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Midazolam and morphine deaths

Dr. John Campbell | July 21, 2026

So many forgotten deaths.

UK Government Pushed Doctors to Use Deadly Drug Combo to Treat COVID Patients

By Jill Erzen | The Defender | July 22, 2026

The U.K. government recommended a risky drug combination for COVID-19 patients in 2020, just 16 days after warning doctors to use the medications together only as a last resort, medical commentator John Campbell, Ph.D., said this week.

For years, Campbell has criticized the widespread use of midazolam and morphine and other palliative drugs to treat COVID-19 patients, questioning why so many patients appeared to receive drugs commonly associated with end-of-life care — even though they didn’t necessarily have a terminal illness.

But in his latest video, he said he had overlooked a key piece of the puzzle: The U.K. government itself had backed guidance recommending the combined use of opioids and benzodiazepines for certain COVID-19 patients — just days after warning that the drug combination could cause fatal respiratory depression.

Campbell, who said a former member of the British Parliament brought the issue to his attention, called the apparent reversal “a national scandal which is being ignored.” … Full article

July 23, 2026 Posted by | Science and Pseudo-Science, Timeless or most popular, Video, War Crimes | , | Comments Off on Midazolam and morphine deaths

Henrik Svensmark fired by his university

By Karl Iver Dahl-Madsen | CLIntel | July 21, 2026

Something unusual and encouraging has happened in Danish climate journalism. Berlingske, one of Denmark’s leading national daily newspapers, has published an excellent and genuinely inquisitive article about the dismissal of physicist Henrik Svensmark from the Technical University of Denmark (DTU), where he has been working since the late nineties. The headline leaves no room for doubt: “Controversial Danish climate and space researcher has received the pink slip from DTU: It is ‘a death blow’ to my research”.

Here is how the article starts:

He is one of Denmark’s most internationally recognized researchers – and at the same time one of the most controversial. But now space and climate researcher Henrik Svensmark has, in his own words, been “good old-fashioned fired” by his employer, the Technical University of Denmark (DTU) in Lyngby. He does not rule out that “political considerations” – as he puts it – were involved in the years-long process that led to his final dismissal. At the same time, Henrik Svensmark does not hesitate to call it “a death blow” to a branch of research that he has largely developed single-handedly and that climate researchers around the world, including the UN’s climate panel (IPCC), have had to relate to – often reluctantly. This is mainly because Svensmark’s research points to what he sees as an overlooked natural factor behind a large part of the climate changes Earth has undergone. He finally does not rule out that the dismissal may be linked to the fact that he is perceived as a controversial researcher in wide circles: “There is nothing controversial about the research itself. But it is true that many perceive my research as controversial, even though it shouldn’t be.” DTU has no comments on the actual dismissal of Svensmark and refers to it as a personnel matter. However, the director of DTU Space, Henning Skriver, confirms that Svensmark’s overarching research area, atmospheric physics, will now “be significantly down-prioritized.”

Science journalist Lars Henrik Aagaard deserves credit for the whole article. Rather than dismissing Svensmark as merely ‘controversial’, he asks the essential question: is DTU retiring an ageing employee, or is it terminating an internationally recognized but scientifically inconvenient research program?

The facts reported by Berlingske are troubling.

Svensmark, aged 68, and well known for his work on cosmic rays and clouds, was still actively working and preparing a new experiment. Only a few months earlier, he says, his superiors had told him that his research was important and that they wanted him to continue.

According to Svensmark, he had also been selected for a professorship in 2016 following positive international evaluations. A new rector however, blocked his expected promotion to full professor, downgrading him to Senior Researcher (and lowering his salary). That move made it harder for him to secure regular research funding in Denmark.

In 2021, DTU again attempted to dismiss him, but he continued, partly, he says, because of protests from scientists connected with MIT and Princeton (Lindzen and Happer).

Berlingske presented this history to DTU. The university declined to address it. DTU Space director Henning Skriver merely stated that atmospheric physics was being “significantly downgraded” as part of a strategic prioritization.

That is not an explanation. It is management fog.

A public university may change its priorities. But it should explain the scientific reasoning. Why was Svensmark encouraged to continue only months before his dismissal? What happened to the positive international evaluation? Why was the professorship removed? Why is an active experiment being stopped?

The details of an individual employment case may be confidential. The scientific grounds for closing a research program shouldn’t be. A hypothesis cannot be dismissed administratively.

Solar activity

Svensmark’s research concerns the influence of solar activity and cosmic radiation on aerosol formation, clouds and therefore the climate.

The proposed mechanism is straightforward in principle. Solar activity affects the amount of cosmic radiation reaching Earth. Cosmic radiation ionizes the atmosphere. Ionization may influence the formation and growth of aerosol particles, some of which become cloud condensation nuclei. Clouds, in turn, strongly affect Earth’s radiation balance.

Parts of this chain have been demonstrated experimentally. The unresolved question is how large the resulting climate effect is under real atmospheric conditions and over different timescales.

Svensmark does not claim that carbon dioxide has no effect. He told Berlingske :

“I am not saying that humans are not part of it.”

His point is that natural variability, cloud processes and solar influence remain insufficiently quantified. He describes the dismissal as “a death blow” to the research program he has spent much of his career developing.

Not a marginal researcher

Svensmark cannot reasonably be dismissed as an unsuccessful or marginal scientist. Google Scholar records more than 7,000 citations to his work and an h-index of 30. Compared with the broader DTU research community, this places him approximately among the upper fifth of researchers and probably higher when measured by total citations. That is a considerable scientific impact for a relatively small and highly contested research field, far removed from the large collaborative networks that generate many citations almost automatically.

The predictable but weakest contribution in the article comes from Jens Hesselbjerg Christensen, professor at the Niels Bohr Institute and a prominent figure in the IPCC community.

Hesselbjerg accepts that cosmic radiation may play a role in cloud formation and long-term climate history. He then claims that much of Svensmark’s research appears to have been designed to confirm his hypothesis and that, whenever it was not confirmed, “an arm or a leg” was simply added.

That is an extraordinary accusation and an intellectually shabby one.

Hesselbjerg identifies no paper, no experiment, no faulty measurement, no improper method and no failed prediction. He merely insinuates that Svensmark has constructed his research to reach a predetermined conclusion. If Hesselbjerg believes that, he should name the experiments and explain precisely what was wrong with them. Otherwise, he should withdraw the allegation.

Developing a hypothesis as new evidence appears is not scientific misconduct. It is science. Aerosol formation involves nucleation, particle survival, growth, atmospheric chemistry and eventual cloud formation. Discovering additional mechanisms is not “adding an arm or a leg”. It is the whole purpose of experimental research.

Hesselbjerg’s performance illustrates a wider problem. Researchers close to the institutional consensus can invoke authority while providing remarkably little argument. A dissenting researcher is expected to prove every link in a complex physical chain before his work is even considered legitimate.

That is not a scientific level playing field.

The research should continue

The immediate objective should not merely be to embarrass DTU. It should be to ensure that Svensmark’s research survives. The most important next step is to develop a climate model that explicitly includes the proposed chain connecting solar activity, cosmic radiation, atmospheric ionization, aerosol growth and clouds, alongside greenhouse-gas forcing and the other established climate mechanisms.

Such a model would make it possible to test a question of enormous scientific and political importance: How much of the observed temperature development is caused by human influence, and how much is caused by natural variation?

That question is far from purely academic. Climate policy depends critically on the answer. If natural influences are larger than assumed, the expected effect of reducing carbon emissions is smaller. If they are negligible, the current attribution of climate change to CO2 becomes stronger. Either result would be valuable.

As explained by Henrik Svensmark: “The next decisive step is to build a climate model in which the influence of solar activity, cosmic radiation, aerosol growth and clouds is represented together with greenhouse gases. That would allow us to test how much of modern climate change is human-caused and how much is natural. This can be done within a few years and with a research budget that is modest compared with the political and economic importance of the question.”

The task is therefore concrete: we need to find a new institutional home for Svensmark, preserve the equipment and scientific expertise, assemble an international research team and raise the necessary funding.

Berlingske has performed a valuable public service by bringing this case into the open. DTU has responded with evasion. Jens Hesselbjerg Christensen has responded with an undocumented attack on a colleague’s scientific integrity.

Neither response is acceptable.

Science advances through experiments, observations and testable models, not through administrative priorities, institutional conformity or casual insinuations.

Henrik Svensmark’s research must not end with a DTU management decision. It must now be given the resources to face the only judgement that matters: the judgement of observations.


Karl Iver Dahl-Madsen is an independent consultant (owns Dahl-Madsen ApS), chairman of the board of the Danish climate-sceptical association Klimarealisme (Climate Realism), and a frequent commentator/debater in Danish media on climate policy, energy, and environmental issues. More: https://klimarealisme.dk/

July 21, 2026 Posted by | Full Spectrum Dominance, Science and Pseudo-Science | , | Comments Off on Henrik Svensmark fired by his university

Trump’s Nominee for CDC Director Calls mRNA Technology ‘Safe and Effective’

CDC Director Nominee shows a serious lack of scientific understanding

By Sharyl Attkisson | July 16, 2026

When asked at a Senate hearing whether she thinks mRNA vaccines are safe and effective, CDC Director-nominee Erica Schwartz replied, “I do believe that mRNA technology is safe and effective.”

Both the question and the answer show a serious lack of scientific understanding.

Here’s the scientifically accurate answer to the question: We don’t know the full safety profile of either of the two types of approved mRNA vaccines or the technology. First, It’s all too new. Second, we’re not even collecting the full data.

Read on for details.

According to FDA scientists and other experts in the field, comprehensive safety data isn’t known until a new drug (including vaccines) has been on the market and in widespread use for 7-12 years.

Even now, we aren’t getting full information on safety profiles because the data isn’t even being collected in the comprehensive manner required for accurate analysis.

The established scientific process requires that all illnesses after vaccination be meticulously recorded regardless of whether a patient or doctor thinks the illness is actually connected to the vaccine.

Most doctors are not following the process. Some misunderstand. Others are willfully ignoring. And no authority is ensuring they do their job.

Doctors typically aren’t even asking their ill patients whether they had an mRNA vaccine (for Covid or RSV), which one(s), and when. So they aren’t collecting that crucial data.

Most people, including physicians, don’t understand that patients being treated for any illness are supposed to be queried. This means, for example, someone who come to the ER with a retinal detachment should be asked if he had Covid vaccine, which, how many, and when, and then the data should be reported to the Vaccine Adverse Event Reporting System (VAERS). Same with someone who becomes sick with a rash, headaches, tendon rupture, stiff neck, depression, or chest congestion. Everything.

Even when patients do tell a physician they think an illness might be vaccine related, the physician frequently, improperly, determines on the front end that he doesn’t need to report the possible adverse event to the established database unless he thinks it’s connected to the vaccine. That’s not how the system works. No doctor is qualified to make that determination about a new medicine. All illnesses are supposed to be recorded so that previously unrecognized adverse events can be unearthed.

Additionally, many doctors and patients don’t understand— and aren’t being told— that adverse events from vaccines and other medicine can arise months or years after the medicine is taken.

Further, they don’t understand that an adverse event can be related to a drug even if the patient did not initially become ill after taking the vaccine or other medicine.

And the blanket question itself, “Are mRNA vaccines safe and effective,” shows a lack of scientific understanding on the part of the questioner. It begs counter-questions: are they safe for whom? Under what circumstances? Effective at what? It’s as ridiculous as asking, “Is medicine safe and effective?” Depends on whether or not you’re allergic to it. Depends on whether you have predispositions for things that make you more susceptible to the side effects. Depends on whether its particular mechanisms work in your individual biology. These are individual calculations.

Even in the general picture, there aren’t blanket answers. We know mRNA Covid vaccines failed woefully—proved ineffective—at a sliding scale of supposed goals: They don’t prevent infection. They don’t prevent spread. They don’t prevent illness. There’s a debate over whether they prevent serious illness, which most people don’t get from Covid, and children almost never get.

Until we stop pretending that the blanket questions make sense, and that the answers are connected in any way to science, we will continue to have leaders who misinform or mislead.

So, from diabetes drugs that can cause fatal genital tears to acne medicine that can cause sexual dysfunction that never goes away, here are some notable examples of unexpected adverse events that were eventually linked to vaccines or other medications.

  1. Sildenafil (Viagra) and blindness [non-arteritic anterior ischemic optic neuropathy (NAION)/sudden vision loss].
    Viagra treats erectile dysfunction by inhibiting PDE5 to increase blood flow. Early marketing focused on cardiovascular risks but not eye issues. Hundreds of post-approval reports eventually linked it to sudden vision loss from NAION. The adverse event was only recognized when enterprising physicians pressed the issue and published case reports, and I reported on them for CBS News. The FDA added warnings in 2005. Labels now advise stopping use and seeking care for sudden vision changes. Similar risks apply to the PDE5 inhibitor class.
  2. Likewise, Viagra was eventually connected to hearing loss.
    Reports emerged of sudden hearing decrease/loss (sometimes with tinnitus/dizziness) after use. Initially not linked, post-marketing data led to label updates advising immediate medical attention. Nobody initially guessed an erectile dysfunction drug could cause deafness and blindness!
  3. Statins such as atorvastatin and simvastatin, and severe and potentially fatal muscle pain/weakness (myalgia, myopathy, rhabdomyolysis).
    Statins are used to lower cholesterol to prevent heart disease/stroke. Early labels noted mild muscle issues, but the drugmakers initially denied severe cases (including rhabdomyolysis, which can cause kidney failure). Independent doctors, lawsuits, and my reporting for CBS News drew attention to this problem. Eventually, the drug makers added warnings, monitoring advice, and dose adjustments.
  4. Statins and cognitive effects (memory loss, confusion).
    Some users reported “brain fog” or confusion, initially dismissed as unrelated. I reported on this, too, for CBS News. Eventually, FDA added reversible cognitive side effects to labels in 2012 based on reports.
  5. Troglitazone (Rezulin) and severe liver failure.
    This thiazolidinedione treated type 2 diabetes. Only did post-1997 use among millions reveal dozens of acute liver failure cases (deaths/transplants), originally denied by the drugmaker. Reporting by the Los Angeles Times and then by me at CBS News forced the issue. Eventually, the drug was withdrawn in 2000 after FDA tied it to about 63 deaths. (Scientists say each recognized and reported death implies 1,000 to 100,000 more that are never reported.)
  6. SGLT2 inhibitors such as canagliflozin/Invokana, dapagliflozin/Farxiga, empagliflozin and potentially fatal Fournier’s gangrene (necrotizing fasciitis of perineum/genitalia).
    These diabetes drugs lower blood sugar via urinary glucose excretion. Believe it own ot, they can cause life-threatening flesh-eating infections in the genital/perineal area. FDA issued a warning in 2018. The labels now carry strong alerts.
  7. mRNA Covid-19 vaccines (Pfizer, Moderna) and myocarditis/pericarditis heart issues.
    As you know, these vaccines were found to cause heart inflammation, mostly in young men after second dose. This was initially unexpected.
  8. Covid-19 vaccines and menstrual irregularities.
    Many women reported changes such as heavier bleeding and cycle shifts, but were largely called conspiracy theorists. This adverse event like so many others linked to Covid vaccines, weren’t identified—at least they weren’t reported— after initial studies.
  9. Covid-19 vaccines and eye issues. Even without persistent data gathering, there have been enough reports of eye inflammation and retinal problems to raise concern over a possible link.
  10. Thalidomide and severe birth defects.
    Once marketed as a sedative and morning sickness aid in the 1950s–60s, it turned out to cause more than 10,000 cases of babies born with shortened or absent limbs and other defects.
  11. Isotretinoin (Accutane) for acne and persistent sexual dysfunction.
    Recent FDA updates added erectile dysfunction, decreased libido, vaginal dryness, and lubrication issues that may persist even when people stop taking the drug.
  12. A similar effect was initially denied by later found with SSRI antidepressants, Selective Serotonin Reuptake Inhibitors. They can cause persistent sexual dysfunction.
  13. Cisapride (Propulsid) for indigestion and cardiac arrhythmias/QT prolongation. It was withdrawn after heart rhythm deaths. I reported for CBS News on the coverup of a baby death in a fraudulent Propulsid study.
  14. Rofecoxib (Vioxx) and cardiovascular events. Nobody guessed a pain medicine and arthritis drug would cause heart attacks and strokes. All concerned initially denied the signals but the medicine was eventually removed from the market for this reason.
  15. Fenfluramine/phentermine (Fen-Phen) weight loss drug and heart damage. This is another drug that caused unpredicted heart problems and was eventually taken off the market after the FDA had approved it as “safe and effective.”
  16. Certain fluoroquinolones and tendon rupture/peripheral neuropathy. Who would have guessed antibiotics could cause a tendon rupture or nerve damage? Eventually, all agreed that fluoroquinolones can do this, and a serious “black box” warning was added to the label.
  17. Antipsychotics and tardive dyskinesia. Drugs meant to control psychotic tendencies can actually cause serious movement disorders, which nobody anticipated initially.
  18. What about when a cancer treatment causes cancer? Tamoxifen for breast cancer can cause and uterine cancer. Nobody predicted that. The warning was later added to the labels.
  19. Ketamine for pain and anesthesia and bladder damage. This side effect only emerged when there was greatly expanded psychiatric use of the medicine.
  20. Lots of medicines cause unexpected bone loss, including medicine that’s prescribed to protect bones! They include bisphosphonates like Fosamax or alendronate, Actonel, Boniva, and Reclast. They are commonly prescribed to treat and prevent osteoporosis by slowing bone breakdown. However, after lots of use, scientists learned they can actually cause serious bone problems, including thigh bone fractures, osteonecrosis of the jaw (dead jawbone), and delayed bone healing. FDA eventually added warnings and updated the labels. Other drugs eventually linked to bone loss and fracture risk include: corticosteroids like prednisone for inflammation, asthma, autoimmune diseases, and allergies; acid reflux drugs like omeprazole/Prilosec or esomeprazole/Nexium); aromatase inhibitors (like anastrozole/Arimidex) for breast cancer; and the injectable birth control shot Depo-Provera.

