The evidence is overwhelming now that chemotherapy only does harm. Would the oncologists promoting chemo to their patients ever take the same were the roles reversed?
Fallacies in Modern Medicine: Statins and the Cholesterol-Heart Hypothesis
By Donald W. Miller, Jr., MD | June 4, 2015
This commentary was published in the peer-reviewed Journal of American Physicians and Surgeons Volume 20, Number 2, Pages 54-56, Summer 2015.
Modern medicine has developed striking ways to treat coronary heart disease, which feature coronary stents implanted percutaneously and coronary artery bypass grafts performed surgically with the aid of a heart-lung machine. And then there are statins to lower cholesterol.
A 70-year-old man sees a physician for a checkup. He has no history of heart disease and no risk factors for it. He does not smoke, has no family history of diabetes or heart disease, and is physically active and not overweight. His blood pressure is 130/70. A lipid panel, however, shows that his calculated low-density lipoprotein cholesterol (LDL-C) is 195 mg/dL. Following the most recent 2013 guidelines framed by an American College of Cardiology (ACC) and American Heart Association (AHA) task force, the physician prescribes a statin for this person, rosuvastatin (Crestor) 20 mg/day, for primary prevention of atherosclerotic cardiovascular disease (ASCVD). [1]
Cardiologists declare that “cholesterol-containing lipoproteins are central to the pathogenesis of atherosclerosis.” [2] Statins, first approved for clinical use in 1987, are very effective in lowering cholesterol. High intensity statin therapy, rosuvastatin 20mg/day and atorvastatin (Lipitor) 40-80 mg, reduces LDL-C by 50 percent or greater. Moderate intensity therapy, rosuvastatin 10 mg, atorvastatin 10 mg, simvastatin (Zocor) 20-40 mg, and pravastatin (Pravachol) 40 mg/day, achieves a 30 to 50 percent reduction of LDL-C. [3]
Some 43 million Americans take statins. [4] In 2010, 11.6 percent of the population took them, 37 million, which includes 19.2 percent of people age 45-64; 39.6 percent, age 65-74; and 44.3 percent of people age 75 and older. [3] Following the 2013 ACC/AHA guidelines, an additional 10.2 million Americans without cardiovascular disease, like the patient above, have now become candidates for statin therapy. [5] One study concludes that 97 percent of black and white Americans aged 66 to 75, including all men in that age group should take statins. [6]
It is a multi-billion dollar business. Pfizer’s Lipitor went on sale in 1997 and became the best-selling drug in the history of prescription pharmaceuticals before its patent expired in 2011. Sales surpassed $125 billion. AstraZeneca’s Crestor was the top-selling statin in 2013, generating $5.2 billion in revenue that year.
Pfizer, in an advertisement, proclaims, “Lipitor reduces risk of heart attack by 36%,” based on the findings of a large randomized trial where 10,305 individuals were assigned to take Lipitor or a placebo (ASCOT-LLA). [7] The trial showed that 1.9 percent of people taking Lipitor suffered a heart attack while 3.0 percent of the placebo group had one. Considered in terms of “relative risk” reduction, the percentage Pfizer cites in the ad is correct. (It is calculated by subtracting 1.9 from 3 and dividing the difference, 1.1, by 3, which equals 36 percent.) But more realistically, the trial showed that Lipitor only reduced the “absolute risk” of having a heart attack by a tiny 1.1 percent (1.9 percent in the statin group compared with 3 percent in the placebo group). [7] Statin-trial investigators tout relative risk reduction (typically 20-40 percent in these trials) rather than the meager, real-world reduction in risk (1-2 percent taking statins).
Investigators cite relative risk to inflate claims of statins’ effectiveness. However, they report deleterious effects in terms of absolute risk, minimizing their magnitude. For example, if 6 percent of the statin group were to get diabetes during a trial compared to 2 percent with the placebo group, they will say that taking statins increases the risk of acquiring diabetes by 4 percent, not that there is 66 percent increased (relative) risk of suffering this adverse event.
Government and the pharmaceutical industry fund these multimillion dollar studies expecting correct results, so statin-trial researchers employ this particular kind of statistical deception to create the appearance that statins are effective and safe. [8] As one medical school professor puts it, “Anyone who questions cholesterol usually finds his funding cut off.” [9]
Eukaryotic animal cells make cholesterol through the “mevalonate pathway.” This pathway also produces, among other things, coenzyme Q10 (CoQ10), heme-A, and dolichol. CoQ10 is particularly important as it functions both as an antioxidant and, with heme-A, in aerobic cellular respiration—in the electron transport chain that generates adenosine triphosphate, the fuel that powers all living things. (Dolichol is required for synthesis of glycoproteins.) Statins inactivate hydroxymethylglutaryl‐coenzyme A (HMG-CoA) reductase, the enzyme cells use to synthesize mevalonate from HMG-CoA. This shuts down the mevalonate pathway. As a result, HMG-CoA reductase inhibitors (statins) block not only the synthesis of cholesterol but also CoQ10 and the other physiologically essential biomolecules that this pathway produces.