July 17, 2026 Posted by | Science and Pseudo-Science | , | Comments Off on Trump’s Nominee for CDC Director Calls mRNA Technology ‘Safe and Effective’

Vaccines Did Not Cause Rachel’s Autism

An Essay on the Book Peter Hotez Wrote to Prove It

Lies are Unbekoming | July 13, 2026

Rachel Hotez is a real adult, now in her early thirties. Every description of her in this essay comes from her father’s own words in his 2018 book. The argument is with the book, not with her.


The vaccine visit

Peter Hotez, in his 2018 book Vaccines Did Not Cause Rachel’s Autism, describes his daughter at her pediatric appointments. Rachel “would cry longer and with much fiercer intensity than our other children.”¹ The sentence appears once. Hotez does not return to it. Rachel is one of four Hotez children. Among them, she is the only one on the spectrum, and the only one whose reaction to the injections her father describes in these terms.

Peter Hotez is not a random pediatrician. He holds an MD and a PhD. He is Dean of the National School of Tropical Medicine at Baylor College of Medicine, Co-Director of the Texas Children’s Center for Vaccine Development, and founding editor-in-chief of the journal PLOS Neglected Tropical Diseases. He served as a U.S. Science Envoy under the Obama administration. In the years after 2020 he became one of the most visible defenders of vaccination policy on American cable news, and in 2022 was nominated for the Nobel Peace Prize for developing a low-cost COVID vaccine. His 2018 book was the opening statement in that public role. His daughter is the girl the title is defending.

The book runs to two hundred pages. Roughly two thirds of them are Rachel. Her first words, her flights across the neighborhood, her sneakers thrown from a moving car onto the Merritt Parkway at sixty miles per hour, her decades of intellectual disability that leaves her at twenty-five sorting donated clothes for fourteen dollars a day at Goodwill.¹ The other third argues that none of this can be attributed to the injections.

The book has an unusual quality. It is not, in the strict sense, a defense of the vaccine schedule. It is a father’s project to explain his daughter without implicating his own life’s work. Front to back, the book reads like the case Hotez was building against himself.

Rachel Hotez was born in the early 1990s. Peter Hotez was on the faculty at Yale, developing a vaccine for hookworm. Ann Hotez had two older children already. Rachel, the third of what would become four, is described in the book as an easy baby, “content to sit in her car seat, in the dining room or another quiet place and read.”¹ Her first year was, in her mother’s words, unremarkable. She sat unsupported later than her siblings, at nine months rather than six, but her parents attributed the delay to individual variation. “Children do not all learn and grow in the same ways, or at the same speed,” Ann later wrote.¹

By eighteen months, Rachel was not walking. She was not talking. Her pediatrician, Simone Simon, raised the concern. Ann Hotez writes that she and Peter had not seen it: “How could it be that Peter, a pediatrician himself, and I, an experienced mother, hadn’t noticed? I think we had seen differences, but we attributed them to what we knew, or thought we knew, about child development.”¹

The referral was to the Birth-to-Three intervention team at the Darcey School. Rachel was starting to talk by twenty months. By twenty-nine months she was functioning at the eighteen-month level in most areas. At Yale in the spring of 1995, she was diagnosed by Dr. Wendy S. Levine with pervasive developmental disorder, not otherwise specified. She was three years old.

Between eighteen and twenty-four months, Rachel had lost the trajectory she was on. Her father’s book identifies this window precisely, as a matter of clinical description, and returns to it as the central subject of his Chapter 9.

The timeline that contradicts itself

Chapter 9 of Hotez’s book is titled “What Does Cause Autism? The Scientific Evidence.” It is the book’s central scientific move. Hotez cites a series of brain imaging studies, most prominently a 2017 paper from Joseph Piven’s group at the University of North Carolina–Chapel Hill.² Piven’s team scanned the brains of infants whose siblings already had autism, and who were therefore considered at higher risk, at multiple points during infancy. They found measurable changes in the brains of children later diagnosed as early as six to twelve months of age. Specifically, the outer layer of the brain, the cortex, expanded faster than normal between six and twelve months, and the brain as a whole grew larger than expected between twelve and twenty-four months. The larger brain size coincides with the age at which most parents first recognize the condition.

Hotez presents this as decisive. If measurable brain changes are present at six months of age, he argues, then the vaccines given at twelve and eighteen months cannot have caused those changes. He writes: “The changes in the brains of kids with ASD are set into motion well before (about a year) many parents recognize any signs of alterations in communication or social behavior.”¹

The argument depends on what a reader does not know, or does not pause to consider.

The current injection schedule for an American newborn begins within hours of birth, with the compound marketed as vitamin K. That injection is not formally part of the vaccine schedule but is administered nearly universally. The formal vaccine schedule begins on the same day, with hepatitis B. It resumes at two months, with DTaP, Hib, pneumococcal conjugate, inactivated polio, rotavirus, and a second hepatitis B dose. At four months, most of the same combination is repeated. At six months, most of it is repeated again, along with the first influenza dose. By six months of age, a child has received approximately twenty vaccine doses, several of which contain aluminum adjuvant. The cumulative aluminum burden by six months of age has been estimated at approximately 4.4 milligrams,³ a figure Hotez himself cites in Chapter 8 while comparing it favorably to dietary aluminum in infant formula.¹

The comparison is where the omission is starkest. When aluminum is eaten in food, less than one percent is absorbed into the body. When aluminum is injected as part of a vaccine, it is designed to stay. The compound is added to vaccines because it holds at the injection site and provokes the inflammatory response that makes the vaccine work. French research groups have documented that aluminum-loaded white blood cells remain at injection sites for years,⁴ and that these particles are then carried through the lymphatic system to distant tissues, including the brain.⁵ Aluminum has been recovered from brain tissue of autism decedents at concentrations substantially higher than in age-matched controls.⁶ Christopher Shaw and Lucija Tomljenovic have documented dose-response relationships between pediatric aluminum burden and autism prevalence across multiple countries.⁷ None of this literature appears in Hotez’s book. Aluminum adjuvant is addressed in a single dismissive paragraph in Chapter 8, primarily by reference to the Children’s Hospital of Philadelphia comparison to formula.¹

The Piven timeline does not exonerate the vaccine schedule. It identifies the window in which vaccine-induced injury would produce measurable effects. Whatever is producing the cortical expansion Piven documented at six to twelve months of age is happening after the birth-through-six-month injection schedule, not before it. Hotez names the window. He does not name the exposures inside it.

Rachel’s regression was observed at eighteen months. She had received the standard childhood schedule available at the time. What Piven’s MRI cannot see, because his study was not conducted until decades later, is what Rachel’s brain looked like at six months, or at twelve months. What is preserved in the record is Ann Hotez’s testimony that she had bonded less to Rachel than to her older children, that Rachel was “quiet” and “content,” that Rachel’s motor milestones were behind but her attention span seemed strong. What is also preserved is Peter Hotez’s own observation that Rachel cried longer and more intensely at the needle than her siblings did.

What happens in the window

Piven’s timeline identifies when brain changes become measurable. It does not identify what causes them. Hotez’s book, having named the timeline, moves on. The question of what happens in the birth-through-six-month window remains open. Answering it requires setting the book aside.

Aluminum hydroxide and aluminum phosphate are added to vaccines because they persist. The industry term for a compound added to a vaccine to intensify the body’s response is adjuvant. Aluminum functions as an adjuvant because it resists clearance. The body’s repair processes engage the compound and cannot dispatch it. The resulting sustained inflammation is what the industry calls efficacy.

The design depends on a biological assumption medicine does not defend openly. It assumes that the body will confine its response to the injection site. It will not.

Beginning in 1998, Romain Gherardi and colleagues at the French National Institute of Health and Medical Research described a condition in adults who had received aluminum-adjuvanted vaccines. Muscle biopsies at the injection sites showed distinctive lesions: aggregates of white blood cells called macrophages, filled with aluminum hydroxide. The lesions were present years after the injection. Aluminum, in other words, did not clear. It remained at the site, engulfed by cells that could not digest it.⁴

Fifteen years later, Zakir Khan and Gherardi’s group published a follow-up study. Using fluorescent aluminum hydroxide particles injected into the muscle of mice, they demonstrated that the particles were carried away from the site by macrophages, drained through the lymphatic system, and arrived in distant tissues, including the brain, over weeks and months. The transport depended on a specific signaling molecule, CCL2, that summons macrophages to sites of inflammation. Blocking CCL2 stopped the process. Restoring CCL2 restored it.⁵

Aluminum is injected. It provokes the inflammation it was designed to provoke. White blood cells arrive to engulf it. They cannot digest it. They carry it, embedded within themselves, through the lymphatic system to wherever the body is calling for their services. In an infant whose blood-brain barrier is still developing, they carry it into the brain.

Christopher Exley’s group at Keele University has recovered aluminum from brain tissue samples of people who died with autism, at concentrations substantially higher than in age-matched controls without autism.⁶ Christopher Shaw and Lucija Tomljenovic at the University of British Columbia have documented dose-response relationships across countries: as the pediatric aluminum injection burden has risen, so has autism prevalence.⁷ The correlations do not prove causation. They form the kind of convergent signal the studies Hotez cites in Chapter 8 would have been designed to test, if the field had been oriented toward asking the question.

In 1913, Charles Richet was awarded the Nobel Prize in Physiology or Medicine for his description of anaphylaxis. What Richet actually demonstrated, in a series of experiments on dogs beginning in 1901, was that injecting a foreign protein into an animal produced a heightened response to any subsequent exposure to the same protein. The first injection sensitized. The second could kill. Richet named the process anaphylaxis, from the Greek for “without protection.”¹⁹ His finding, that the injection route sensitizes the body against future encounters with the same substance, is Nobel-documented history.

Every childhood vaccine contains foreign proteins injected in the presence of aluminum designed to hold. The Richet mechanism is not disputed. It is simply not applied to childhood vaccination in the mainstream literature, because to apply it would be to concede what the industry is designed to deny.

The reader does not need to accept every step of this reasoning to hold the essay’s central point. Aluminum accumulates. It travels. It has been recovered from the brains of people who died with autism. The mechanism that would produce sensitization from injected foreign proteins won a Nobel Prize. None of this appears in the book Hotez wrote to close the question.

Rachel’s regression window opened at eighteen months, months after the aluminum-containing doses at two, four, and six months would have completed the biopersistence and transport described above. What her brain looked like at six months of age is not preserved in the record. Neither is her cerebrospinal fluid aluminum concentration at any age. Neither is any measurement that would let a family ask, decades later, whether their daughter’s condition was set in motion by exposures her father’s book does not consider possible.

The years of intervention

By age five, Rachel had a formal IQ evaluation. Her verbal IQ was 84, near the low end of normal. Her performance IQ was 60. Later testing put her performance IQ in the 40s.¹ She was placed on Prozac, then Zoloft, then Luvox, then Risperdal. Each medication, in Hotez’s account, produced worse effects than the last. She was eventually taken off all psychiatric medication.

Rachel had two psychiatric admissions at Yale-New Haven’s Winchester 1 inpatient unit. An EEG revealed right-sided temporal lobe spike discharges. She was placed on tegretol for a period, with what Hotez describes as “possibly some improvement.”¹ The tegretol was eventually discontinued because Rachel would not comply with the blood draws needed for level monitoring. The Hotez family lived through years that Peter describes with candor: “dreary or frightening,” “wearing us down,” “she seldom gave much back emotionally, compared with the other children.”¹

The family relocated to Houston in 2011, where Peter had accepted a position at Baylor College of Medicine. Rachel finished her secondary education at Lamar High School with a certificate. She could not sustain the transition program at Houston Community College. Two brief residential placements failed. She lived, and continues to live, at home with her parents.

By 2016, Hotez had begun writing what he calls “science tikkun” pieces for PLOS. In early 2017 he published an op-ed in the New York Times titled “How the Anti-Vaxxers Are Winning.”⁸ The book followed in 2018.

The name that does not appear

Hotez names his opponents. Andrew Wakefield, characterized as an “elaborate fraud” quoting Brian Deer’s BMJ series. Robert F. Kennedy Jr., named in connection with campaigning around thimerosal and working with the parent group Safe Minds. The film Vaxxed, called “phony.” Hotez identifies a “toxic combination of hysteria and pseudoscience,” “phony propaganda,” “fake news, half-truths, and conspiracy theories.”¹

He does not name William Thompson.

William Thompson is a senior scientist at the U.S. Centers for Disease Control and Prevention. In August 2014, through his attorneys at Morgan Verkamp LLC, Thompson issued a public statement about a 2004 study he had co-authored in the journal Pediatrics.⁹ The study, DeStefano et al., examined children in the Atlanta area, comparing when they received the MMR vaccine to whether they later developed autism. It concluded there was no link.¹⁰ Thompson’s 2014 statement was direct:

I regret that my coauthors and I omitted statistically significant information in our 2004 article published in the journal Pediatrics. The omitted data suggested that African American males who received the MMR vaccine before age 36 months were at increased risk for autism. Decisions were made regarding which findings to report after the data were collected, and I believe that the final study protocol was not followed.⁹

Thompson provided documents to Congressman Bill Posey. On July 29, 2015, Posey read Thompson’s statement into the Congressional Record on the floor of the U.S. House of Representatives.¹¹ Thompson has never recanted the statement. He remains employed at the CDC. His full statement, released through his attorneys at Morgan Verkamp LLC, is archived among the Vermont Legislature’s official witness testimony documents from its 2015 hearings on vaccine policy.⁹

Vaxxed, the film Hotez dismisses in his book as “phony,” is a documentary built around Thompson’s disclosure. It contains recordings of Thompson’s conversations with the biologist Brian Hooker. Hotez’s characterization of the film appears in a book that never names its subject.

The DeStefano 2004 study Thompson repudiated is one of the studies Hotez cites in Chapter 8. It appears in the list of investigations that, in his summary, demonstrate no link between MMR and autism.¹ The reader is not told that a senior author of one of those investigations has publicly stated that statistically significant findings were omitted.

The pattern extends beyond Thompson. Hotez’s Chapter 10, titled “Struck by Lightning,” offers his central injury statistic: approximately one severe adverse event per one million vaccine doses. He derives the figure by dividing roughly 300 annual compensated claims from the National Vaccine Injury Compensation Program by roughly 300 million annual doses.¹ The comparison to being struck by lightning is presented as authoritative.

The math depends on the pieces used. Hotez’s numerator is the number of NVICP claims that were compensated. NVICP dismisses more claims than it pays. Claims must be filed within three years of the injury appearing. Causation must be proven to a narrow list of conditions the program formally recognizes. Conditions that appear months or years later are not included. The denominator, 300 million doses, is total administered doses. The Vaccine Adverse Event Reporting System, VAERS, is meant to be the surveillance instrument that catches adverse events at the population level. In 2011, a study conducted by Harvard-Pilgrim Health Care under a grant from the Agency for Healthcare Research and Quality, principal investigator Ross Lazarus, was submitted to the federal government. Its finding on VAERS capture rate was that “fewer than 1% of vaccine adverse events are reported.”¹²

Hotez does not mention the Lazarus report. The underreporting problem does not appear in his book. His lightning comparison depends on the Lazarus figure being wrong by a factor of a hundred, and that dependence is not addressed. A correction of two orders of magnitude would move his rate from one per million to one per ten thousand. At the CDC’s own current estimate of 1 in 36 children diagnosed with autism,¹³ the question of what a serious adverse event actually is, and how it is counted, is the question the book was written to close.

Hotez closes it by not opening it.

The perpetual gene

In 2017, Peter and Ann Hotez arranged with the Baylor Department of Genetics to sequence their own DNA and Rachel’s. Whole exome sequencing produces the sequences of the protein-coding regions of the genome. The stated purpose was to identify any variants that might be linked to autism or intellectual disability.

Hotez describes the result with unusual restraint. Rachel had “some genetic variants, including one affecting a gene that could be linked to ASD or mental disabilities.”¹ He writes that the family plans to submit her results to the Baylor Johns Hopkins Center for Mendelian Genetics and to the NIH-supported Undiagnosed Diseases Network, “in order to determine if Rachel’s genetic variants might also be present in other individuals on the autism spectrum or with other mental health conditions.”¹

The sentence is worth reading twice. The whole exome sequencing did not identify a cause of Rachel’s condition. It identified variants of uncertain significance that Hotez hopes might one day be shown to be relevant.

This is the outcome the book has been building toward. Chapter 9 promises that autism is genetic. Hotez cites work from the Simons Foundation and Princeton estimating that as many as one thousand genes may eventually be identified as contributing to autism.¹⁴ At the time of the book’s publication, sixty-five had been identified. The remaining nine hundred and thirty-five are described as awaiting discovery.

The reader is asked to accept a paradigm that has produced sixty-five candidate genes across three decades of intensive investigation, and to expect that the next three decades will produce the remaining nine hundred and thirty-five. The larger project has similar dynamics. The Human Genome Project promised to identify the genetic basis of common disease, and produced approximately twenty thousand genes rather than the one hundred thousand originally predicted. Its subsequent genome-wide association studies for autism have identified small-effect variants that account for a modest fraction of the heritability those studies were designed to explain. The gene has been coming for thirty years.

Meanwhile the environmental candidates Hotez does name in Chapter 9 are curated. He cites Phillip Landrigan’s 2010 review identifying prenatal exposures associated with autism-like presentations: valproic acid, thalidomide, misoprostol, chlorpyrifos.¹⁵ He cites maternal rubella exposure during pregnancy as a cause of congenital rubella syndrome, which he says “can closely resemble autism.”¹ From this he draws a conclusion that returns the reader to the book’s title with a strange inversion: “The ‘R’ component of the MMR vaccine is actually the rubella vaccine that protects a mother from transmitting rubella virus to her baby, and in so doing functions as an effective vaccine against autism.”¹

The MMR vaccine, according to Hotez, prevents autism. This appears in a book titled Vaccines Did Not Cause Rachel’s Autism.

He raises maternal fever and points to Ian Lipkin’s Columbia group work on maternal viral exposures.¹⁶ He notes a 2017 Kaiser Permanente study that found a 1.2 odds ratio for autism among children whose mothers received influenza vaccine in the first trimester.¹⁷ He dismisses the finding as “not statistically significant after adjusting for multiple comparisons.” An odds ratio of 1.2 in a study of nearly two hundred thousand children is not a finding a scientist would dismiss if he were looking for the cause. It is a finding a scientist would dismiss if he had already located the cause elsewhere.

Aluminum adjuvant does not appear in Chapter 9. In a chapter titled “What Does Cause Autism,” the compound most commonly injected into infants under six months of age, one with documented biopersistence, translocation, and central nervous system deposition, is not discussed as a candidate.

Nor does the chapter engage the growing literature comparing vaccinated to unvaccinated cohorts. Anthony Mawson’s 2017 pilot study of homeschooled U.S. children reported that the vaccinated group had substantially higher rates of neurodevelopmental disorders, allergies, and chronic conditions than the unvaccinated group.¹⁸ Studies of this design remain the most direct empirical test of the question Hotez’s book is written to close. None appear in his citations. The one study design that could definitively answer the question, he dismisses in a single line elsewhere in the book: a randomized trial of vaccinated versus unvaccinated children would, he writes, be “unethical.”¹

The book’s opening dedication lists the funding sources for Hotez’s Center for Vaccine Development at Texas Children’s Hospital. Among them: the Bill & Melinda Gates Foundation. The Carlos Slim Foundation. Gavi, the Vaccine Alliance. UNICEF. The World Health Organization. The Kleberg Foundation. The Blavatnik Charitable Foundation. The Brockman Medical Research Foundation. The Japanese Global Health Innovative Technology Fund. The Southwest Electronic Energy Medical Research Institute. The National Institutes of Health. The Centers for Disease Control and Prevention. The Walter Reed Army Institute of Research. The United States Public Health Service. Baylor College of Medicine. Texas Children’s Hospital.¹

Hotez addresses this preemptively in Chapter 8. He notes that he holds patents on his vaccines but has “not received a penny” and has “no real prospects for financial gain.”¹ The disclaimer is technically accurate. It is also beside the point. Institutional capture does not require kickbacks. It requires career. A scientist whose salary, laboratory, institutional affiliation, and public standing all depend on the paradigm the book defends is not neutral, whether or not he personally profits from any particular product.

Rachel at twenty-five

At the end of the book, Rachel is twenty-five. She lives with her parents in Montrose, Houston. Her routine is fixed. She wakes at four in the morning to Skype her friend Sabrina in Denver. Her morning walk takes her to Randall’s supermarket for a plain bagel, no butter. Later she walks to Subway for a six-inch tuna sandwich, no cheese, mustard on hearty Italian. Along the way she talks to shopkeepers, asks strangers about their dogs, and occasionally brings home men she meets on the street, whom her father has to ask to leave.

She has, at the time of the book’s writing, just been enrolled in a Goodwill training program. A chance airport encounter between her parents and the wife of the Goodwill Houston board chairman produced the introduction. Rachel sorts donated clothes for stains and rips. She works two hours a day. After taxes, Ann Hotez writes, “it is about $14 a day and $215 so far.”¹

This is what the book’s title is defending. A young woman with a performance IQ in the 40s who cannot count money, cannot sustain a classroom, cannot hold employment for more than two hours per day, and lives with her aging parents in a neighborhood where they worry, in the book’s own words, about her safety at night. Hotez writes with clear love for his daughter. He describes Rachel as loyal to her friends, curious about people, empathetic toward animals, quick to strike up conversation in the neighborhood. She is all of these things. She is also a young woman whose life was, at some point, altered.

Her father spent two hundred pages arguing that the alteration cannot be attributed to what he did for a living. The William Thompson statement of August 2014 is not addressed. The aluminum burden accumulated during the first six months of life is not examined as a candidate cause. The injury statistic in Chapter 10 depends on assuming that VAERS captures nearly all vaccine-related injuries, an assumption the Harvard-Pilgrim report says is wrong by a factor of a hundred. The alternative causation runs through a genetic paradigm that has produced sixty-five candidate genes in thirty years and promises another nine hundred and thirty-five to come. On one page inside a book denying vaccine-autism links, the MMR vaccine is inverted into an anti-autism intervention.

The book was written to close the question. What it does instead is document, in loving and exhaustive detail, the life of the girl whose story the question was always about. Rachel cried at the injections longer and more intensely than her siblings did. Between eighteen and twenty-four months, she lost skills. Her EEG showed spike discharges. Her intellectual disability is profound. She sorts clothes at Goodwill.

The door her father wrote his book to close is still open. He walked past it in every description of his own daughter.


References

  1. Hotez PJ. Vaccines Did Not Cause Rachel’s Autism: My Journey as a Vaccine Scientist, Pediatrician, and Autism Dad. Baltimore: Johns Hopkins University Press; 2018.
  2. Hazlett HC, Gu H, Munsell BC, Kim SH, Styner M, Wolff JJ, et al. Early brain development in infants at high risk for autism spectrum disorder. Nature. 2017;542(7641):348–351.
  3. Offit PA, Jew RK. Addressing parents’ concerns: do vaccines contain harmful preservatives, adjuvants, additives, or residuals? Pediatrics. 2003;112(6 Pt 1):1394–1397.
  4. Gherardi RK, Coquet M, Cherin P, Belec L, Moretto P, Dreyfus PA, et al. Macrophagic myofasciitis lesions assess long-term persistence of vaccine-derived aluminium hydroxide in muscle. Brain. 2001;124(Pt 9):1821–1831.
  5. Khan Z, Combadière C, Authier FJ, Itier V, Lux F, Exley C, et al. Slow CCL2-dependent translocation of biopersistent particles from muscle to brain. BMC Med. 2013;11:99.
  6. Mold M, Umar D, King A, Exley C. Aluminium in brain tissue in autism. J Trace Elem Med Biol. 2018;46:76–82.
  7. Tomljenovic L, Shaw CA. Do aluminum vaccine adjuvants contribute to the rising prevalence of autism? J Inorg Biochem. 2011;105(11):1489–1499.
  8. Hotez PJ. How the anti-vaxxers are winning. New York Times. February 8, 2017.
  9. Thompson WW. Statement of William W. Thompson, Ph.D., regarding the 2004 article examining the possibility of a relationship between MMR vaccine and autism. Released through Morgan Verkamp LLC; August 27, 2014. Archived by the Vermont Legislature, H.98 witness testimony, May 6, 2015. Available at: https://legislature.vermont.gov/Documents/2016/WorkGroups/House%20Health%20Care/Bills/H.98/Witness%20Testimony/H.98~Jennifer%20Stella~William%20Thompson%20Statement~5-6-2015.pdf
  10. DeStefano F, Bhasin TK, Thompson WW, Yeargin-Allsopp M, Boyle C. Age at first measles-mumps-rubella vaccination in children with autism and school-matched control subjects: a population-based study in metropolitan Atlanta. Pediatrics. 2004;113(2):259–266.
  11. Posey B. Remarks on the William Thompson statement. Congressional Record. July 29, 2015;161(120).
  12. Lazarus R, Klompas M, Bernstein S, et al. Electronic Support for Public Health–Vaccine Adverse Event Reporting System (ESP:VAERS). AHRQ Grant Final Report, Grant No. R18 HS 017045; 2011.
  13. Maenner MJ, Warren Z, Williams AR, Amoakohene E, Bakian AV, Bilder DA, et al. Prevalence and characteristics of autism spectrum disorder among children aged 8 years — Autism and Developmental Disabilities Monitoring Network, 11 sites, United States, 2020. MMWR Surveill Summ. 2023;72(2):1–14.
  14. Krishnan A, Zhang R, Yao V, Theesfeld CL, Wong AK, Tadych A, et al. Genome-wide prediction and functional characterization of the genetic basis of autism spectrum disorder. Nat Neurosci. 2016;19(11):1454–1462.
  15. Landrigan PJ. What causes autism? Exploring the environmental contribution. Curr Opin Pediatr. 2010;22(2):219–225.
  16. Mahic M, Mjaaland S, Bøvelstad HM, Gunnes N, Susser E, Bresnahan M, et al. Maternal immunoreactivity to herpes simplex virus 2 and risk of autism spectrum disorder in male offspring. mSphere. 2017;2(1):e00016-17.
  17. Zerbo O, Qian Y, Yoshida C, Fireman BH, Klein NP, Croen LA. Association between influenza infection and vaccination during pregnancy and risk of autism spectrum disorder. JAMA Pediatr. 2017;171(1):e163609.
  18. Mawson AR, Ray BD, Bhuiyan AR, Jacob B. Pilot comparative study on the health of vaccinated and unvaccinated 6- to 12-year-old U.S. children. J Transl Sci. 2017;3(3):1–12.
  19. Richet C. Anaphylaxis. Nobel Lecture, December 11, 1913. Nobel Media AB. Available at: nobelprize.org.

July 15, 2026 Posted by | Book Review, Science and Pseudo-Science, Timeless or most popular | | Comments Off on Vaccines Did Not Cause Rachel’s Autism

The Animal Substance

From the Calf’s Wound to the Current Schedule

Lies are Unbekoming | July 10, 2026

Decorous and admissible language fails me, in alluding to that which might have seemed incredible thirty years ago—the commanding of vaccination on a second child of a family, when vaccination has killed the first; and then sending the father to prison for refusal.

— Emeritus Professor F.W. Newman, 1874


Author’s Note

This essay draws heavily on Dissolving Illusions: Disease, Vaccines, and the Forgotten History by Dr. Suzanne Humphries and Roman Bystrianyk. Their decade of archival work—recovering the primary documents, mortality tables, medical journal articles, and photographs that mainstream history has quietly buried—made this essay possible. The vaccine farms, the horse stables, the named children, the foot-and-mouth outbreaks, the Beddow Bayly address, the Lancet admissions about equine origin: all of it comes from records that were sitting in libraries and archives waiting for someone willing to look.

Where I have drawn from their book, the sources they cite are primary documents—USDA bulletins, Lancet articles, court records, contemporary medical journals. I have verified and expanded where useful, but the archaeological work is theirs.

Readers who want the full documentary foundation for what follows should read Dissolving Illusions. It is the essential reference on this subject. What I offer here is a narrower cut: the material itself—what has been in the vials, from Jenner’s day to the current schedule.

I have also drawn on Michael Willrich’s Pox: An American History for the Camden and vaccine-farm details, on the peer-reviewed work of H.V. Wyatt for the 1916 Rockefeller passages, on the CDC’s own excipient documentation for the current inventory, and on the work of Dr. Sherri Tenpenny for details on the specific-pathogen-free egg supply chain.

The interpretation is my own. The evidence belongs to the record.


The calf was led from the stable to the operating room and strapped to the table. Its belly was shaved. The skin was washed, then scarified with a surgeon’s knife—superficial linear incisions cut into the shaved abdomen and thighs. Vaccine material was smeared into the bleeding cuts with an ivory or metal instrument. The animal was released to a pen. About a week later, when the wounds had ulcerated and infectious material flowing from them, the calf was brought back. The contents of the ulcers were scraped out, mixed with glycerin, drawn into vials, and shipped.

This was smallpox vaccine. The procedure was documented in the medical and agricultural literature of the period, photographed and described without controversy. The USDA published detailed accounts of the vaccine farms in its Bureau of Animal Industry reports. The medical journals published the technique. Physicians described the work in their own textbooks. Nobody was exposing anything. This was standard industrial practice. The vaccine industry did this on a national scale, in facilities called “vaccine farms,” from 1870 until well into the twentieth century. The material went into the arms of American schoolchildren.

The calf, once bled of its material harvest, was frequently returned to the herd. Some vaccine farms rented their calves from local dairies. When the collection was complete, the animals went back into the food supply.

This is what The Science looked like. Not as an abuse of the paradigm—as the paradigm itself, functioning as designed. For a hundred and fifty years, the vaccine industry’s core problem was biological: how to obtain the raw material. The solutions—cow, horse, sheep, goat, monkey, chicken, mouse, dog, pig, and eventually the aborted human fetus—define the history of what medicine considered “immunization.” Every generation of vaccines has been an animal product, in one direction or another. What follows is the record.


Part One: Before the Farms (1796–1870)

Edward Jenner named his product after the Latin word for cow, vacca. He believed cowpox in cows originated from a disease in horses called “the grease”—an eruption on the horse’s heels caused by an inflammatory condition of the skin. In 1829 the Lancet published a note revealing that the lymph Jenner had been circulating for three or four years around Berkeley was drawn not from cows but from horses. He had, according to the Lancet, “decisively ascertained before he died” that the disease he was using was equine, not vaccine [cow] pox.

By 1834, the medical literature reflected complete confusion about what the substance actually was. A contemporary article listed three competing theories: Jenner’s grease-of-the-horse origin, the theory that the material was smallpox modified by passage through cows, and the theory that cowpox was a disease as native to cows as scarlatina was to man—unrelated to smallpox at all. A French practitioner cited in the same article maintained that in France there was no evidence cowpox had ever appeared in cows at all.

The confusion was not academic. It described what was being scratched into people’s arms.

For a hundred years after Jenner, the standard procedure was arm-to-arm vaccination. Material containing pox was rubbed from the arm of one inoculated child into cuts made on the arm of the next. In this way, the substance was passed forward through generations of children, sometimes serially through dozens or hundreds of hosts before anyone thought to trace its origin. Whatever else was in the material of the last child—syphilis, tuberculosis, hepatitis, the mercury and heavy metals of nineteenth-century treatment—travelled with it.

The procedure required a “good take”: a substantial pustule forming at the site of the wound. To ensure this, doctors made incisions at multiple sites on the same arm at one sitting—up to four wounds per child. The photograph titled “Multiple site vaccination of 1898, showing a typically good arm” shows the result: an arm ruined across four separate infection sites.

Arm-to-arm vaccination was outlawed in England in 1898. Multi-site vaccination continued in various parts of the world until 1975.

A second method existed for towns where arm-to-arm passage was impractical. Human pox scabs were dropped into a jar. Water was added. The jar was shaken. The resulting material was used as vaccine for the entire town.

In 1952, Dr M. Beddow Bayly summarised what a century and a half of “smallpox vaccine” had actually consisted of. His words, delivered in a public address, describe the state of the vaccine supply as it stood in the middle of the twentieth century:

When we recall that vaccine material is derived, in the first place, either from a smallpox corpse, the ulcerated udder of a cow, or the running sores of a sick horse’s heels, the choice depending upon the country of its origin and the firm which manufactures it, it is hardly to be wondered at that it has far-reaching ill effects on the human constitution.

He continued, quoting the Lancet‘s earlier admission: “no practitioner knows whether the material he employs is derived from smallpox, rabbit-pox, ass-pox, or mule-pox.” Bayly noted that England’s own Ministry of Health had “long confessed to complete ignorance of the ultimate source of its own supply.” A British Medical Journal contributor of the period stated that the strain used for routine material preparation in England was “believed to have been derived from a case of smallpox in Cologne during the last century.”

The corpse. The udder. The horse’s heel. The unknown source. This was the substance for a hundred years.


Part Two: The Industrial Calf (1870–1930)

In 1870, calf-based production began in the United States. The original starting material was imported from France and inoculated into a herd of cows at a farm near Boston. Within a few years, “vaccine farms” had sprung up across the country. The proprietors were mostly medical doctors who identified an opportunity to profit from rising demand. The farms produced smallpox vaccine at first. They soon diversified into diphtheria material and other biologics.

The procedure was the one described in this essay’s opening. Calves were rented or purchased, strapped to operating tables, scarified, inoculated, released to incubate, and brought back for the material harvest. Some farms returned the animals to their owners. Others slaughtered them. The New England Vaccine Company, one of the larger commercial operations, ran a farm at Wakefield, Massachusetts, rented calves from a farmer named Owen Clark, and returned them to circulation once the material had been collected.

The commercial products of this era carried the names of firms that later became household pharmaceutical corporations. H.K. Mulford and Company. Parke Davis. Wyeth. Lederle. The vaccine industry was born on these farms.

Two consequences followed. Neither was hidden. Both were documented at the time in the medical and agricultural literature.

Foot-and-Mouth Disease

Production on living calves that were subsequently returned to the food supply produced periodic outbreaks of foot-and-mouth disease in cattle populations. The disease is highly contagious among cattle and causes economic devastation. Outbreaks occurred in 1870, 1880, 1884, 1902, and 1908.

The 1902 outbreak lasted six months and affected 244 herds. Of these, 205 were slaughtered—3,872 cattle, along with 360 hogs and 220 sheep and goats. The USDA published detailed instructions for the burial of the carcasses: trenches deep enough for five feet of cover dirt, hides slashed to prevent exhumation for the leather trade, quicklime poured over the meat.