Lovastatin (Mevacor), the first statin, is a naturally occurring molecule isolated from a fungus named Aspergillus terreus. Newer statins are synthetic variations of these mycotoxins that fungi produce. Fungi make statins, as a “secondary metabolite,” to kill predatory microbes. They also kill human cells. In a review of How Statin Drugs Really Lower Cholesterol and Kill You One Cell at a Time by James and Hannah Yoseph, Peter Langsjoen writes:
Many practicing physicians have a healthy understanding of the current level of corruption and collusion among big pharmaceutical companies, governmental agencies such as the NIH and FDA, and major medical associations such as the American Heart Association, but the reader of this book will come away with the disturbing conclusion that it is even worse than imagined. Statins may be the perfect and most insidious human toxin in that adverse effects are often delayed by years and come about gradually. Further, statins frequently impair mental function to such a degree that by the time patients are in real trouble, they may lack the mental facilities to recognize the cause. [10]
This toxin targets brain cells and skeletal muscle. The brain makes up 2 percent of body weight but contains 25 percent of the body’s cholesterol. Its dry weight is 50 percent cholesterol. LDL-C delivers cholesterol to the body’s cells, except for the brain since this cholesterol-carrying lipoprotein does not cross the blood-brain barrier. Statins do. Brain cells, neurons and glial cells, manufacture their own cholesterol and the mevalonate pathway’s other products. [11] A broad spectrum of adverse cognitive reactions occur from taking statins. They include confusion, forgetfulness, disorientation, memory impairment, transient global amnesia, and dementia. [12]
Myopathy is the most common adverse effect of statin treatment, manifested by muscle aches and pains, weakness, instability, and easy fatigue. [8,13] The most severe manifestation of statin-induced muscle damage is rhabdomyolysis, which carries a 10 percent mortality rate. Fragments of ruptured muscle block renal tubules and cause kidney failure. [12] In one randomized trial of 1,016 healthy men and women given statins or a placebo, 40 percent of the women taking statins suffered exertional fatigue or decreased energy. [14]
Several randomized controlled trials have reported a statistically significant increase in cancer taking statins. [8,15] In most of these trials, a small reduction in cardiovascular deaths in the statin group is counterbalanced by an increase in deaths from other causes, notably cancer, with the result that there is in no significant difference in all-cause mortality between people taking a placebo and those prescribed statins. [16]
Statins can also cause diabetes, emotional disorders (depression, aggressiveness, suicidal ideation), hepatitis, cataracts, and strokes. [12,13,17] In January 2014 the FDA issued new safety information on statins, pointing out that “a small increased risk of raised blood sugar levels and the development of type 2 diabetes have been reported with the use of statins;” and it required drug companies add this information in the package insert with the drug. [18] Since then (as of August 2014), attorneys have filed more than 1,000 lawsuits against Pfizer, representing 4,000 women who say that taking Lipitor gave them diabetes.
Statin trials typically run for only 2 to 5 years. Investigators terminated the influential JUPITER trial endorsing statins for primary prevention of ASCVD after (a median) 1.9 years, far too short a time to reveal one of the worst “side effects” of long-term statin treatment: accelerated senescence. [19] Statins speed up the transition from midlife vigor to debilitated old age. [12]
Heart surgeon Michael DeBakey and his team, 52 years ago, found no correlation between blood cholesterol levels and severity of atherosclerosis in 1,700 patients undergoing surgical treatment of ASCVD. [20] I have observed the same thing with my heart surgery patients (unpublished observations). Evidence for the cholesterol-heart hypothesis, i.e., the lipid hypothesis, wilts upon close scrutiny, as is also the case with the diet-heart hypothesis, which indicts saturated fat along with cholesterol for causing atherosclerosis. Approached with an open mind and without confirmatory bias (ignoring evidence that disagrees with one’s beliefs), substantial evidence now proves beyond a reasonable doubt that these hypotheses are wrong. [21-25]
If not cholesterol, what causes atherosclerosis? My colleague, the late Russell Ross, professor of pathology at the University of Washington discovered the cause: Atherosclerosis is an inflammatory disease. [26] Initiated by endothelial dysfunction, with or without injury, and mediated by macrophages and T lymphocytes, the ensuing inflammatory response promotes proliferation and migration of smooth muscle cells. Russell demonstrated that atherosclerosis is a chronic inflammatory and fibroproliferative process that is fundamentally no different than that seen in cirrhosis, rheumatoid arthritis, and chronic pancreatitis.
The small benefit statins offer in dealing with ASCVD comes from their non-lipid-lowering anti-inflammatory effects, especially with their ability to suppress nuclear factor-kappa B (NF-kB), a transcription factor concerned with intensifying the inflammatory response. [27] But even if they had no harmful side effects, the “number needed to treat” (NNT) for statins weighs against their use. If a statin reduces the (absolute) risk of having a heart attack by just 2 percent, its NNT is 50. For every 50 people taking a statin, 1 person will benefit while 49 other people (98 percent) will not gain any benefit from taking the drug and will expose themselves to the potentially serious broad spectrum of adverse events that statins cause (carrying a risk considerably greater than 2 percent). Statins do more harm than good. (Nutraceuticals curcumin and resveratrol also quell inflammation, like statins, by suppressing NF-kB—with no side effects).
A catalog of factors that play a causal role in inflammatory ASCVD would include: 1) eating trans fats and too many carbohydrates and omega-6 vegetable oils (and not enough saturated fats); 2) deficiencies in various vitamins (vitamins A, C, D, E, K2, B6, B9-folic acid, and B12); 3) mineral deficiencies (magnesium, selenium, copper) and excess (iron); 4) lipid oxidation products; 5) possibly bacterial infection (Chlamydia pneumoniae); 6) diabetes; 7) abdominal obesity; 8) hypertension; 9) smoking; and 10) stress.