The origin of the 1902 outbreak was traced to the New England Vaccine Company and to Dr E.E. Tyzzer’s experimental work at the Wakefield farm—the farm where calves were rented from Owen Clark for the production of vaccine material. The 1908 outbreak was traced to a Japanese vaccine strain imported by another manufacturer (”Manufacturer B” in the USDA report) to improve their standard product. The strain carried foot-and-mouth disease. Because Manufacturer B killed its calves after harvest, the material remained internal for a period. Manufacturer A, on the other hand, rented calves and returned them to circulation—which is how the disease entered the general cattle population.

People who worked with cattle developed severe blistering conditions. The 1902 medical literature contains detailed case reports of butchers who received cuts on their hands while working with animals and subsequently developed bullous eruptions across their bodies. Dr John Bowen documented the connection at Massachusetts General Hospital in 1904.

Human recipients of the smallpox vaccine developed the same conditions. Multiple case series were published between 1902 and 1911 documenting acute pemphigus in children who had recently been vaccinated. The New Orleans outbreak of the same period produced cases in children who had never been near a butcher. The vaccine itself was the vector.

The Camden School District, October 1901

In early October 1901, an eight-year-old girl in Camden, New Jersey died of smallpox. Her father followed her, then seven of her siblings. In the panic, the Camden school board announced it would enforce an 1887 vaccination law. The board took bids. The Mulford pharmaceutical company won the contract.

By the end of October, the arms of approximately 5,000 Camden schoolchildren had been scraped with a metal prong and rubbed with Mulford’s calf-derived material.

On the first of November, William Brower, sixteen years old, died. He had been vaccinated nineteen days earlier. Over the following weeks, eight more children died. Every one of them, with a single exception, had received the Mulford vaccine at school. Children vaccinated at the free downtown clinic or by their own physicians were unaffected.

A parallel outbreak occurred at Pennsylvania Hospital, where 4,500 patients and staff had been vaccinated with the same product. Mulford’s official explanation, delivered by the company’s advertising and sales manager—a 29-year-old chemist named Albert C. Barnes, later famous as the founder of the Barnes Foundation art collection—appeared in the New York Times. The dead children, Barnes wrote, came from a “lower class of people” whose “carelessness” had “poisoned the wounds.” The Camden Board of Health had commissioned the investigation from a Mulford employee. It did not occur to anyone that this constituted a conflict of interest.

Bacterial spores that cause death by lockjaw are common in soil and manure. Production on calves in stables and pens exposed the material harvest to constant potential contamination. The calves were strapped to tables in rooms that had, in many cases, been used for stabling. No sterile technique protected the wound-culture from the animal’s environment.

The dead children were not the exception. They were the visible edge of a system that produced its raw material in barns.

Lübeck, 1930

The end of the animal-vaccine-farm era was marked, in Germany, by an incident that made the same principle explicit in a different biological medium.

Between December 1929 and April 1930, 251 newborns in the town of Lübeck received three oral doses of Bacille Calmette-Guérin (BCG), a tuberculosis preparation derived from the bovine tuberculosis organism, within the first ten days of life. The programme had been approved by the town’s health council and its medical association. Posters advertised it. Newspapers endorsed it.

Seventy-seven of the vaccinated infants died. One hundred seventy-three developed what was identified as active tuberculosis.

The cause was traced to the laboratory where the BCG material was prepared. In the same unlocked incubator space, what was identified as virulent human tuberculosis organisms (the Kiel strain) were also being cultivated. There was no separate designated area for vaccine preparation. There was no animal testing to confirm the safety of the batches before administration. The vaccine and the other organism shared a room. Dr Georg Deycke, head of the general hospital, was later convicted of negligent homicide and sentenced to two years. Ernst Altstaedt received fifteen months.

The BCG preparation itself is a live bovine organism. The organism was originally isolated from the udder of a tuberculotic cow in France by Albert Calmette and Camille Guérin, then serially processed by passing it through 230 subcultures over thirteen years. The Lübeck disaster ended the oral route of administration worldwide. The preparation itself, still derived from the same 1908 bovine isolate, remains in use today.


Part Three: The Horse Stables (1895–1940s)

What medicine calls diphtheria antitoxin, introduced in 1895, was manufactured from horse blood.

The procedure was straightforward. A horse was injected with escalating doses of material extracted from what medicine calls diphtheria toxin over a period of weeks. The body responded to the injected substance by producing what is identified as antibodies. The horse was then bled—large quantities of blood drawn from the jugular vein—and the serum extracted. The serum was drawn into vials and injected into humans.

The first commercial producers included Parke Davis, Mulford, and the New York City Board of Health. By the early 1900s, horses were being used to produce a range of preparations: the diphtheria material, what was called tetanus antiserum, meningococcus material, and staphylococcus preparations in multiple varieties. A 1911 New Zealand pharmacopoeia list includes—among many other products—an item called simply “Normal Horse Serum,” administered as a therapeutic agent.

The horse serum was foreign protein. The human response to repeated injection of foreign animal protein was documented by Charles Richet in 1901, work that won him the 1913 Nobel Prize. Richet showed that injection of foreign proteins created sensitisation—the body responds with increasing intensity to subsequent exposures. Serum sickness—fever, joint pain, rashes, kidney inflammation, and in severe cases death—was a documented consequence from the early years and remains listed on package inserts as a caution today.

The mortality curve tells its own story. In Leicester, England, the death rate from what medicine calls diphtheria had been declining steadily for the fifty-seven years from 1838 to 1895. In 1895, horse-serum preparation was introduced. The death rate then rose to approximately ten to fifteen times its previous level and stayed elevated for the next five years.

In New York City, the death rate among children under ten fell from 785 per 100,000 in 1894 to under 300 by 1900—a decline that was already well underway when the serum came into use. By 1920, when the toxoid preparation was introduced, the rate had already fallen below 100. The mainstream story credits the horse serum and then the preparation with these declines. The curves credit sanitation, nutrition, and improved living conditions.

Jim

On the second of October 1901, a horse named Jim was euthanised at the St. Louis city stable. Jim was a former milk wagon horse, retired to the poorhouse two years earlier and put to work producing what was called diphtheria antitoxin for the St. Louis Board of Health. Over the course of his career, Jim produced more than thirty US quarts—about twenty-nine litres—of serum.

Two days before his euthanasia, on the thirtieth of September, Jim had been routinely bled. The blood was drawn into flasks and processed into serum. By the time Jim showed signs of illness and was killed, the serum had already been bottled and distributed.

Jim had what was identified as tetanus. The serum drawn from him on the thirtieth of September was contaminated with spores from this condition in incubation phase.

Dr Amand Ravold, the physician responsible for the operation, was aware of the danger. He ordered the September thirtieth batch destroyed. It was not destroyed. Bottles labelled “August 24”—a date when Jim’s serum had been clean—were filled with the contaminated September thirtieth material. The bottles were distributed to the physicians of St. Louis.

The first child began convulsing on the twenty-sixth of October 1901. Her name was Veronica Keenan. She was one of two Keenan children who had received the serum as a preventive measure. Neither had shown symptoms related to what the material supposedly protected against. Within a week, all three Baker children were dead. The symptoms included arched back and convulsions.

Thirteen St. Louis children died. The last, on the seventh of November.

The court of inquiry named Ravold and the janitor Henry Taylor as responsible. Both were dismissed. Ravold went on to a distinguished career. He served as president of the St. Louis Medical Society. His 1942 obituary made no mention of the deaths.

The St. Louis deaths and the Camden deaths, occurring in the same weeks of the same autumn, produced sufficient public pressure that Congress passed the Biologics Control Act of 1902. Historians describe this as the origin of American vaccine regulation. What it regulated was the horse stables and the calf farms.

Use of what was called diphtheria antitoxin dropped sharply nationwide in the aftermath. In Chicago, physicians and parents refused it. The death rate from what medicine calls diphtheria in Chicago that year rose by a third.


Part Four: The Monkey House (1955–present)

By the mid-1950s, the vaccine industry had largely abandoned the calf farms and the horse stables. The new production medium was cell culture. The new species was the rhesus macaque and, later, the African green monkey.

The Rockefeller Passages

Between 1910 and 1916, at the Rockefeller Institute in Manhattan, Simon Flexer and his associates were serially passing material through the spinal cords of rhesus monkeys. The material was extracted from a monkey, injected into the spinal cord of another monkey, and then extracted again after paralysis developed. This passage process was repeated many times, selecting for material that would replicate highly in the neural tissue of monkeys. Occasionally, the passages were reinforced with fresh material from human cases.

By 1916, the resulting material had become highly destructive to nervous tissue and capable of high replication in multiple cell types.

In May 1916, an outbreak began in Brooklyn. It reached 23,000 cases and 5,000 deaths. It moved through New England and the Middle Atlantic states, reaching Delaware, Maryland, and the District of Columbia. The death rate was twenty-five percent—sixteen times higher than typical presentations. The proportion of two-year-olds affected was the highest ever recorded. The outbreak began in early May, well before the normal summer season. None of these features were ever recorded again in any similar outbreak.

The first known case lived a few blocks from a rail line that connected via the Brooklyn Bridge and 63rd Street directly to the Rockefeller Institute, three miles away, where Flexner’s laboratory was conducting the passages. The material being cultivated there had been selected, through serial passage, for unprecedented ability to damage the nervous system of the hosts receiving it. Dr H.V. Wyatt published this analysis in 2011.

No investigation was undertaken at the time. No inquiry into the laboratory’s work. No examination of what material had been cultivated or where it might have gone. The outbreak was attributed to Italian immigrants. Immigration records show the outbreak began before the accused children arrived. The official explanation required no investigation because it required no explanation.

Salk, Sabin, and the Monkey Kidney

The preparations introduced in the 1950s were produced by growing material on the kidney cells of monkeys. The kidneys were removed from live rhesus macaques—hundreds of thousands of them, over the years—minced, and used as culture medium.

In 1960, Bernice Eddy, a researcher at the National Institutes of Health, discovered that the monkey kidney cells routinely used were contaminated with a virus. She called it Simian Virus 40 (SV40). When injected into hamsters, SV40 produced tumors. When mixed with human cells in culture, it transformed them—the standard laboratory signature of a cancer-causing virus.

The material had been in mass administration in the United States since 1955. Approximately 98 million Americans had received it. Every dose administered before 1963 contained SV40. Doses after 1963 were required to be screened, but the screening was, in Stanley Kops’ documented analysis, incomplete—the seed strains themselves were never fully verified. SV40 has been detected in material produced through the 1990s.

SV40 has since been found in human tumors: mesothelioma of the lung, several types of brain tumor, and cancers of the bone, breast, colon, and kidney. Dr Michele Carbone, one of the principal researchers in the field, called SV40 “the perfect war machine”—it affects at least four major cellular mechanisms that either promote tumor growth or interfere with the cell’s cancer defences. It is not found in the healthy tissue surrounding these tumors.

When Carbone and Dr Harvey Pass prepared to publish their findings, a senior NIH figure told Carbone that if he or Pass spoke to the press “against his wishes,” they would be “punished.” Pass said afterwards: “I didn’t think you got punished for science.”

Formaldehyde was the killing agent for the preparation. SV40 was shown in 1961 to survive formaldehyde treatment beyond the standard twelve-day treatment period. The manufacturer’s cited standard remained twelve days.

Monkeys are still used in production today.

The Cutter Incident

In April 1955, several batches of Cutter Laboratories’ preparation—produced on monkey kidney cells—contained what was identified as live, unattenuated material that had survived the formaldehyde process. Within days, children who had received the Cutter preparation began developing paralysis. The final tally: 40,000 children affected, 200 permanently paralyzed, ten dead.

Cutter’s product was the only one recalled. In 1990, Freedom of Information Act documents revealed that Wyeth had also produced batches with similar properties during the same period. Wyeth’s product remained on the market. Congressman Percy Priest, chair of the investigation, later stated: “We felt that no lasting good could come to science or the public if the Public Health Services were discredited.”

Swedish researchers, testing their own supplies in the aftermath of Cutter, discovered that thirty percent of batches previously certified as safe contained what was identified as live material when re-tested. Dr Sven Gard, the Swedish expert, later stated that the American material in 1955 caused as much paralysis as it prevented.


The pattern would repeat. Regulation followed disaster, then legitimised the practice it was meant to constrain. Expansion followed.

Part Five: The Current Inventory

The vaccine schedule administered to American children in 2026 is the direct descendant of the calf farms, the horse stables, and the monkey house. What has changed is the range of species. What has not changed is the principle: biological material derived from animals is grown, harvested, processed, and injected into the human body.

The list below is drawn from the CDC’s Vaccine Excipient Summary and from the FDA-approved package inserts of the current vaccines. It is not a critic’s characterisation. It is the manufacturers’ declaration of what is in their products.

From cattle. Fetal bovine serum. Bovine serum albumin. Bovine calf serum. Calf serum protein. Bovine extract. Bovine muscle tissue. Bovine protein. Lactalbumin hydrolysate. Lactose. Present in DTaP, Td, Tdap, IPV, rotavirus (RotaTeq), hepatitis A (Vaqta), Japanese encephalitis (Ixiaro), MMR, MMRV, varicella, zoster, HepA-HepB, Pentacel, Kinrix, and Pediarix.

Fetal bovine serum extraction represents industrial blood harvest. When pregnant cows arrive at slaughter, workers discover the pregnancy during processing. While the mother cow is dying but not yet dead—before the umbilical cord is cut—they ram a large-bore needle directly into the beating heart of the unborn calf, draining all the blood. The fetus is killed through exsanguination while still connected to the dying mother. This blood is then centrifuged to separate the serum, creating the product injected into children. The different names on package inserts—fetal bovine serum, calf serum, newborn calf serum—indicate the age of the fetus when killed, with older fetuses yielding more blood and different grades of serum.

From chickens. Egg protein. Ovalbumin. Chicken protein. Chick embryo cells. Chick embryo fibroblasts. Chick kidney cells. Present in every standard influenza vaccine (Fluzone, Fluvirin, Fluarix, Flulaval, Afluria, Agriflu, FluMist), yellow fever, rabies (RabAvert), MMR, and MMRV.

The influenza vaccine supply chain requires 500,000 eggs weekly to produce 76 to 80 million doses annually. These eggs come from specialized hatcheries maintaining “specific pathogen-free” (SPF) status—tested for approximately thirty designated organisms. Critically, coronaviruses are excluded from the testing panel despite being endemic in chickens, existing there symbiotically like yeast on human skin. This means every dose produced from SPF eggs contains chicken coronaviruses that are then injected into humans. This undisclosed viral presence exposed generations to pre-existing reactions, which later manifested in severe reactions when COVID vaccines were administered—explaining the widespread anaphylaxis requiring emergency equipment at vaccination sites.

From pigs. Hydrolysed porcine gelatin. Standard porcine gelatin. Present in Zostavax, MMR-II, yellow fever, several influenza vaccines, Japanese encephalitis (JE-Vax), and varicella. Trypsin—an enzyme extracted from pig pancreas—is used as a processing agent in the manufacture of the rotavirus vaccines. In 2009, a porcine virus type 2 (a virus associated with wasting disease in pigs) was discovered in both licensed brands of infant rotavirus vaccine. It had entered the manufacturing stream through the pig-based enzyme.

From monkeys. Monkey kidney tissue is still used in material production. Vero cells—an immortalised cell line derived in 1962 from the kidney of an African green monkey—are used in production of the polio, rotavirus, and rabies vaccines currently distributed.

From dogs. Madin-Darby Canine Kidney (MDCK) cell protein and MDCK cell DNA. Present in Flucelvax influenza vaccine. The cell line was established from the kidney of a cocker spaniel in 1958.

From mice. Mouse serum protein. Present in the Japanese encephalitis vaccine JE-Vax.

From insects. Baculovirus. Insect cell lines derived from Spodoptera frugiperda (the fall armyworm moth). Present in Flublok recombinant influenza vaccine.

From humans. MRC-5 cells and WI-38 cells. Both are cell lines derived from the lung tissue of aborted human fetuses. MRC-5 was established in 1966 from the lung of a fourteen-week-old male fetus. WI-38 was established in 1962. Present in hepatitis A (Havrix), Twinrix, MMR-II, MMRV, varicella, zoster, rabies (Imovax), the adenovirus vaccine, and Pentacel. The residual DNA of the aborted fetus—cellular DNA fragments from the original 1962 or 1966 tissue, carried forward through decades of cell passage—remains in the final vaccine as an unavoidable component of the manufacturing process. Human serum albumin (drawn from pooled human plasma) is present in the ACAM2000 smallpox vaccine and the rabies vaccine.

The current inventory is an incomplete list. Each package insert specifies the ingredients for a single vaccine. The consolidated list is the responsibility of researchers who compile it from many sources.

The list is what medicine considers, in 2026, to be the state of the art.

What Was Actually Happening

The historical record is not ambiguous. For a hundred and fifty years, the vaccine industry took biological material from the wounds, glands, blood, kidneys, and cell lines of animals—first cattle, then horses, then monkeys, and eventually chickens, dogs, mice, pigs, insects, and the tissues of aborted human fetuses—processed it minimally, drew it into vials, and scratched, scarified, or injected it into the bodies of healthy children.

The material was, by definition, foreign to the recipient. Whatever else was in it—the spores from the stable floor, the SV40 from the monkey’s kidney, the foot-and-mouth material from the calf that had been rented back to a dairy, the porcine virus from the pig-based enzyme processing—travelled with the intended payload. The vaccine industry’s own package inserts describe these contaminants as “residual.” The word describes what remains. It does not describe how much of it is there, or what it does when injected.

The suppression of independent inquiry continues into the present. In 2016 and 2017, Italian researchers Gatti and Montanari—scientists whose rigorous methodology had made them trusted industrial product testers for European governments—analysed vaccine contents using electron microscopy and mass spectrometry. They found multiple vaccines contaminated with undeclared metallic particles including stainless steel, tungsten, lead, zirconium, and other industrial materials never listed on ingredient disclosures. When they published these findings, their laboratory was raided by authorities in 2018, equipment confiscated, government contracts terminated, and they were run out of the country. The message remained constant across generations: measure the vaccines and lose everything.

The mainstream story is that this practice worked. The mortality curves say otherwise. What medicine calls diphtheria declined ninety-seven percent between 1900 and the mid-1940s before the preparation was introduced. What medicine calls whooping cough declined more than ninety percent before the preparation of the mid-1940s. What medicine calls measles declined more than ninety-eight percent before the preparation of 1963. Scarlet fever, for which no preparation was ever widely deployed, declined by a comparable amount over the same period. The declines correlate with sanitation, nutrition, refrigeration, and the exit from overcrowded slum housing.

The population whose grandparents received the Mulford calf material, whose parents received the horse-serum preparation, and whose children receive the fetal-bovine-serum-cultured products of the current schedule is the same population. It is the reader’s family, in serial generations, submitting the same veins to the same industry.

The record contains named children. Bessie Baker was six. Veronica Keenan was four. William Brower was sixteen. The Lübeck infants had names their mothers had chosen a few weeks before administering the poster-advertised preparation. The children paralyzed by the Cutter material are alive today in some cases, in wheelchairs.

The vaccine industry did not begin in the mid-twentieth century. It began on the operating table where the calf was strapped. The industrial infrastructure—the farms, the horses’ stables, the monkey colonies, the fetal cell repositories—was constructed generation by generation to solve one problem: how to produce, at scale, biological material that could be pushed through a needle into a healthy person. The material has always been what the animal or the fetus produced.

The name given to this practice was “vaccination.” What was actually happening was the industrial-scale injection of foreign biological substance into the bodies of populations that had, in most cases, no way to refuse. The material was drawn from a calf’s wound, or from a horse’s neck, or from a monkey’s kidney, or from the lung of a human being who was aborted in 1966 and whose cells are still being passaged in laboratories in 2026.

This is what The Science looked like. This is what The Science is.

The photograph from the vaccine farm era shows a calf strapped to a wooden table, its belly shaved and cut. The image exists in the archival record. It was published without controversy. It described the standard practice.

The photograph is what was injected.


How to Explain This to a Six-Year-Old

Your child asks: “What does the needle do?”

You say: “A long time ago, doctors wanted to help people not get sick. But they didn’t really understand how sickness works. So they had an idea: What if we take the sick stuff from a cow or a horse, and put it in a healthy person? Then maybe that person’s body will learn how to fight the sickness.”

Your child asks: “Did it work?”

You say: “That’s the interesting question. Sometimes people got sicker instead of better. And sometimes they died. The doctors noticed this, but they didn’t stop. They kept doing it anyway.”

Your child asks: “Why?”

You say: “Because once they started, it was hard to stop. More and more people used it. Factories were built to collect this stuff from animals. People got jobs doing this work. Money was being made. When people asked questions about whether it was actually helping, they got in trouble or lost their jobs.”

Your child asks: “What happened?”

You say: “People’s bodies got used to the animal stuff. And sometimes other bad stuff was mixed in with it—poison from the stables, diseases from other animals. People got sick from the animal stuff itself, not from the sickness they were supposed to be protected from.”

Your child asks: “Do they still do this?”

You say: “Yes. They just use different animals now—chickens, monkeys, and even cells from people. They call it science. They call it protecting you. But if you look at what actually happened to people’s health, it went up because houses got better, food got cleaner, people had less crowded places to live. The animal stuff wasn’t the reason people got healthier. But the people making and selling it got rich, so they kept saying it was.”

Your child asks: “So should I get the needle?”

You say: “That’s a question for your family to decide together. What I’m telling you is what actually happened, and what’s actually in the needle. The needle contains animal stuff. Sometimes it helps. Sometimes it hurts. But the doctors and the companies making it won’t be honest about that part. They’ll say it’s completely safe. They have money riding on you believing that. So you need to know what’s actually true.”

Your child asks: “How do I know what’s true?”

You say: “Ask the question: Where did this come from? What’s actually in it? What happened to people who got it? What happened to people who didn’t? Then read the answers yourself. Don’t let someone else tell you what’s true just because they’re a doctor or they’re in charge. Look at the actual evidence. That’s how you know.”


The Shape of the Ritual

A healthy child is brought to a room. A trained man in a robe or coat receives the child. He holds a small vial. Inside the vial is a substance drawn from the body of an animal — a cow, a horse, a chicken, a monkey, a pig, or a human being who was killed before birth. The substance was obtained by inflicting injury on the animal: the calf strapped and cut, the horse bled from the neck, the monkey’s kidneys removed and minced, the fetus exsanguinated through a needle to the heart while still connected to its dying mother. The substance is drawn from the animal’s suffering.

The man in the coat holds the child. He pierces the child’s skin with a metal instrument. He pushes the substance from the animal into the child’s flesh. The child cries. The parents are told this is protection. They pay for it, or the state pays for it. They are told that refusal would be an act against the child.

The shape is old. It is older than germ theory, older than the Latin word vaccina, older than Edward Jenner and the horse’s heel and the calf farm at Wakefield. In its previous incarnations it was called by other names. In each case the mechanics are the same: a substance from a suffering animal, mediated by a priest-class, delivered into the flesh of the healthy in exchange for a promise. In the older forms, the promise was protection from famine, plague, or unseen enemies. In the current form, the promise is protection from disease. The substance and the ceremony have not changed. The vocabulary has changed.


The hermetic-alchemical tradition — the current of Western esoteric thought that reached Renaissance Europe through Pico della Mirandola and Johannes Reuchlin, was carried forward at the court of Elizabeth I by John Dee, was systematised in the Rosicrucian manifestos of 1614 and 1615, and became the intellectual substrate of Freemasonry and its later derivatives — held that the human being is base matter to be worked upon. The goal of the operation, called the Great Work, was the transformation of that base matter into something higher through serial technical operations conducted by adepts. The material was to be purified, mixed, tested, and refined until it moved. This was not metaphor. The alchemists attempted the operation on matter and on themselves.

Among the specific images transmitted through this tradition was the creation of an artificial being — an animate creature made from dead matter by human hands rather than by God. Medieval sources, drawn into the Western hermetic mainstream by the sixteenth-century Christian Kabbalists, describe the operation as following the pattern of a calf. The calf was the template. Frances Yates, the twentieth century’s principal historian of the hermetic tradition, documented in The Occult Philosophy in the Elizabethan Age how this material entered Christian Europe through Pico, Reuchlin, and above all John Dee, whom Elizabeth I employed as her royal astrologer and code-named 007. The 1615 Rosicrucian Confessio Fraternitatis announced the program that would shape the next four centuries: “that which in before times hath been unseen shall be spoken forth and uttered.” The program had two parts. The first was the transformation of the human being into something the operator had produced. The second was the eventual open confession of the operation to a public that had been ritually prepared to accept it.

The tradition that carried this program into the Anglo-American world was Rosicrucian, Freemasonic, and — in its American form — the network of secret societies of which Skull and Bones (founded 1832 at Yale) is the best-documented example. The industrial builders of the American vaccine industry were embedded in this world. Frederick Taylor Gates, who directed Rockefeller philanthropic funding into the Rockefeller Institute where Flexner conducted the 1910–1916 monkey passages, was a Baptist minister who described medical research in explicitly missionary terms. The Mulford, Parke Davis, Wyeth, and Lederle firms were Anglo-Protestant industrial operations. What linked them was not shared religion but shared intellectual atmosphere: the hermetic-alchemical conviction that the human being is material to be improved through technical operation, and that the technicians conducting the operation stand outside the moral rules that apply to the operated-upon.

Michael Hoffman, in Secret Societies and Psychological Warfare, argues that modern medical practice — the injection of animal cells into the brain, the transplantation of pig organs into humans, the fetal-cell-cultured pharmaceuticals — is the industrial-scale completion of this ancient project. He calls the resulting creature the hunimal: the human-animal hybrid produced by the operation. The individual doctor does not know this. The individual mother in Camden did not know it. Nor did the chemist Albert C. Barnes at Mulford. Knowledge of the design was not a requirement for the design to be executed. The design proceeded by generations of technical improvement on a substrate — the calf, the horse, the monkey, the aborted human being — that was always the same substrate.


Whether one accepts Hoffman’s specific framework — that the operation is hermetic-alchemical in origin, that its trajectory is intentional across centuries, that the industrial infrastructure of modern medicine is the material completion of a magical program — the correspondence exists on its own. The mechanics of the practice map, point for point, onto the mechanics of an operation that older traditions considered forbidden. The forbidden operation was the mixture of the human being with animal substance for the purpose of transforming what the human being was. The older tradition understood this as a fundamental violation of the created order — the crossing of a line that God had placed between kinds.

The Book of Leviticus contains numerous injunctions against the mixture of substances: two kinds of seed in one field, two kinds of thread in one garment, animal blood consumed with meat. These injunctions were categorical. They were not explained. They were held to reflect a structural feature of creation — that the kinds were distinct, that the blood belonged to the animal, that the human being was not to be mixed with what was not human. The injection of foreign animal substance into the human bloodstream is, by the terms of this older understanding, the exact operation the older tradition forbade.

The vaccine industry did not invent this. It industrialised it. What was performed in one temple with one calf on one day of the year became a schedule administered to every child in every country over a lifetime of appointments. The scale is new. The operation is not.


The photograph exists. The children’s names exist. The package inserts exist. What remains is the recognition of what the practice is at its root. It is not medicine that has gone wrong. It is an operation that was never medicine. It was, from the strapped calf onward, a ceremony that took its substance from the wounds of animals and placed that substance into the flesh of the healthy in the name of protection, while producing — generation by generation — the transformation of the recipient into something the operator had made.

The word for this operation, in every tradition that has a word for it, is the same word. The tradition that gave the modern West its ethical vocabulary — the biblical tradition — called the operation an abomination, and the being produced by it, an unclean thing. The hermetic-alchemical tradition, working in the opposite direction, called the operation the Great Work and the being produced by it, the artificial man. The two traditions agree on what is happening. They disagree on whether it is good.

The vaccine is the substance of the Great Work, industrialised. The vaccinated body is the vessel of the operation. The generations that have received the material — from Bessie Baker to the child in the pediatrician’s office this afternoon — are the material upon which the operation was performed.


Truth Be Told: I’ve Accepted an Invitation to Speak on The Unvaccinated

On September 17th, I’ll be giving a one-hour presentation titled The Unvaccinated as part of a six-hour livestream called Truth Be Told. This is the first time I have accepted an invitation to an event, and I have been honoured with the opening act. The livestream begins at 12pm EST.

Vaccination is the subject closest to my heart, and this is another opportunity to spread the word. The format will preserve the pen name.

Jamie Andrews (Decentralized Science Projects) and Agent131711 (Dinosaurs) will also be presenting. Jamie’s Virology Control Studies work led to an interview here last year. Agent’s research shaped my essays on vitamin D and dinosaurs. Tickets are here. The code UNBEKOMING is $5 off and applies automatically at that link. Replay available afterwards. Hope you can make it.


Primary Historical Sources

Bayly, M. Beddow. “Inoculation Dangers to Travellers.” Speech at Caxton Hall Westminster, October 2, 1952. Published by the London and Provincial Anti-Vivisection Society.

Bowen, John T. “Acute Infectious Pemphigus in a Butcher, During an Epizotic of Foot and Mouth Disease, with a Consideration of the Possible Relationship of the Two Affections.” Journal of Cutaneous Diseases Including Syphilis, vol. XXII, no. 6, June 1904, pp. 254–264.

Mohler, John R., and Milton J. Rosenau. “The Origin of the Recent Outbreak of Foot-and-Mouth Disease in the United States.” US Department of Agriculture, Bureau of Animal Industry, Circular 147, 1909.

Salmon, D.E. “Foot-and-Mouth Disease; Warning to all Owners of Cattle, Sheep, and Swine.” US Department of Agriculture, Bureau of Animal Industry, Circular No. 38, December 1902.

United States Department of Agriculture. “Method of Slaughtering and Burying Cattle.” Yearbook of the United States Department of Agriculture, 1915, pp. 20–21.

St. Louis 1901 Diphtheria Antitoxin Incident

“1901 Diphtheria Antitoxin Contamination Incident.” Wikipedia, May 4, 2026, en.wikipedia.org/wiki/1901_diphtheria_antitoxin_contamination_incident.

Wellcome Collection. “Jim, the Horse of Death.” wellcomecollection.org/stories/jim–the-horse-of-death.

Camden 1901 Smallpox Vaccine Incident

Dixon, Mark E. “Why Nine Camden Children Died from Smallpox Vaccines in 1901.” Mainline Today, September 2016.

“Why Nine Camden Children Died from Smallpox Vaccines in 1901.” Slate, February 9, 2021, slate.com/technology/2021/02/smallpox-vaccine-innoculation-history-19th-century-virus-squads.html.

Lübeck 1930 BCG Disaster

Donald, Peter R., et al. “Pathogenesis of Tuberculosis: The 1930 Lübeck Disaster Revisited.” European Respiratory Review, vol. 31, no. 164, June 28, 2022, article 220046.

Nakayama, Don K. “A Novel Microbe, Immunization Deaths, and Vaccination on Trial: BCG and the Lübeck Disaster of 1930.” SAGE Open Nursing, vol. 11, 2025, article 23779608251313994.

“Lübeck Disaster.” Wikipedia, September 27, 2025, en.wikipedia.org/wiki/Lübeck_disaster.

1916 Polio Outbreak

Wyatt, H.V. “The 1916 Poliomyelitis Epidemic and the Rockefeller Institute.” History and Philosophy of the Life Sciences, vol. 33, no. 1, 2011, pp. 95–110.

Polio Vaccination and SV-40

Cutrone, Rochelle, et al. “Some Oral Poliovirus Vaccines Were Contaminated with Infectious SV40 After 1961.” Cancer Research, vol. 65, no. 22, November 15, 2005, pp. 10273–10279.

Carbone, Michele, et al. “Simian Virus 40 Transformation, Malignant Mesothelioma and Brain Tumors.” Expert Review of Respiratory Medicine, vol. 5, October 2011, pp. 683–697.

Kops, Stanley. “Re: Debate on the Link Between SV40 and Human Cancer Continues.” Journal of the National Cancer Institute, vol. 94, no. 3, February 6, 2002, pp. 229–230.

Cutter Incident 1955

“Historical Vaccine-Associated Incidents.” History of Vaccines, historyofvaccines.org/blog/historical-vaccine-associated-incidents.

Richet and Anaphylaxis

Richet, Charles. Anaphylaxis. University Press, 1913. (Nobel Prize in Physiology or Medicine, 1913.)

Current Vaccine Excipients

Centers for Disease Control and Prevention. “Vaccine Excipient Summary — Appendix B.” CDC Pink Book, cdc.gov/pinkbook/hcp/table-of-contents/appendix-b-vaccines.html.

Food and Drug Administration. Package Inserts for Currently Licensed US Vaccines. FDA, fda.gov.

Gatti and Montanari Research

Gatti, Antonietta M., and Stefania Montanari. “New Quality-Control Investigations on Vaccines: Micro- and Nanocontamination.” International Journal of Vaccines and Vaccination, vol. 4, no. 1, 2017, pp. 00072.

Historical Analysis and Background

Humphries, Suzanne, and Roman Bystrianyk. Dissolving Illusions: Disease, Vaccines, and the Forgotten History. Create Space Independent Publishing Platform, 2013.

Willrich, Michael. Pox: An American History. Penguin Press, 2011.

The Hermetic-Alchemical Tradition and Modern Medicine

Hoffman, Michael. Secret Societies and Psychological Warfare. Independent History and Research, 2018.

Yates, Frances A. Giordano Bruno and the Hermetic Tradition. University of Chicago Press, 1964.

Yates, Frances A. The Rosicrucian Enlightenment. Routledge and Kegan Paul, 1972.

Yates, Frances A. The Occult Philosophy in the Elizabethan Age. Routledge and Kegan Paul, 1979.

Scholem, Gershom. On the Kabbalah and its Symbolism. Translated by Ralph Manheim, Schocken Books, 1965.

Idel, Moshe. Golem: Jewish Magical and Mystical Traditions on the Artificial Anthropoid. State University of New York Press, 1990.

Brown, E. Richard. Rockefeller Medicine Men: Medicine and Capitalism in America. University of California Press, 1979.

Robbins, Alexandra. Secrets of the Tomb: Skull and Bones, the Ivy League, and the Hidden Paths of Power. Little, Brown and Company, 2002.

July 13, 2026 Posted by | Book Review, Science and Pseudo-Science, Timeless or most popular | Comments Off on The Animal Substance

Doing The Opposite: Studies Show Gigantic Wind Farms Significantly Warm The Night

By P Gosselin | No Tricks Zone | July 5, 2026

Germany’s online Report24 has an article titled: “Studies Show Gigantic Wind Farms Significantly Warm the Night”.

Proponents of the energy transition often ignore or conceal the negative local climate impacts of wind turbines. Report24 references a 2012 study published in Nature Climate Change by Liming Zhou and his research team, which investigated the impact of large wind farms on land surface temperatures in Texas.