Cholesterol combats inflammation in addition to its other roles, which include maintaining cell membrane integrity (cell membranes are 50 percent cholesterol), facilitating cell signaling, and serving as the structural foundation for bile salts, various hormones, and vitamin D. Dealing with inflammation cholesterol acts as the body’s fire brigade, putting out inflammatory fires and helping repair damage. (Blaming cholesterol for atherosclerosis is like blaming firemen for the fire they have come to put out.)
Cementing this molecule’s physiologic importance, there are now more than 100 peer reviewed studies showing that low cholesterol levels lead to early death. [28] One of them is a study by Schatz and colleagues exploring the relationship between cholesterol levels and death rates over a 20-year period in 3,572 men aged 71-93 years. Those with the lowest cholesterol had a 35 percent increase in mortality compared with the highest cholesterol. [29] Another one, following 490 people aged 75 years for over 6 years, found that those with cholesterol levels below 193 mg/dL had a 52 percent increase in death rates compared to those with cholesterol levels above 232 mg/dL. Death rates rose by 18 percent for every 38mg/dL decrease in cholesterol levels. [30]
It is becoming increasingly clear that the cholesterol-heart hypothesis is a fallacy of modern medicine. In the future medical historians may liken the prescribing of statins to lower blood cholesterol with the old medical practice of bloodletting. Taking that vital substance out of the body is comparable to today’s practice of blocking production of cholesterol, an equally vital component, with drugs.
References
- Stone NJ, Robinson JG, Lichtenstein AH, et al. 2013 ACC/AHA guideline on the treatment of blood cholesterol to reduce atherosclerotic cardiovascular risk in adults: a report of the American College of Cardiology/American Heart Association Task force on Practice Guidelines. J Am Coll Cardiol 2014;63:2889-2934.
- Kohli P, Whelton SP, Hsu S, et al. Clinician’s guide to the updated ABCs of cardiovascular disease prevention. J Am Heart Assoc 2014;3:e001098 Available at: http://jaha.ahajournals.org/content/3/5/e001098.full Accessed March 17, 2015.
- Newsom, LD. Primary prevention of atherosclerotic cardiovascular disease:Controversies and clinical considerations. Ann Pharmacother 2015;49(April): 484-493.
- Pencina MJ, Navar-Boggan AM, D’Agostino RB, et al. Application of new cholesterol guidelines to a population-based sample. N Engl J Med 2014;370:1422-1431.
- Centers for Disease Control and Prevention. Health United States, 2013: With special feature on prescription drugs. Available at: http://www.cdc.gov/nchs/data/hus/hus13.pdf Accessed March 20, 2015.
- Miedema AMD, Lopez FL, Blaha MJ. Eligibility for statin therapy according to new cholesterol guidelines and prevalent use of medication to lower lipid levels in an older US cohort: The atherosclerosis risk in communities study cohort. JAMA Intern Med 2015;175(1):138-140.
- Sever PS, Dahlof B, Poulter NR, et al. Prevention of coronary and stroke events with atorvastatin in hypertensive patients who have average or lower-than-average cholesterol concentrations, in the Anglo-Scandinavian Cardiac Outcomes Trial-Lipid Lowering Arm (ASCOT-LLA): a multicenter randomized controlled trial. Lancet 2003;361:1149-1158.
- Diamond DM, Ravnskov U. How statistical deception created the appearance that statins are safe and effective in primary and secondary prevention of cardiovascular disease. Expert Rev Clin Pharmacol 2015;8(2):189-199.
- Rosch P. Quote in: Cholesterol skeptics and the bad news about statins. Center for Medical Consumers, Cholesterol Skeptics: Conference Report. Available at: http://medicalconsumers.org/2003/06/01/cholesterol-skeptics-conference-report/ Accessed March 21, 2015.
- Langsjoen P. Review of How Statin Drugs Really Lower Cholesterol and Kill You One Cell at a Time by James and Hannah Yoseph. J Am Phys Surg 2013;18:30.7
- Mauch DH, Nagler K, Schumacher S. CNS synaptogenesis promoted by glia-derived cholesterol. Science 2001:294(5545):1354-1457.
- Graveline D. Adverse Effects of statin drugs: a physician patient’s perspective. J Am Phys Surg 2015;20:7-11.
- Golomb BA, Evans MA. Statin adverse effects: a review of the literature and evidence for a mitochondrial mechanism. Am J Cardiovasc Drugs 2008;8(63):373-418.
- Golomb BA, Evans MA, Dimsdale JE, et al. Effects of statins on energy and fatigue with exertion: results from a randomized controlled trial. Arch Intern Med 2012;172:1180-1182.
- Ravnskov U, Rosch PJ, McCully KS. The statin-low cholesterol-cancer conundrum. QJM 2012;105:383-388.
- Colpo A. The Great Cholesterol Con: Why everything you’ve been told about cholesterol, diet and heart disease is wrong! Lulu.com; 2006.
- Culver AL, Ockene IS, Balasubramanian R, et al. Statin use and risk of diabetes mellitus in postmenopausal women in women’s health initiative. Arch Intern Med 2012;172(2):144-152.