Researchers analyzed satellite data from 2003 to 2011 covering an area in Texas that hosts four of the world’s largest wind farms. In the areas with wind farms, nighttime surface temperatures in summer increased by up to 0.65 °C more than in comparable areas without turbines. The calculated warming trend was up to 0.72 °C per decade.

Why do turbines cause warming? At night, the ground cools down, making the air near the surface colder than the layers above. The turbine rotors disrupt this natural stratification, mixing the layers and forcing warmer air down to the surface, which warms and dries out the ground. Germany is plastered with circa 30,000 turbines spread across the country. In addition to the growing urban heat island (UHI) effect, Germany’s local climate is being disrupted by its widespeard use on wind turbines.

Politicians and mainstream media have been deliberately ignoring these facts since 2012. The local warming caused by wind farms is falsely blamed on CO₂-driven climate change in order to maintain the narrative of “saving the climate.”

Also, deforestation is underway in Germany in order to clear the way for largescale windparks, severely damaging a natural ecosystem that acts to cool the local climate.

July 5, 2026 Posted by | Science and Pseudo-Science | Comments Off on Doing The Opposite: Studies Show Gigantic Wind Farms Significantly Warm The Night

No, Futurism, One Momentary Hot Spot on Antarctica Doesn’t Prove a Climate Crisis

By Anthony Watts | ClimateRealism | June 23, 2026

A recent article in the online journal Futurism, titled, “Scientists Horrified as Huge Heatwave Hits Antarctica,” claims climate change caused a “huge heatwave” in Antarctica, bringing temperatures on the Antarctic Peninsula nearly 36°F above average and briefly pushing readings above freezing. This is highly misleading. A single weather event says nothing meaningful about long-term climate trends, and the article ignores both Antarctica’s enormous geographic variability and the exceptionally cold conditions simultaneously occurring elsewhere on the continent. The heatwave Futurism suggested wasn’t a continent-wide crisis, but was a localized, unalarming event.

The article is largely a rewrite of a Guardian story focused on temperatures measured on the Trinity Peninsula, the northernmost extension of Antarctica. Researchers reported temperatures reaching approximately 15.4°C (59.7°F) during a brief warm spell on June 6.

What readers are not told is that the Antarctic Peninsula is not representative of Antarctica as a whole.

In fact, the warmest part of Antarctica is the Antarctic Peninsula. Nicknamed the “banana belt,” it stretches northward toward South America and experiences milder maritime conditions. During the austral summer, temperatures can occasionally exceed 10°C (50°F). The peninsula extends northward toward South America and is heavily influenced by maritime weather patterns and ocean currents. It is by far the warmest part of Antarctica and has long experienced periodic warm-air intrusions, föhn wind events, rain episodes, and above-freezing temperatures. These events are unusual, but they are not unprecedented.

In fact, the article itself acknowledges that the warmth was associated with “extremely strong westerlies.” In other words, this was a weather event driven by warmer atmospheric circulation patterns, not a direct measurement of climate change.

What Futurism misses is the fact that weather is not climate.

Climate is measured over decades. A single day, a single week, or even a single season tells us very little about long-term temperature trends. If every unusually warm day is presented as proof of climate catastrophe, then intellectual consistency would require every unusually cold day to be presented as evidence against it. The media rarely applies that standard.

The timing of this story is especially revealing because while headlines were breathlessly reporting a temporary warm spell on the Antarctic Peninsula, much of the rest of Antarctica was experiencing brutally cold conditions.

According to observations highlighted by meteorologist Cap Allon, the Amundsen-Scott South Pole Station recorded a temperature of -73.6°C (-100.5°F) on June 16, with a daily maximum of only -69.9°C (-93.8°F). That was the South Pole’s first sub–70°C reading since 2023.

One part of Antarctica briefly experiences an unusual warm episode, while another part of the continent drops below -100°F. That is how weather works on a continent larger than the United States and Mexico combined.

Yet only one of those events, specifically the anomalous high temperature one, generated international headlines.

The article further claims that the heatwave follows “decades of increasingly warm temperatures observed on the white continent.” That statement is false.

Antarctica is not warming uniformly. While portions of the Antarctic Peninsula experienced warming during parts of the late twentieth century, numerous studies have shown little warming or even cooling across large sections of East Antarctica, with East Antarctica making up the bulk of the continent. Antarctic sea ice has also exhibited substantial variability from year to year and decade to decade.

The continent is governed by complex interactions involving ocean currents, atmospheric circulation, volcanic influences (including subsurface heating under West Antarctica where the peninsula lies), sea ice dynamics, and natural climate oscillations. That complexity disappears in Futurism’s opinionated article.

Instead, readers are given the now-familiar formula: identify a dramatic weather event, attach it to climate change, mention the “Doomsday Glacier,” and imply catastrophe is around the corner. Climate Realism has debunked claims of the Thwaites Glacier’s imminent collapse repeatedly, previously. The article’s reference to Thwaites Glacier is a particularly misleading red herring, because it has nothing to do with the reported weather event. The mention serves one purpose: reinforcing a broader climate crisis narrative.

This is increasingly common in climate reporting. Any unusual weather event becomes an opportunity to recycle the same talking points about glaciers, sea level rise, tipping points, and future disasters, regardless of whether they are directly related to the event being discussed.

Concerning the Antarctic Peninsula, the facts are these: Antarctica has always been susceptible to periodic warm-air intrusions because of its geography and proximity to relatively warmer ocean waters; the Southern Ocean, atmospheric rivers, and strong westerly winds can occasionally transport substantial heat into the region. These processes existed long before climate change became a political issue.

Most importantly, a single warm event cannot establish a trend. Scientists understand this principle when analyzing climate data. Journalists should understand it as well.

A proper climate analysis requires decades of observations across the continent, careful examination of regional variability, and separation of weather noise from climate signals, not the slap dash presentation of misleading, sensational claims assembled by Futurism.

Antarctica remains the coldest continent on Earth. While the Antarctic Peninsula briefly experienced unusually mild conditions, the South Pole itself was simultaneously plunging near or below -100°F in multiple other locations.

That fact alone should remind readers that one weather event, no matter how dramatic the headline, is not evidence of a “climate emergency,” it’s simply weather. One warm spell on the Antarctic Peninsula becomes proof of climate catastrophe, while simultaneous temperatures below -100°F at the South Pole are ignored. That’s not objective journalism, that’s agenda-driven alarmism.

July 5, 2026 Posted by | Deception, Science and Pseudo-Science | Comments Off on No, Futurism, One Momentary Hot Spot on Antarctica Doesn’t Prove a Climate Crisis

Survivorship Bias: The Logical Error at the Heart of Modern Medicine

An Essay on Why You Only Hear From the People Who Lived

Lies are Unbekoming | June 18, 2026

The bullet holes show where a plane could be hit and survive. The places without holes are where the lost planes were hit.
Illustration: Martin Grandjean, McGeddon, and Cameron Moll. Wikimedia Commons, CC BY-SA 4.0. Source: File:Survivorship-bias.svg

Wald at the Statistical Research Group

In 1943, the U.S. military was about to armor its bombers in exactly the wrong places. The analysts had examined every bomber that came back from combat, mapped the bullet holes across the airframe, and proposed reinforcing the spots where the holes clustered. The data was right there. You could see it on the planes.

They referred the question to the Statistical Research Group at Columbia University, a small team of mathematicians assembled for the war effort. The Hungarian-born statistician Abraham Wald examined the bullet-hole maps and gave the opposite recommendation. Armor the engines and the cockpit. The places where the returning planes had no bullet holes.

The military analysts had committed a logical error so simple they could not see it. They were studying the planes that came back. The planes shot through the engines and the cockpit did not come back. They had gone down across two oceans and the territory between them. The returning planes did not reveal where bombers were vulnerable. They revealed where bombers could be hit and still fly home.

The bullet holes on the survivors mapped survivable damage, not dangerous damage. Armoring where the holes were meant armoring the places that did not need armoring. The damage that mattered was on the planes you could not examine because they were destroyed.

Wald’s memorandum was classified. Decades later, when his work was declassified and republished, the principle he had identified came to be called survivorship bias.¹ ² It is the most pervasive and least understood logical error in any field that draws conclusions from a visible population of survivors.

The same error sits at the center of the modern screening-and-treatment industry.

The Error Generalized

Survivorship bias operates wherever a process selects what gets seen. The destroyed do not file reports, and the audience reads only what made it through.

Mutual fund performance averages exclude funds that closed. Hedge fund return statistics quietly drop the funds that liquidated. The historical returns of “the market” routinely omit bankruptcies, delistings, and total losses. Funds that lived publish their numbers; funds that died publish nothing. The retail investor reads the winners.

The literature on entrepreneurship was built the same way. CEOs who succeeded wrote memoirs about their habits and their early-morning routines. The thousands of equally disciplined founders who failed wrote nothing because their companies went under. The “success habits” identified by reading the survivors are, in many cases, just habits, shared by the dead and the living alike, with no causal relationship to outcomes.

In architecture, the buildings that survive are studied for their construction methods. The buildings that collapsed in storms, earthquakes, or fires are no longer there to be examined. Old buildings appear well-built because the badly-built ones are gone.

In each case, the visible sample is selected by the very property you are trying to measure. You cannot learn about plane vulnerability from intact planes. You cannot learn about cancer survival from cancer survivors.

Survivorship Bias in Medicine

A woman undergoes mammography in her early fifties. The scan finds a small lesion. She receives a biopsy, a lumpectomy, six weeks of radiation, and five years of tamoxifen. She is alive ten years later. She becomes an advocate, walking in Race for the Cure and telling her sister, her daughter, and the women in her neighborhood that screening saved her life. Get the test.

Her experience is real, her gratitude genuine. The conclusion she draws does not follow from either.

What she does not know, what she cannot know, is what would have happened to her without the screening, the biopsy, the surgery, the radiation, and the years of medication. She cannot run the counterfactual on herself. The version of her that did not get the treatment does not exist, and she has no way to consult it.

The institutions that promoted her screening have access to data she does not. Population-level data, accumulated across decades of randomized and observational studies, is consistent with four overlapping forms of survivorship bias, each of which inflates the apparent success of the system. Together they are sufficient to explain most of what the industry presents as the triumph of “early detection.”

Survivorship Bias Proper

The most direct form: only the living testify.

Eight months after her mastectomy, a woman dies of chemotherapy-induced sepsis. She does not appear at the October fundraiser. Six weeks after his prostatectomy, a man dies of cardiac complications. He writes no op-eds about prostate health. The radiation that “cured” the first cancer induces a second one five years later, and the patient’s family attends a funeral, not a marathon. The chemotherapy regimen that ostensibly drove the tumor into remission also drove the patient’s bone marrow into failure, and she dies of sepsis a year later, recorded by quiet bureaucratic convention as a “cancer death.”

When the public hears about cancer treatment, it hears from the patients who survived. Those who did not survive are statistically invisible. They are counted in mortality columns nobody reads, while their grateful surviving counterparts address the television cameras. The audience for screening campaigns sees a heavily filtered population, filtered by the treatments themselves.

The filtering goes further than visibility. It reaches into the mortality data itself. When a patient on chemotherapy dies of cardiotoxic heart failure, the death is typically coded as a cancer death. When a patient with treatment-induced bone marrow failure dies of sepsis, it is coded as a cancer death. When the surgical complication kills the patient on the operating table, it is generally coded as a cancer death. When the second cancer induced by radiation given for the first cancer kills the patient ten years later, the second cancer is frequently recorded as primary, the radiation that caused it noted in passing if at all. The coding conventions tilt systematically in one direction: failures of treatment are folded back into the column labeled “disease.” The treatment is shielded from blame. The cancer absorbs it. The mortality statistics that institutions cite to justify aggressive treatment are themselves an artifact of how treatment failures are recorded.

Lead-Time Bias

Finding a cancer earlier does not mean treating it earlier extends life. It means knowing about it longer.

Two women with identical lesions, identical biology, and identical eventual outcomes. Both die at age seventy. Woman A is screened at fifty, her cancer is detected, and she is “treated” for the next twenty years. Woman B is unscreened. She develops symptoms at sixty-five, is diagnosed, and dies at seventy.

The standard reporting metric is five-year survival from diagnosis. By that measure, Woman A counts at 100% survival. Woman B counts at 0%. The treatment looks miraculous. Nothing has actually changed. Both women died at seventy. Woman A simply spent twenty years as a patient.

The five-year survival statistic is the standard currency of cancer reporting. In the presence of widespread screening, it is also a metric that can rise to 100% without saving a single life. Between 1950 and 1995, the five-year survival rate for prostate cancer in the United States rose from 43% to 93%. The age-adjusted mortality rate from prostate cancer over the same period barely moved.³ The screened population learned about their cancer earlier. They did not die later.

When you read that “early detection” has improved five-year survival rates for breast, prostate, or thyroid cancer, you are reading a statistic structurally biased toward the appearance of benefit even when no benefit exists.³ ⁴

Length-Time Bias

Screening preferentially detects slow-growing lesions. Aggressive cancers grow rapidly between screening intervals and present symptomatically, not through the scan. Indolent lesions sit for years, available to be detected at the next mammogram or PSA test.

The population of cancers caught by screening is therefore enriched for slow biology, for lesions that were less likely to kill in the first place. Patients with these lesions tend to do well, not because the screening saved them but because their cancers were not going to kill them on any rapid timescale. The aggressive cancers, the ones that genuinely threaten life, frequently arise and progress in the gaps between scans.

Screening catches the cancers least in need of catching. The system then takes credit for the favorable outcomes of patients who would have done well regardless.

Overdiagnosis

The fourth and most powerful form. Many of the lesions detected by screening are not, in any meaningful sense, going to harm the patient. They are stable, non-progressive anatomical findings that medicine has chosen to label as cancer.

Bleyer and Welch, examining three decades of U.S. mammography data, estimated that 31% of breast cancers detected by screening represented overdiagnosis: disease that would never have produced symptoms or shortened life.⁵ The Cochrane systematic review of mammography trials concluded that for every life potentially saved by screening, ten women receive treatment for a cancer that would not have harmed them. The same review found no reduction in all-cause mortality from screening.⁶

In South Korea, the introduction of widespread thyroid ultrasound produced a fifteen-fold increase in thyroid cancer diagnoses over two decades. Mortality from thyroid cancer did not change. The country had not experienced a thyroid cancer epidemic. It had begun finding microscopic lesions that had always existed in the population, at autopsy in people who died of other causes, and that had never killed anyone before they were found and treated.⁷

Autopsy studies of men who died of unrelated causes have found prostate cancer cells in roughly a third of those in their forties, rising to two-thirds by their late sixties.¹⁵ The lifetime mortality from prostate cancer is approximately 3%. Most older men carry the disease into a natural death from something else; they die with prostate cancer, not from it. The PSA test cannot distinguish between the cancer that would have killed and the cancer that would have been silently carried into the grave. It detects both. It produces a diagnosis in both. The men with non-threatening lesions, who vastly outnumber the rest, are subjected to surgery, radiation, and hormonal therapy for a condition that was never going to harm them. They survive what was never threatening. They credit the system. They tell other men to get tested.

The ERSPC trial, the largest prostate cancer screening trial ever conducted, found that PSA screening reduced prostate cancer mortality by a small absolute amount over thirteen years. To prevent one death, approximately twenty-seven men had to be diagnosed and treated, most of whom would not have died from their disease and many of whom were rendered incontinent, impotent, or both by the intervention.⁸

Every overdiagnosed patient is, by definition, a successful “treatment outcome.” She survived a treatment for a condition that was never going to harm her. The system takes credit. She testifies on its behalf.

Why the System Selects for Evangelism

The four biases would matter less if the visible patients were a representative sample. They are not. The system that produces them also amplifies them.

Hospitals run survivor outreach programs. Pharmaceutical companies fund patient advocacy organizations. The pink ribbon ecology, Susan G. Komen, the National Breast Cancer Foundation, the dozens of subsidiary charities, operates almost entirely on survivor testimony. October fills American mailboxes with pink-ribboned testimonials. The American Cancer Society’s national publicity is built on survivor stories. The patient who survived is the asset.

The corporate machinery built around the survivor is substantial. Estée Lauder co-founded the pink ribbon symbol with Self magazine in 1992 and now sells pink-ribbon cosmetics each October. The National Football League runs an annual “Crucial Catch” campaign with players wearing pink cleats and accessories. Major League Baseball stages pink-bat games on Mother’s Day. Yoplait produced pink-lidded yogurt for two decades under the “Save Lids to Save Lives” campaign. Ford sold pink-ribbon merchandise through “Warriors in Pink.” General Mills, KitchenAid, the National Hockey League, the airlines, the cosmetics counters at every major department store all participate. The advertising spend on these campaigns runs into hundreds of millions of dollars annually, dwarfing the portion of the proceeds that ever reaches research and dwarfing many times over the portion of research funding that addresses environmental causes of breast cancer rather than treatment. The campaigns sell screening. The screening produces patients. Most patients survive, because most of what is found in screening was not going to kill them, and they testify. The testimony funds the next round of campaigns.

The patient who died is, from a marketing perspective, a problem. Her death cannot be celebrated. Her family is often grieving and angry. Her doctors generally do not call the local newspaper. She becomes a statistic in a column nobody reads, while the surviving patient in the next room becomes the face of the cause.

This selection is not a conspiracy. It is a structural feature of how the industry communicates. Living patients can be photographed; dead patients cannot. The living speak at events; the dead are credited, by quiet convention, to “the disease.”