- FDA expands advice on statin risk. FDA Consumer Health Information/U.S. Food and Drug Administration. January 2014. Available at: http://www.fda.gov/downloads/ForConsumers/ConsumerUpdates/UCM293705.pdf Accessed March 22, 2015.
- Ridker PM, Danielson E, Fonseca FA, et al. Rosuvastatin to prevent vascular events in men and women with elevated C-reactive protein. N Engl J Med 2008;359:2195-2207.
- Garret HE, Horning EC, Creech RG, DeBakey M. Serum cholesterol values in patients treated surgically for atherosclerosis. JAMA 1964;189:655-659.
- Iso H, Jacobs Jr DR, Wentworth D, et al. Serum cholesterol level and six-year mortality from stroke in 350,977 men screened for the multiple risk factor intervention trial. N Engl J Med 1989;320:904-910.
- Ravnskov U. The Cholesterol Myths: Exposing the Fallacy that Saturated Fat and Cholesterol Cause Heart Disease. Washington, D.C.: New Trends Publishing; 2000.
- Ravnskov U. A hypothesis out-of-date: The diet-heart idea. J Clin Epidemiol 2002;55:1057-1063,
- Taubes G. Good Calories, Bad Calories: Fats, Carbs, and the Controversial Sciance of Diet and Health. New York: Anchor Books; 2008.
- Evans D. Cholesterol and Saturated Fats Prevent Heart Disease: Evidence from 101 Scientific Studies. Guilford, Surrey, UK; Grosvenor House Publishing: 2012.
- Ross R. Atherosclerosis—an inflammatory disease N Engl J Med 1999; 340:115-126.
- Hölschermann H, Schuster D, Parviz B, et al. Statins prevent NF-kB transactivation independently of the IKK-pathway in human endothelial cells. Atherosclerosis 2006;185:240-245.
- Evans D. Low Cholesterol Leads to an Early Death: Evidence from 101 Scientific Papers. Guilford, Surrey, UK; Grosvenor House Publishing: 2012.
- Schatz IJ, Masaki K, Yano K, et al. Cholesterol and all-cause mortality in elderly people from the Honolulu Heart Program: a cohort study. Lancet 2001;358(9279):351-355.
- Tuikkala P, Hartikainen S, Korhonen MF, et al. Serum total cholesterol levels and all-cause mortality in a home-dwelling elderly population: a six-year follow-up. Scand J Prim Health Care 2010;28(2):121-127.
Donald Miller (send him mail) is a retired cardiac surgeon and Emeritus Professor of Surgery at the University of Washington School of Medicine in Seattle. He is a member of Doctors for Disaster Preparedness
Why The Netherlands Just Banned Non-Commercial Use Of Monsanto’s Glyphosate-Based Herbicides
By Arjun Walia | Collective Evolution | May 30, 2015
The Netherlands has just become the latest country, following Russia, Mexico, and many others, to say no to Monsanto. The sale and use of glyphosate-based herbicides (the most commonly used herbicides in the world) has just been banned for non-commercial use in the country, effective later this year. This means that people will no longer be able to spray RoundUp on their lawns and gardens and will instead have to find another (hopefully more natural) means of pest control.
This is definitely a step in the right direction.
The move comes as no surprise, considering that the number of countries around the world who are choosing to ban this product is growing at an exponential rate. Bans and restrictions are being implemented due to the fact that glyphosate (the main ingredient in RoundUp) has been directly linked to several major health issues, including: birth defects, nervous system damage, Alzheimers, Parkinson’s, various forms of cancer, and kidney failure. (Sri Lanka recently cited deadly kidney disease as their reason for banning his product. You can read more about that and access the research here.) Indeed, The World Health Organization recently acknowledged the fact that glyphosate can cause cancer, and you can read more about that here.
Not only that, there are multiple environmental concerns associated with the use of this chemical.
What’s even more disturbing is the fact that studies have shown that RoundUp herbicide is over one hundred times more toxic than regulators claim. For example, a new study published in the journal Biomedical Research International shows that Roundup herbicide is 125 times more toxic than its active ingredient glyphosate studied in isolation. You can read more about that here. The eye opening abstract reads as follows:
“Pesticides are used throughout the world as mixtures called formulations. They contain adjuvants, which are often kept confidential and are called inerts by the manufacturing companies, plus a declared active principle, which is usually tested alone. We tested the toxicity of 9 pesticides, comparing active principles and their formulations, on three human cell lines. Glyphosate, isoproturon, fluroxypyr, pirimicarb, imidacloprid, acetamiprid, tebuconazole, epoxiconazole, and prochloraz constitute, respectively, the active principles of 3 major herbicides, 3 insecticides, and 3 fungicides. Despite its relatively benign reputation, Roundup was among the most toxic herbicides and insecticides tested. Most importantly, 8 formulations out of 9 were up to one thousand times more toxic than their active principles. Our results challenge the relevance of the acceptable daily intake for pesticides because this norm is calculated from the toxicity of the active principle alone. Chronic tests on pesticides may not reflect relevant environmental exposures if only one ingredient of these mixtures is tested alone.” (source)
Equally disturbing is the fact that RoundUp has been found in a very high percentage of air and rainfall test samples. You can read more about that here.