The grateful survivor is also psychologically necessary for the treatment to continue being offered in its current form. The patient who has undergone radical mastectomy, six rounds of chemotherapy, weeks of radiation, and years of endocrine therapy must believe, on pain of intolerable cognitive dissonance, that this was necessary and life-saving. To accept that she may have been treated for a lesion that would not have harmed her, that she lost her breast, her hair, her fertility, her cardiac reserve, perhaps her marriage, to a system that misjudged the threat, is psychologically devastating. The mind protects itself. She becomes an advocate.

The advocate then promotes the system to other women, who undergo screening, get diagnosed, get treated, and become advocates in turn. Each cycle generates more survivors, each of whom credits the system that produced them. The dead and the harmed are silent by definition.

The financial scale of this ecosystem is not small. The United States spends roughly two hundred billion dollars annually on cancer-related medical care. Mammography alone is a multi-billion-dollar industry. The PSA test, despite repeated expert task force recommendations against routine screening, generates billions in downstream procedures. The pink ribbon charities raise hundreds of millions per year, much of which goes to “awareness,” that is, to producing more screening, more diagnoses, more treatment, and more survivors. The asset class, the patient, is manufactured by the process that then takes credit for her survival.

In 2018, a Goldman Sachs equity research report posed the question explicitly to its biotechnology clients: “Is curing patients a sustainable business model?”⁹ The analysts noted that one-time cures undermine recurring revenue streams. The pharmaceutical industry’s most profitable customers are chronic patients, not cured ones. The screening-and-treatment cancer model is, from a financial perspective, an excellent business. It produces patients. It treats them for years. Many of them survive, which is what the model needs them to do, because survivors testify and dead patients do not.

Inside the Testimonial

The cancer survivor has four pieces of information. She was screened. Something was found. She received treatment. She is alive years later. From these facts, she draws a single conclusion: the screening and treatment saved her life.

The inference is intuitive but unsupported. The same four facts admit at least three other explanations.

The screening detected a lesion that would never have harmed her. She survived not because of the treatment but in spite of it. The treatment did damage that she absorbed because the rest of her body was healthy enough to recover.

The lesion was real but slow-growing. She would have lived equally long, with less suffering, by doing nothing.

The lesion was real and biologically active, but her body’s repair mechanisms, what the establishment calls her constitution and what terrain medicine recognizes as her terrain, would have managed it. The treatment was incidental to her survival.

She cannot distinguish between these explanations from her own experience. None of them is available to her introspectively. The only way to determine which is correct, at the level of a population, is the randomized controlled trial, the kind of trial that, in most screening contexts, has either not been done with adequate follow-up or has produced equivocal results that the institutions promoting screening do not publicize.

Her testimonial is sincere. It is also, with respect to the question of whether the treatment worked, evidence of nothing in particular. The dead woman two beds down the hall, who received identical screening and identical treatment and died of cardiotoxic chemotherapy, would have a different testimonial if she could give one. The system that asks the survivor to speak does not ask the dead woman’s family to speak. The asymmetry produces the appearance of a treatment success rate the underlying data does not support.

What Survives the Error

Once you see survivorship bias, you cannot unsee it. What remains is not paranoia but a discipline: asking, in every medical context, which population you are looking at and which population is missing.

When a screening campaign reports that “five-year survival rates have improved,” ask whether overall mortality has changed. Five-year survival can rise to 100% without saving a single life if all the increase comes from earlier detection of lesions that were going to be survived anyway. Overall mortality, deaths per hundred thousand population per year, is much harder to manipulate. It is also the only figure that answers the question the survival rate appears to answer.

A cancer survivor telling you her treatment saved her life can be sincere and still wrong about causation. The conviction is real. The causal claim it carries is not derived from anything she has direct access to. You can be glad she is alive without accepting her account of why.

An oncology center’s published survivor outcomes report a filtered population. Ask about the patients who did not complete treatment. Ask about the patients who died of treatment-related complications and were classified as cancer deaths. Ask about the patients whose follow-up was lost because they moved into hospice care or stopped responding to calls. The shape of the population that gets reported is the shape of the population that survived long enough to be counted.

Facing a screening recommendation yourself, the question to ask is the one the military analysts in 1943 did not ask: what does this examination fail to show me? The bullet holes on the surviving bombers concealed the bullet holes on the destroyed ones. The success stories of the screening industry conceal the women treated for lesions they did not have, the men rendered impotent by surgery for cancers that would never have grown, the second cancers induced by the radiation, and the patients whose treatments killed them and who are now counted, by quiet bureaucratic convention, as having died from their disease.

The full investigative case on these screenings, what they detect, what they miss, what they manufacture, and what they cost, has been documented at length in earlier work.¹⁰ ¹¹ ¹² ¹³ ¹⁴

What the Trials Actually Show

Defenders of mammography routinely cite two trials. The Health Insurance Plan of New York trial, begun in 1963, and the Swedish Two-County Trial, conducted between 1977 and 1985, both reported reductions in breast cancer mortality among screened women. Both have been criticized on methodological grounds. The HIP trial’s randomization was uneven, with baseline differences between arms and exclusion rules applied asymmetrically. The Two-County Trial used cluster randomization that did not consistently balance comparison groups and lacked blinded cause-of-death assessment. Its mortality estimates shifted across successive reanalyses. The Cochrane systematic review of mammography trials classified both as carrying significant risk of bias.⁶

The most rigorously conducted breast cancer screening trial, the Canadian National Breast Screening Study, followed nearly ninety thousand women for twenty-five years. It found no reduction in breast cancer mortality from mammography screening, and no reduction in all-cause mortality.¹⁶

All-cause mortality is the figure that resists the gaming. When a study reports that breast cancer deaths fell among screened women but all-cause deaths did not, the women who avoided a death coded as breast cancer died of something else within the same window: heart failure from chemotherapy-induced cardiotoxicity, second cancers induced by the radiation given for the first, complications from the surgery, strokes after years of endocrine therapy. The Cochrane review and meta-analyses across multiple cancers have repeatedly found that all-cause mortality is essentially identical in screened and unscreened populations. The treatment that prevents one death produces another. The cancer-specific column improves; the death column does not. Survivorship bias gives the illusion of a saved life. All-cause mortality data shows that the life, where treatment did anything at all, was traded rather than added.

The Bombers and the Patients

Abraham Wald died in 1950 in a plane crash in the Nilgiri mountains of southern India. The principle he identified outlived him by three-quarters of a century and now sits, unrecognized by the institutions that depend on it, at the center of the modern medical industry.

The bombers that came back showed where a bomber could be shot and still come back. They did not show where a bomber was vulnerable. The cancer survivors who give interviews, walk in fundraisers, and tell their friends to get screened show where the modern oncology machine is not lethal. They do not show where it works.

The patients who died of their treatments are not at the marathon. The patients treated for lesions that would never have killed them have no way to know they were never in danger. The patients whose cancers were going to be survived regardless have no way to credit their own bodies rather than the chemicals introduced into them. They are the bullet holes on the wings: survivable damage, mapped and celebrated, while the damage that mattered remains invisible because the people who suffered it are no longer in the room.

Wald told the military to armor the engines and the cockpit, the places the surviving planes were not hit. The same instruction applies to medicine: look at what the survivors do not show you. The damage that matters is on the planes that did not come back, and on the patients who can no longer testify.


How to Explain This to a Six-Year-Old

Imagine all your friends drink a magic potion that’s supposed to keep them safe. Half of them disappear. The other half come back and tell you the potion worked great.

If you only listen to the friends who came back, you’ll be sure the potion is wonderful. You’ll tell other kids to drink it too.

But the other half are gone. They can’t tell you whether the potion hurt them. You don’t know whether the potion saved the friends who came back, or whether those friends would have been fine without ever drinking it.

When a cancer survivor tells you the treatment saved her life, listen to her kindly. But remember the friends who disappeared. They are part of the story too.

Quick Reference: The Four Biases

Survivorship bias. Only the living testify. Cancer patients who died of their treatments do not give interviews. The visible patient population is filtered by the treatments themselves.

Lead-time bias. Finding a cancer earlier does not extend life. It extends the time you know about it. Five-year survival rates can rise to 100% without saving a single life.

Length-time bias. Screening preferentially catches slow-growing lesions that were less likely to kill in the first place. Aggressive cancers arise and progress between scans, often undetected until they present symptomatically.

Overdiagnosis. Many screen-detected lesions would never have caused harm. The patient is treated for something that was not a threat, survives easily, and credits the treatment.

References

  1. Mangel, M., & Samaniego, F. J. (1984). Abraham Wald’s work on aircraft survivability. Journal of the American Statistical Association, 79(386), 259–267.
  2. Wald, A. (1943). A Method of Estimating Plane Vulnerability Based on Damage of Survivors. Statistical Research Group, Columbia University. (Declassified and republished by the Center for Naval Analyses, 1980.)
  3. Welch, H. G., Schwartz, L. M., & Woloshin, S. (2000). Are increasing 5-year survival rates evidence of success against cancer? JAMA, 283(22), 2975–2978.
  4. Welch, H. G., & Black, W. C. (2010). Overdiagnosis in cancer. Journal of the National Cancer Institute, 102(9), 605–613.
  5. Bleyer, A., & Welch, H. G. (2012). Effect of three decades of screening mammography on breast-cancer incidence. New England Journal of Medicine, 367(21), 1998–2005.
  6. Gøtzsche, P. C., & Jørgensen, K. J. (2013). Screening for breast cancer with mammography. Cochrane Database of Systematic Reviews, Issue 6, CD001877.
  7. Vaccarella, S., Franceschi, S., Bray, F., Wild, C. P., Plummer, M., & Dal Maso, L. (2016). Worldwide thyroid-cancer epidemic? The increasing impact of overdiagnosis. New England Journal of Medicine, 375(7), 614–617.
  8. Schröder, F. H., et al. (2014). Screening and prostate-cancer mortality in a randomized European study: results of the ERSPC at 13 years of follow-up. The Lancet, 384(9959), 2027–2035.
  9. Richter, S., et al. (2018). The Genome Revolution (biotechnology equity research report). Goldman Sachs Global Investment Research, April 10, 2018.
  10. Unbekoming. The 12 Screenings That Manufacture the Patients They Claim to Find.
  11. Unbekoming. The Screening Trap.
  12. Unbekoming. Breast Cancer: What They Didn’t Tell You.
  13. Unbekoming. The PSA Trap: How a Flawed Test Built a Billion-Dollar Industry and Destroyed Millions of Men.
  14. Unbekoming. The Unbekoming Cancer Compendium.
  15. Sakr, W. A., Grignon, D. J., Crissman, J. D., Heilbrun, L. K., Cassin, B. J., Pontes, J. J., & Haas, G. P. (1994). High grade prostatic intraepithelial neoplasia (HGPIN) and prostatic adenocarcinoma between the ages of 20-69: an autopsy study of 249 cases. In Vivo, 8(3), 439–443.
  16. Miller, A. B., Wall, C., Baines, C. J., Sun, P., To, T., & Narod, S. A. (2014). Twenty five year follow-up for breast cancer incidence and mortality of the Canadian National Breast Screening Study: randomised screening trial. BMJ, 348, g366.

July 5, 2026 Posted by | Science and Pseudo-Science, Timeless or most popular | , | Comments Off on Survivorship Bias: The Logical Error at the Heart of Modern Medicine

‘The Medical-Pharmaceutical Killing Machine: Facing Facts Could Save Your Life’

Children’s Health Defense Team | December 11, 2024

This is a reprint of Chapter 1 in “The Medical-Pharmaceutical Killing Machine: Facing Facts Could Save Your Life,” by Children’s Health Defense.

The book, also available on Amazon documents “systemized medical abuse” that accelerated during the COVID-19 pandemic.

Chapter 1. From Quackery To Criminality

The medicinal use of mercury offers a long-running example of medically induced harm. Although centuries of whistleblowers have warned that dosing patients with it constitutes reckless quackery—and the U.S. government presently places mercury at number three on its “Substance Priority List,” right under arsenic and lead—the heavy metal has figured prominently in the “medical armamentarium” from as far back as the sixth century BC through the present day.

In his important book Evidence of Harm, author David Kirby exposed the pharmaceutical industry’s controversial practice of including mercury preservatives in vaccines. Pointedly using the word “criminal,” Kirby wrote in the foreword to another book about mercury (The Age of Autism by Dan Olmsted and Mark Blaxill) that the “blind belief in a known poison” has been “misguided, immoral, and in some cases, patently criminal.”

The “Messianic” Benjamin Rush

In many ways, the medical practices and beliefs of U.S. Founding Father, physician, and University of Pennsylvania medical school professor Benjamin Rush may have set the stage for modern medicine’s stubborn adherence to dangerous protocols—despite clinical evidence of harm—and its silver-bullet fascination with vaccines “as substitutes for right living,” as Eleanor McBean put it in her 1957 book The Poisoned Needle: Suppressed Facts About Vaccination.

The reportedly “messianic” and “uncompromising” Rush’s late-1700s stock-in-trade was a radical protocol involving bloodletting and purging with—what else?—mercury, a practice that medical historians later dubbed “heroic medicine.” Rush had his own proprietary brand of laxative called “Thunderclappers,” consisting of approximately 60% mercury chloride (also called calomel), which he promoted as “a purgative of explosive power.” As Rush honed his clinical methods, he passed them on to a phalanx of enthusiastic students and disciples during yellow fever epidemics in Philadelphia, where he would bleed and purge up to 100 patients a day. Although use of calomel was not uncommon among doctors of that era, Rush prescribed up to 10 times more than his medical peers and also recommended the removal of huge amounts of patients’ blood, erroneously believing that the blood would replenish itself in a matter of a day or two. “A patient’s failure to respond to this disastrous therapy,” one historian wrote in 2004, “won [the patient] only another round of bleeding and purging.” In another modern writer’s colorful description, “So much blood was spilled in the front yard that the site became malodorous and buzzed with flies.”

No less a figure than George Washington underwent a rapid and gruesome death after Rush protégé Dr. Elisha Dick (and two other Johnny on-the-spot physicians) poisoned Washington with mercury and removed 40% of the beleaguered general’s total blood volume—a quantity that, to this day, “continues to amaze and appall laymen and physicians alike.” From many historians’ point of view, Washington’s doctors caused his death, a death that may well have changed the course of history.

Rush was enthusiastic about promoting his “heroic medicine” protocol, “proclaim[ing] the success of his cure to the public and his medical colleagues” in newspapers, advertisements, and brochures, and even “harangu[ing] people in the streets.” In addition, he was an early and explicit proponent of smallpox vaccination. In 1803, he joined with 30 other Philadelphia doctors in signing a public notice “expressing their confidence in vaccination and recommending it for general use.” Significantly, smallpox vaccination represented a turning point in the “medicalization of the general public” in both early nineteenth-century America and Europe, and a boon for the burgeoning medical profession:

Since the late eighteenth century, doctors had intensified their efforts to win government support for their plans to bring the whole population under medical control. . . . Thus Jenner’s method of cowpox vaccination presented medical practitioners with a new chance to increase their prestige and influence on public health affairs [bold added]. Doctors also foresaw an increase in their income through vaccination fees and hoped to establish themselves, with the help of the vaccine, among those classes of the population who had not consulted doctors before.

From 1813 to 1822, the young U.S. government appointed James Smith as the nation’s “federal vaccine agent,” charging him with “maintaining a supply of the smallpox vaccine and distributing it nationwide”; Smith had been a student of Rush’s at the University of Pennsylvania and was a fellow member of the “well-educated medical elite.” Although other physicians of the day argued that smallpox vaccination was both dangerous and ineffective, then—as now—defenders of the practice prevailed by using “more or less perverted statistics,” with one doctor urging his “professional brethren to be slow to publish fatal cases of small-pox after vaccination” and others passing off vaccine-induced fatalities as some other disease.

Reflecting on Rush’s medical legacy, U.S. Army medical officer P.M. Ashburn made remarks in 1929 that highlight one of the many reasons why Rush’s cautionary tale is still pertinent today. Ashburn wrote that by virtue of Rush’s “social and professional prominence, his position as teacher and his facile pen,” the Philadelphia physician “was more potent in propagation and long perpetuation of medical errors than any man of his day,” thereby “blacken[ing] the record of medicine.” This observation illustrates how social prestige—coupled with “unyielding devotion to dogma”—often helps practitioners of dangerous medicine beat back their critics.

In Rush’s time, those critics included fellow physician Elisha Barlett, who opined about Rush’s medical theories, “In the whole vast compass of medical literature, there cannot be found an equal number of pages containing a greater amount of utter nonsense and unqualified absurdities,” as well as feisty British journalist and pamphleteer William Cobbett, who dared to publish tracts asserting that Rush’s yellow fever treatments were both ineffective and dangerous—and “a perversion of nature’s healing powers.” In response, Rush sued Cobbett for libel and won, in “one of the largest libel awards in American history at the time.”

One of Cobbett’s fascinating observations—which reverberates uncannily in the COVID era—was that extreme fear (in this instance, of yellow fever) made members of the public far more willing to subject themselves to Rush’s “experiments” than they otherwise might have been. Cobbett wrote:

[Rush] seized, with uncommon alacrity and address, the occasion presented by the Yellow Fever, the fearful ravages of which were peculiarly calculated to dispose the minds of the panick-struck people to the tolerance, and even to the admiration, of experiments, which, at any other time, they would have rejected with disdain.