Significant concentrations of it have also been found in the urine of people across Europe, you can read more about that here.
One recent study published in the Journal of Environmental & Analytical Toxicology has now proven that animals and humans who consume GMO foods – those that are loaded with glyphosate chemicals, the main ingredient in Monsanto’s RoundUp – have extremely high levels of glyphosate in their urine.
It’s also noteworthy to mention that there are Wikileaks documents showing how the United States planned to “retaliate and cause pain” on countries who were refusing GMOs. You can read more about that story and view those documents here.
It’s troubling to think that so many children are within proximity of and playing on lawns that have been sprayed with this stuff. Cancer is not a mystery, it is not a stroke of bad luck, it’s time for the world to wake up and realize what research has been confirming for years.
More Information on Pesticides & Herbicides Here:
**There are also multiple articles linked within the article above that provide more information**
Scientists Link Autism To These Toxic Chemicals During Fetal Development
Another Groundbreaking Study Emerges Linking Agricultural Pesticides To Autism
Scientists Can Predict Your Pesticide Exposure Based On How Much You Eat
This Is What Happens To Your Body When You Switch To Organic Food
What Parents Need To Know About Monsanto: “By 2025 One In Two Children Will Be Autistic”
Monsanto’s Glyphosate Linked To Birth Defects
Groundbreaking Study Links Monsanto’s Glyphosate To Cancer
New Study Links Gmos To Cancer, Liver/Kidney Damage & Severe Hormonal Disruption
Multiple Toxins From GMOs Detected In Maternal And Fetal Blood
Sources Used:
Climate Stupidity and Human Survival
By Denis Rancourt | Dissident Voice | May 26, 2015
The human animal has an instinct to identify potential dangers and to warn others. It is a built-in survival mechanism of any animal that lives in a group. And it is a strong and constant activity, re-enforced by environmental stressors.
This plays out on several time scales, from the immediate in the case of a potential physical assault, to the weekly in checking the weather forecast, to seasonal in preparing for winter, to life-long in planning for inevitable aging, to leaving good things for our grandchildren…
It is in our fiber to look ahead and to plan ahead, especially in the face of foreseeable or detected dangers.
The whole process can spin out of control when the danger is difficult to perceive, yet could be lethal. Think of baboons who are on the lookout for a stalking lion. The slightest shadow movement can make them scream and run for the trees. It’s a tense and highly volatile situation.
At this stage in our evolution we are faced with a pathological extension of our collective survival reflex, which is entirely fabricated by our high priests (government funded scientists and talking heads).
If these high priests were not here to tell us that the atmospheric concentration of the minor constituent CO2 is increasing, and that “global mean surface temperature” has increased by some 0.5 C in the last 100 years, then we would never know about these imperceptible causes of our certain eventual collective death as a species.
The priests explain that our certain extinction will occur from a rising sea level and changing regional climates. That these changes will cause mass migrations, ecosystem collapses, agricultural failures, famines, and disease. They also inform us that those who will suffer most are the most vulnerable inhabitants of the planet, as though this were a new feature of the effects of natural disasters.
Therefore, they urge, we must tax carbon emissions, apply cap and trade, and create a global carbon economy to limit CO2 in the atmosphere. And who better to coordinate it all than the World Bank, IMF, and such, given their stellar records in managing equitable development on this little rock. (Or is that economic enforcement of US regime supremacy?)
Forgive me for saying, but this all sounds rather nutso to me.
Nothing could be more like a religion than this crazy movement. We are expected to accept that an essential and growth-limiting plant nutrient (CO21 ) is a toxic pollutant, that the world will be destroyed because of our collective and intrinsic wickedness of emitting CO2, via floods no less.
Take a deep breath (exhale if you dare) and allow me to state a few facts that might help put things into perspective.
The planet has been teeming with life for billions of years.
During that time, the global mean temperature has almost always been some 10 C higher than in the present geologic anomaly,2 in a manner uncorrelated with CO2 concentration.3 That is the history of this same planet that we live on. During that time, the CO2 concentration has typically been 10 times higher than today’s value, and it has rarely been as low as modern values, nor has it ever been lower than modern values.
Atmospheric CO2 and average global temperature
There is no reason to believe that humans would not fare well on an Earth that is 10° C warmer, never mind 1° or 2° C. Land value would increase in the polar regions, and there would be intense reforestation and forest densification of the equatorial regions, with little possibility for controlling growth where it is hot and humid.From what we know of our planet and the history of its biosphere, warming is not going to kill us off any time soon. None of the known mass extinctions (a relative term) in Earth’s history can be reliably attributed to “sustained warming,” whereas ice ages that have occurred recently (during human presence on the planet, in the last 1 M years) are expected to correspond to periods of decreased planetary life density, but saw mammals and human populations completely adapt. Basically, neither warming or cooling can kill us by any know mechanism ever observed. If anything, the opposite of “killer warmth” is observed on today’s Earth, where both human populations and living biomass are concentrated near equatorial latitudes:
World population density map
Therefore, we still have much time left to achieve human extinction by much more direct means than warming (or cooling) of any kind. We also have a lot of time and occasions to practice accommodating mass migrations caused by our wars and economic violence, in order to prepare for the “climate migrations”. Somehow there seems to be more public-opinion, political, and lobbying effort in implementing and developing the instruments of a global carbon economy than in developing the instruments to prevent wars of aggression, to cope with the consequences of natural disasters, to stop displacing and dispossessing local inhabitants, to enforce the Geneva Conventions, to stop the wholesale destruction of entire nations (Libya, Iraq, Afghanistan, Syria, Iran…?), to respect international law, … not to mention reparations to the survivors of recent slavery, genocide, dispossession, and crass exploitation.
Rather than widespread vehement and actuated insistence on democratic control of local resources and institutions, based on individual realities on the ground, instead we have a sizable yet ineffective population of vocal do-gooders enthralled in spasmodic incantations against atmospheric CO2 emissions, as part of a Gaia-inspired religion perverted by the Christian concept of original sin; all of which de facto supports the carbon globalization schemes engineered by the US regime to attempt to constrain their emerging competitors and extort a development tax.
When has “globalization” ever been about justice, or about anything other than economic predation? When have good-will global efforts ever had any significant positive impact?4
Why pollute local struggles and liberation of the individual with tenuous claims about imperceptible dangers? And why put so much energy into insisting that the danger from CO2 is real? This seems like a classic example of seeking an overarching religious belief “solution” to real local problems that one cannot or will not confront.
“Climate justice” needs to be “justice”. The comfortable-middle-class fetish for carbon co-opts the analysis, defuses the thrust for defending identity-tied interests, and sends legitimate demands straight into the atmosphere. Or, at best, it is simply irrelevant to real struggles.
In the main population, if all the fanatics that are screaming that the sky is falling would scream to stop the war machine that occupies every corner of the globe, then we could start moving away from the real manufactured disasters that wash over the planet continuously, which don’t require satellite spectrometers to detect.
Instead of asking whether we can detect warming, whether intense weather events are actually more frequent, whether species extinction rates can reliably be measured, and so on, why not address the obvious: Humans are exploiting and terrorizing other humans, human conditions are constantly being attacked, and natural habitat is being destroyed for corporate benefit by eliminating local sovereignty.
Why turn to sanitized and intractable up-in-the-air questions when injustice and actual destruction is all around us? And why, oh why, pretend that humans can manage global carbon fluxes, manage the radiation balance of the planet, and control climate?
It’s a planet!
If we are going to have a global religion, why not believe that justice leads to both short-term and long-term safety? Not justice that is planned and given to us, but justice that we acquire through struggle and liberation.
On the other hand if you must be irrelevant and must have your carbon fetish, then at least put it into a planetary perspective5 :
- The present (2010) rate of fossil fuel burning (0.8 x 10^13 kg-C/y) is 8% of global primary production (GPP)
- The latter plant growth (GPP) uses only 0.07% of solar light striking the planet
- Thus, fossil fuel burning represents 8% of 0.07% = 0.006% of solar energy rate of input (the sun is a sun and the Earth is a planet…)
- The CO2 production from the burning of fossil fuel is approximately equal to that from human and domestic animal breathing
- The combined biomass of humans and domestic animals is 0.04% of Earth’s living biomass
- Ants have transformed the planet’s surface and its ecology far more than have humans
- The total amount of fossil fuel burned to date (historically to 2010) by humans is 3.7 x 10^14 kg-C, less than half of the carbon contained in the atmosphere as a minor constituent gas
- Dissolved CO2 in the oceans is 50 times more than the total amount in the atmosphere
- Living and dead biomass-carbon (in soils, sediments, plant-cover, etc.) is probably much greater than carbon as CO2 in air and water
- Thus, the total post-industrial fossil fuel burned to date represents less than 1% of the planet’s global bio-available and exchangeable carbon, not to mention geological sources
- As such, atmospheric CO2 is readily exchanged with and buffered by compartments of labile carbon that are much larger than the atmosphere, via flux mechanisms that science is barely beginning to understand (e.g.,1 )
- Keenan et al., “Increase in forest water-use efficiency as atmospheric carbon dioxide concentrations rise,” Nature 499, 324-327 (18 July 2013); Donohue et al., “Impact of CO2 fertilization on maximum foliage cover across the globe’s warm, arid environments,” Geophysical Research Letters 40(12), 3031-3035 (28 June 2013).
- Wikipedia article, “Geologic temperature record,”accessed May, 2015, see “Overall view”.
- Rothman, D.H., “Atmospheric carbon dioxide levels for the last 500 million years,” Proceedings of the National Academy of Science, 99(7), 4167-4171, (2 April 2002).
- Rancourt, D.G., “Some big lies of science,” Activist Teacher (8 June 2010).
- Rancourt, D.G., “Is the burning of fossil fuel a significant planetary activity?,” Activist Teacher (21 August 2010); Rancourt, D.G., “CO2 emission from fossil fuel burning is not more than from breathing,” Activist Teacher (22 August 2010)
*****
This article accompanies a 2-hour public lecture I gave at the University of Ottawa on March 27, 2015, entitled “The science and geopolitics of climate change”: VIDEO-LINK-Part-1, VIDEO-LINK-Part-2. The physics calculations of Earth’s radiation balance described in Part-1 of the talk are from this paper: Rancourt, D.G., “Radiation physics constraints on global warming: CO2 increase has little effect,” archive.org (3 December 2011). Links to my articles and interviews about climate are here.
The Ethics of Climate Change
Calls for massive reductions in global greenhouse gas emissions ignore the impacts on the poor
By Bob Lyman | Watts Up With That? | May 23, 2015
People who believe in the theory of catastrophic human-induced global warming claim that they want to “save the planet” and that this is the moral thing to do. They insist, however, that saving the planet requires stringent reductions in people’s use of fossil fuel energy to reduce greenhouse gas emissions. They never talk about what that means to the poor. I think that, before people decide on the ethics of the debate, they need to consider what the impact would be of sharply reducing energy consumption on the wellbeing of world’s population, and especially on the poor.
In 2014, the International Energy Agency (IEA) issued a Special Report entitled “Modern Energy for All”. In it, the IEA stated that modern energy services are:
…crucial to human wellbeing” and to a country’s economic development.
Access to modern energy is essential for the provision of clean water, sanitation and healthcare and for the provision of reliable and efficient lighting heating, cooking, mechanical power, transport and telecommunications services.”
Today billions of people lack access to the most basic energy services. Nearly 1.3 billion people are without access to electricity and 2.7 billion people rely on traditional use of biomass (wood, charcoal and animal dung) for cooking, which causes harmful indoor air pollution.
Pause to think about that for a few minutes. Hundreds of millions of people are without the modern energy services that were available to our ancestors who lived in the nineteenth century. They get up with the dawn and go to bed close to nightfall because they have no electrical lighting. They have to go a river or well (if they are lucky) for water to drink or wash in. They have no way to power an appliance, including a refrigerator, so all food has to be eaten quickly or it may go bad. They have to walk long distances everyday to search for firewood or dried animal dung. There is no light to extend the day to provide time for reading or entertainment. They have no telephones. They have no way to pump water for irrigating crops. They have no motorized transportation, so they cannot go very far. Almost all their time is spent simply doing the simple tasks that in Canada and other advanced countries are done by machines. Worse, every day they breathe in the fumes from the dirty cooking fires, developing lung disorders. In fact, according to the IEA, every year 4.3 million premature deaths can be attributed to household air pollution resulting from the use of traditional biomass fuels for cooking.
The international community has long been aware of the close correlation between income levels and access to modern energy; not surprisingly, countries with a large proportion of the population living on an income of $2 per day tend to have low electrification rates and few motorized vehicles. The problem is spread throughout the developing world, but it is particularly severe in sub-Saharan Africa and developing Asia, which together account for 95% of people in abject energy poverty.
The latent demand for electricity is immense. An estimated 400 million people in India still lack access to electricity. A recent study looked at the expansion of electricity that would be needed on an economy-wide basis in sub-Saharan Africa to comprehensively address energy access. To reach moderate access, where electricity generation capacity is around 200-400 megawatts (MW) per million people, the region would need a total of 374 MW of installed capacity. That’s about twelve times the level of capacity in the region today. All energy sources would be needed to help provide that much capacity.
This is where aspiration runs into reality. In desperately poor countries, they do not have the luxury to spend millions of dollars on energy. Renewable energy sources like wind and solar energy can sometimes be useful where there is no electricity transmission system to take centrally-generated power to rural areas, but it is expensive and often requires technology to install and operate. Further, wind and solar are “intermittent” sources, meaning that they only produce energy when the wind blows or the sun shines respectively. Electrical energy is expensive to store and this can only be done in small amounts.
For reliable electrical energy supply for any possibility of industrial development and for transportation, developing countries need large scale power generation based on low cost, generally available fuels. In India, and in many parts of Africa, this means coal.
Coal reserves are available in almost every country worldwide, with recoverable reserves in around 70 countries. In fact, coal is the backbone of modern electricity in most parts of the world. It now provides about 30% of the primary energy and 41% of global electricity generation. It is plentiful and relatively cheap. Over the decade from 2000 to 2010, China showed the world how massive expansion of coal-fired electricity generation could modernize its economy and bring electrification to almost all parts of the country. As a result, hundreds of millions of Chinese have lifted themselves out of energy and economic poverty and dramatically improved both their income and quality of life.
Yet, coal is the most carbon-intensive of fossil fuels. It is the fuel source most despised by those who want to drastically reduce emissions. The Obama Administration in the United States has, as part of its climate change agenda, pressured the World Bank to stop lending to coal-fired electricity projects and the World Bank has complied. The U.S. Administration has also withdrawn funding from the Export-Import Bank for such projects. Fortunately for the developing countries, a new Asian Infrastructure Investment Bank has been established with major funding from China, which will include funding of new coal projects.
Those pursuing the climate change political agenda are prepared to condemn the world’s poor living without modern energy to remain in their backward situation. For them, billions of blighted lives are preferable to increasing greenhouse gas emissions.
Even in the developed countries, the policies advanced for climate reasons fall heavily on the poor.
Electricity prices continue to surge in Europe where costs are often triple those in the U.S. EU governments have various schemes, taxes, subsidies, and mandates, such as Cap and Trade, feed-in tariffs, and surcharges that make Europeans pay more for power. Perhaps the best (worst?) example is Germany, where nearly 20% of families now live in “fuel poverty,” spending more than 10% of household income on energy. Germany’s energy transition (“Energiewende”) is expected to cost an astounding $735 billion, and many are demanding changes. Overall in Europe, 1.4 million more households are expected to be in fuel poverty by 2020.
In the name of climate change, governments are forcing utilities to sign long-term contracts paying as much as four times the going wholesale electricity rate for renewables. Power markets have become so distorted that wind farms in the UK and in Ontario, for instance, have been paid millions to NOT produce electricity.
Supporters of “green” energy policies keep saying that poverty will be reduced if only efficiency would improve, but that position doesn’t hold up. Energy efficiency in the EU has improved around 20% since 2005. In the UK, for instance, energy efficiency has increased nearly 30% since 2003, yet electricity prices have almost doubled and homes in fuel poverty have nearly quadrupled. Europe’s main fuel poverty problem isn’t a lack of efficiency, it’s soaring prices.
Apart from the higher prices, another meaningful measure of energy poverty in Germany is the number of supply stoppages (“power cuts”) ordered by utility companies. Basic suppliers are entitled to interrupt their electricity or gas deliveries in the event of arrears in payment of more than 100 euros after a warning notice followed by a repeated threat to terminate service. According to a survey of the German Network Agency (Bundesnetzagentur), in 2013 warnings of electricity supply termination were issued to 5.7 million private households in Germany. The supply of electricity was actually interrupted to roughly 320,000 households.
There are many different moral standards to which one might refer in defining what is the most “ethical” way for people to act when considering their use of energy and other goods to improve their lives. Those environmentalists who claim that “nature” is more important than humans and that any measure, regardless of how costly, should be taken to reduce the effects of humans on the planet will never be satisfied. In my view, human wellbeing, and especially the plight of the world’s poor, deserves a prominent place in judgments about what is ethical behavior. Sharply reducing fossil fuel use means reducing economic development, condemning poor societies to remain poor, and requiring the poor people of today to sacrifice for the sake of addressing an unproven problem in a distant future — this is truly immoral.
Social science provides a lot of useful insight as to why logic and data are rarely convincing
By Matt Manos | Watts Up With That? | May 23, 2015
… In their latest speeches on global warming, Obama and the Pope weren’t trying to convince skeptics that Catastrophic Anthropogenic Global Warming (CAGW) is real. Instead, they were sending signals to their supporters on what “all right thinking people” should be saying. This is classic in-group/out-group communication. Obama and the Pope were setting up the talking points for their in-group members to use to determine who can be considered part of the tribe and who should be rejected for being outside of it. This is a process called Othering. Othering turns political foes into non-beings. Others have no value. Others can be discounted and ignored. Others can be mocked.
Obama and the Pope are examples of bellwethers; the sheep with the bell that the other sheep follow. Bellwether is not a derogatory term, it’s a descriptive term. The job of a political bellwether is to indicate the position that their followers should take in their everyday conversations. Obama and the Pope’s latest speeches function as position papers for the delegates of all right thinking people. You meet these people at work, church, school, at the coffee house, etc. The delegates will mirror the words that the President or the Pope used to identify other in-group members, normalize beliefs and mock out-group members. One of the main themes of both speeches was shame. Shame on those who aren’t right thinking people. Shame that they aren’t as intelligent and capable as “us.”
That type of smugness is almost impossible to penetrate. When a skeptic questions a warmist’s view on global warming/climate change, the warmist hears something vastly different than what the skeptic is saying. A skeptic might say, “The models don’t match the actual measured results.” What the warmist hears is how stupid deniers are because that’s what John Stewart told him he should think. If the warmist doesn’t prove that he thinks skeptics are stupid then he might be confused for a denier! And no one wants to be identified with being a denier because they’re mocked, don’t get tenure and don’t get invited to the right parties. No amount of science can penetrate the ROI the warmist has internalized in believing in CAGW.
Many of the warmists are running on pure rational ignorance. Rational ignorance is a belief that the cost/benefit to researching every issue is so low as to be a net negative in time utilization. Thus the ignorance is rational and everyone utilizes this mental process on certain topics. People who are rationally ignorant about global warming look to bellwethers that support their gut stance. Rationally ignorant warmists would look to world leaders, mockutainers and warmist scientists for guidance on how to communicate their position on global warming.
Penetrating rational ignorance is tough because the position warmists have taken isn’t based on logic. Their position is actually based on an appeal to authority. To question the rationally ignorant warmist is to question the field of science as a whole (to be a science denier) or to question the leadership of their favorite bellwether personalities. This will cause the rationally ignorant warmist to become defensive and try to stand up for their favorite bellwether. The rationally ignorant will also point to their favorite bellwethers and say, “Who am I to doubt all these intelligent people?” It’s intellectually offshoring. It’s lazy. It’s human nature.
The scientific method rejects outright in-group/out groups, Othering, bellwethers and rational ignorance. A scientist is supposed to follow the results of an experiment even if the results don’t support his hypothesis. The scientist is clearly not supposed to rig the data to ensure he gets invited to a party with the right people or continued funding. But science has a poor track record on controversial topics. It often takes decades to accept new theories that are clear winners (e.g., continental drift).
Scientists are still social animals. Social animals follow hierarchy and incentives. If you really want to win the debate on global warming, change the opinions of the bellwethers. Change the economic incentives for the global warming scientific paper mill. Otherwise you’re stuck debating only the people who are unable to change their minds because it would cost them personally to do so. Rare is the person intellectually honest enough to bite the hand that feeds or is willing to violate social norms to speak the truth.