Interestingly, after Rush’s libel victory, Cobbett exacted a modicum of revenge by assembling data from municipal records (acknowledged today as “an epidemiological tour de force”), which pointed to a 56% mortality rate among Rush’s yellow fever patients that contrasted starkly with the physician’s own claim of a greater than 90% survival rate. When word of those dismal statistics got out to the public, Rush’s medical practice suffered. Undaunted, Rush went on to become Treasurer of the U.S. Mint under President John Adams. As the author of America’s first psychiatric textbook, he is also revered today as “the father of American psychiatry.” Rush proposed the same general treatments for madness that he favored for physical ailments, supplemented by straitjackets and other “modes of punishments” for tough cases.

For his part, in 1800, a disgusted Cobbett returned to London, where he continued to hold medicine’s feet to the fire, including condemning smallpox vaccination as “quackery.”

A “Patently Criminal” Model

Some modern medical historians are willing to go so far as to characterize medicine, in periods and places like 18th-century America, as “deplorable,” and to suggest that back then, “a doctor was just as likely to kill you as save you.” Most, however, frame medical barbarity as a thing of the past. Shielded by high-end machines, complex drug technologies, glossy scientific publications, and lingo like “rigorous” and “evidence-based,” the current medical-pharmaceutical-regulatory establishment and its hagiographers would have the public believe that “safe and effective” now rules the day.

There is ample evidence to show that pledges of safety often are either disingenuous or false, and there are indications that Kirby’s description of the medical model as sometimes “patently criminal” was squarely on the mark. At the level of individual medical practitioners, law firms specialized in malpractice note that if a doctor “appears to be indifferent to patients’ well-being or safety,” that indifference can be grounds for criminal liability. A search of the word “criminal” on the website of Medpage Today (a conventional news service that is generally protective of medicine’s reputation) brings up countless articles about doctors and other health care providers running “pill mill” operations, carrying out fraud, taking kickbacks, tampering with drugs, faking data, sexually assaulting or abusing patients, and engaging in other types of “unprofessional” and unethical conduct. The site’s “Investigative Roundups” feature stories (often formulated as questions to soften the impact) with titles like “Columbia protected predator doc?”, “Psychiatrist held patients against their will?”, “$15K surgery shakedown?” or “Doc pushed unneeded surgery?” Other Medpage Today headlines flamboyantly bandy about words like “deadly,” “loophole,” “games,” “tactics,” “unethical,” and “secretive.”

Sometimes, individuals who defend the medical status quo blame whichever reports of misbehavior manage to surface (many do not) on “a few bad apples.” Others, such as Harvard scientist and patient safety advocate Lucian Leape, do the reverse, shifting the blame from “bad people” to nebulous “bad systems;” Leape suggests that a cycle of disrespect is “learned, tolerated, and reinforced in the hierarchical hospital culture.” The fact is, however, that medical harms flow from both individuals and institutions. Most health care providers operate in broader organizational and corporate contexts—and it is policymakers and decision-makers at those levels who often give medical-pharmaceutical corruption and criminality a green light. This is illustrated by the phenomenon (for which there is even an academic field of study) called “clinicide,” defined as serial medical killers responsible for “the unnatural death of multiple patients in the course of treatment;” not infrequently, the killers’ host institutions countenance or “enable” this clinicide by choosing to ignore red flags.

As an extension of the “bad apples” argument, some upholders of the status quo point to the fines that the U.S. Department of Justice (DOJ) routinely levies on hospitals and pharmaceutical corporations, suggesting that these are an adequate mechanism to catch and punish players engaged in malfeasance. However, given that medical-pharmaceutical culprits not infrequently are criminal recidivists and that the fines generally amount to “little more than a slap on the wrist,” it is fair to ask “whether such a monetary punitive system really does much to prevent bad behavior.”

Moreover, DOJ rarely prosecutes or holds corporate leaders accountable, despite having a “powerful legal tool” at its disposal to go after the executives at the helm of medical misconduct; it has done so only 13 times since the year 2000. Instead, many signs point to a wink-and-a-nod sub rosa understanding between the various parties, with the penalties doing nothing to prevent future harms but instead furnishing a generous flow of kickbacks that prosecutors and regulators can funnel into various sectors of the federal budget (see Illegal But Profitable). In fact, under the False Claims Act, the U.S. Department of Health and Human Services (HHS) gets a 20 to 1 return on every dollar it “invest[s] in prosecutions and investigations.”

Illegal but Profitable

In 2018, the nonprofit consumer advocacy organization Public Citizen published a report summarizing 27 years of pharmaceutical industry criminal and civil penalties. The report concluded:

To our knowledge, a parent company has never been excluded from participation in Medicare and Medicaid for illegal activities, which endanger the public health and deplete taxpayer-funded programs. Criminal prosecutions of executives leading companies engaged in these illegal activities have been extremely rare. Much larger penalties and successful prosecutions of company executives that oversee systemic fraud, including jail sentences if appropriate, are necessary to deter future unlawful behavior. Otherwise, these illegal but profitable activities will continue to be part of companies’ business model.

Iatrogenocide Takes Center Stage

Even before COVID, available data indicated that 20th- and 21st-century Western medicine had failed to improve health in any meaningful way, instead trading off the industrial-age diseases of yore for modern chronic disease epidemics, many or most with iatrogenic causes or contributors. Unfortunately, recent events suggest that medicine—forging an unhealthy partnership with government—may now be more dangerous than it has ever been.

Until 2020, the Americans who were most concerned about medical risks and medical criminality belonged to groups already adversely affected, such as those injured by vaccines or opioids. However, with the advent of life-threatening COVID “countermeasures” and lethal protocols in U.S. hospitals and in other countries such as the UK, medical pharmaceutical gangsterism—seemingly occurring with government cognizance—has begun attracting more widespread notice. When governments began parlaying the dubious health “emergency” into an excuse to authorize and mandate the COVID vaccines and boosters—and proceeded full tilt even when unprecedented injuries and deaths immediately began piling up—some segments of the public saw the contours of an officially sanctioned medical crime.

As Holocaust survivor and human rights activist Vera Sharav communicated in her docuseries Never Again Is Now Global, medical coercion and the suspension of constitutional freedoms have never led anywhere good. Unfortunately, history shows that governments intent on “state repression, brutality and genocide” can usually count on the readiness of some doctors to serve as accomplices, even if their complicity has the potential to turn them into “mass murderers on an exponential scale.”

July 5, 2026 Posted by | Book Review, Science and Pseudo-Science, Timeless or most popular | | Comments Off on ‘The Medical-Pharmaceutical Killing Machine: Facing Facts Could Save Your Life’

The Machine

An Essay on the American Vaccine Program from License to Prosecution

Lies are Unbekoming | July 3, 2026

On November 14, 1986, Ronald Reagan signed the National Childhood Vaccine Injury Act into law.¹ The legislation ended more than a decade of tort litigation against vaccine manufacturers by transferring civil liability for injury and death from the companies producing the products to the American taxpayer. The pharmaceutical industry had threatened to leave the childhood vaccine market. Reagan’s signature ensured they would stay, at a price paid by parents who would never be told what had been arranged on their behalf.

Twenty-five years later, in Bruesewitz v. Wyeth, the Supreme Court closed the last remaining exit. The 2011 decision, written by Justice Antonin Scalia, held that federal law preempts all design-defect claims against vaccine manufacturers in state courts.² Justice Sotomayor’s dissent, joined by Justice Ginsburg, identified the practical effect: no federal agency, no state court, no jury of citizens would henceforth ensure that vaccine manufacturers accounted for scientific advances when designing their products. The manufacturers had been placed outside the accountability structure that governs every other industry in the United States.

The 1986 Act and the 2011 ruling together defined the shape of what now exists. Every function of the vaccine program — licensing, recommendation, purchase, safety monitoring, patent holding, research funding, injury adjudication, and courtroom defense — resides in the federal government. When the products kill a child, the state prosecutes the parents.

Leslie Manookian, founder of the Health Freedom Defense Fund, mapped this architecture in a twelve-point summary published to her readers.³ What follows walks through the machine she described, in five stages. Each stage encloses the next. By the fifth, the shape of the trap around the American parent becomes fully visible.

1. The License

The Food and Drug Administration licenses vaccines on the basis of clinical trials that do not use inert placebo controls. This fact is documented in the FDA’s own package inserts and in sworn testimony by the industry’s most senior figures.

In January 2018, attorney Aaron Siri deposed Dr. Stanley Plotkin in New Hope, Pennsylvania — the vaccinologist widely regarded as the industry’s founding figure and co-editor of the standard reference textbook Plotkin’s Vaccines.⁴ Under oath, Siri walked Plotkin through the pre-licensure clinical trials for each product on the recommended childhood schedule. The pattern that emerged was uniform.

The safety review period following each dose was 48 hours for the IPOL polio vaccine. 48 hours for ActHIB. Four days for Engerix-B, the hepatitis B vaccine administered to newborns on their first day of life. Five days for Recombivax HB, the other hepatitis B product. Siri produced, for comparison, the package insert for Enbrel — a drug given to adults with rheumatoid arthritis — and asked Plotkin to confirm that its pre-licensure clinical trials monitored patients for up to 80 months. Plotkin confirmed. A drug given to sick adults was studied for six and a half years. Vaccines given to healthy newborns were studied for 48 hours to five days.

Plotkin then confirmed, product by product, that these trials had no saline placebo control group. Not Recombivax HB. Not Engerix-B. Not IPOL, whose trial subjects received the polio vaccine concurrently with DTP, making it impossible to attribute any reaction to either product. Not ActHIB. The MMR II vaccine, which Plotkin himself was present for the licensure of, had, in his own words, no control group “for the studies that I’m recalling.” When the Hiberix Hib vaccine was later licensed, the manufacturer used ActHIB itself as the “placebo” — testing one Hib vaccine against another.

On the necessity of a saline control, Plotkin was direct: “Without a control group, if you’re looking for a phenomenon occurring in the vaccine group, you cannot judge that phenomenon without having a control group.” That is the industry’s founding figure, testifying under oath, describing the epistemic condition of the products his industry markets.

The pattern in the trials produces a specific consequence. When a new vaccine is tested against an existing licensed vaccine as its control, any injury rate common to both groups becomes invisible. The comparison measures relative difference, not absolute harm. If the existing vaccine produces seizures at a rate of 1 in 500, and the new vaccine produces seizures at a rate of 1 in 500, the trial reports no significant difference — and both products remain on the market.

The Gardasil trial illustrates what happens when a saline group is included but the result is inconvenient. Merck’s pre-licensure clinical trial for its HPV vaccine assigned 9,412 subjects to a “placebo” arm. Of these, only 594 received actual saline. The remaining approximately 8,800 received AAHS — the aluminum-containing adjuvant used in the Gardasil formulation itself. Merck reported the two groups combined, showing 2.3% of the “placebo” arm developing what the trial recorded as systemic autoimmune events, matched by 2.3% in the Gardasil arm. The vaccine was declared safe on the strength of no difference.

Siri produced the underlying trial data. Broken out separately, the saline placebo group of 594 girls and women showed zero such events. The aluminum group showed approximately 2.5%. Merck had recorded the difference and reported the combination.

Plotkin was asked why the two groups had been combined for that analysis when they were broken out separately for local reaction analysis on the preceding pages. His response, verbatim: “So going into the study, they just assumed aluminum wouldn’t cause autoimmunity and so that’s how they proceed in designing it.” A pre-licensure trial for a product administered to schoolgirls declared the vaccine safe by defining the aluminum adjuvant as inert, then combining subjects receiving that adjuvant with subjects receiving nothing.

Once a vaccine reaches the schedule, the failure to test it against saline becomes permanent. For each product Siri walked Plotkin through, he asked whether a proper placebo-controlled study could now be conducted. Plotkin confirmed, product by product, that it could not — running such a trial would be “unethical” in children whose vaccines are already recommended. The absence of a control group at the point of licensure becomes the reason no control group can ever be introduced. The regulatory record is locked at the point of the initial deception.

When a Freedom of Information Act request submitted by the Informed Consent Action Network in 2018 asked the Department of Health and Human Services to produce the biennial vaccine safety reports required by Section 300aa-27 of the 1986 Act, HHS was forced to respond that it had not produced a single such report in the thirty-two years since Reagan signed the law.⁵ The statutory obligation to review safety had been ignored for the entire life of the program.

The FDA license then triggers the second function. The Centers for Disease Control and Prevention convenes the Advisory Committee on Immunization Practices, which votes on whether to add the newly licensed vaccine to the recommended childhood schedule. ACIP members are drawn from the same institutional networks that developed and defended the products. Once added, the vaccine appears on the schedule that is distributed to every state health department in the country. The recommendation is not a mandate. It becomes one at the next stage.

Under oath in the same deposition, Plotkin acknowledged that he had served as medical and scientific director of Sanofi Pasteur in the 1990s, that he operated a personal consulting entity called Vaxconsult, and that he had received payments over the preceding two decades from Merck, GSK, Pfizer, Sanofi, and, in his own phrasing, “essentially all of the major manufacturers.” He had also consulted for the FDA. The industry’s founding figure had confirmed the case against the products his industry markets. He was also paid by every major manufacturer of those products.

Plotkin Under Oath: Nine Hours That Exposed the Vaccine Industry

2. The Mandate

The federal government does not directly mandate childhood vaccines. That function is delegated to the states.

Every state in the union has passed legislation requiring specified vaccines for school attendance. The specific list varies. The mechanism is uniform. Parents who wish to enroll their children in public school — and in many states private school — must produce documentation that their children have received the vaccines on the state’s list. The state list is drawn from the CDC schedule; the CDC schedule from the ACIP recommendation; the ACIP recommendation from the FDA license. The FDA license rests on trials that were never controlled against a genuine placebo.

The chain is complete before the parent enters the pediatrician’s office.

Under the Vaccines for Children program, established in 1993, the federal government purchases half of all childhood vaccines administered in the United States. Recent VFC spending has exceeded $5 billion annually.⁶ The federal government is the largest single purchaser of the products it licenses, the products it recommends, and the products the states mandate.

This creates a market structure without parallel elsewhere in American pharmaceutical policy. The maker of a blood pressure medication faces market discipline. Doctors may prescribe it or not, patients may fill the prescription or not, insurance may cover it or not. The maker of a childhood vaccine faces no equivalent constraint. The state compels administration; the federal government guarantees a buyer; demand is legislated. Revenue is secured before a single dose is delivered.

The mandate has hardened as it has aged. Every state at some point permitted medical, religious, and in some cases philosophical exemptions from the vaccine schedule. Over the past decade, state legislatures have moved to close them. California eliminated its personal belief exemption in 2015 through SB 277 following the Disneyland measles cluster. In 2019, New York eliminated its religious exemption; Maine followed the same year. Connecticut eliminated its religious exemption in 2021. The pattern has been consistent: a highly publicised incident, a legislative response drafted with industry input, and the removal of the exit ramp. The federal government does not need to mandate. The state legislatures have been prevailed upon to do it, and to progressively narrow the terms under which the mandate can be refused.

Leslie Manookian, in the interview she gave me,¹⁹ described the shape of what has been built here. “When we succeed and thrive outside the extant medical paradigm, we pose an existential threat to the medical complex which is why the main actors fight our information, experiences, and independence so fervently.” The compelled purchase is what makes the mandate machinery operate. Without it, the products would compete on their merits. With it, they do not compete at all.

Interview with Leslie Manookian

3. The Shield

The 1986 Act shielded manufacturers from every category of liability that governs other industries. The immunity covered injuries caused by design choices themselves — the composition of the product, the adjuvants used, the decisions about testing. A safer alternative product could exist and the manufacturer could refuse to adopt it, and the injured child’s family could not sue.

Justice Scalia’s opinion in Bruesewitz addressed a case brought by Robalee Bruesewitz on behalf of her daughter Hannah, who had suffered residual seizure disorder and developmental delay after receiving the DPT vaccine manufactured by Wyeth. The Bruesewitz family had exhausted the Vaccine Injury Compensation Program. They then attempted to sue Wyeth in state court, arguing that a safer alternative vaccine design existed and Wyeth had refused to adopt it. The Supreme Court held that federal law preempts such claims. The manufacturer’s choice to continue producing a design that injured children could not be litigated.

Sotomayor’s dissent identified the consequence. Vaccine manufacturers now occupy a regulatory space in which no external mechanism — regulatory agency, court, or jury — holds them accountable for design decisions. This is not an inference. It is a description of the legal structure the majority created.

Behind the shield sits a further conflict. The Department of Health and Human Services — the parent agency of the FDA, the CDC, the National Institutes of Health, and the Health Resources and Services Administration that runs the injury compensation program — holds patents on multiple childhood vaccines. HHS scientists Douglas Lowy and John Schiller developed the recombinant protein technology underlying Merck’s Gardasil and receive royalties on its sale.⁷ Similar patent and royalty arrangements extend to other products in the childhood schedule. The regulator collects revenue on the products it approves.

The research infrastructure that would produce independent safety findings is subject to a parallel capture. Studies funded by the CDC, the NIH, or by the manufacturers themselves consistently produce findings favorable to the schedule. The vaccinated-versus-unvaccinated comparison studies that would settle the fundamental question about long-term outcomes have not been funded. When independent researchers attempt them — Anthony Mawson’s 2017 study of homeschooled populations,⁸ Paul Thomas’s cohort analysis of his own pediatric practice⁹ — the results are attacked, retracted, or ignored, and the researchers face professional consequences.

The capture extends inside the agencies themselves. In August 2014, Dr. William Thompson, a senior epidemiologist at the CDC and co-author of the 2004 DeStefano study widely cited to reject any link between the MMR product and neurodevelopmental injury, submitted a statement through his attorney acknowledging that he and his co-authors had “omitted statistically significant information” from the published paper and had disposed of documents to conceal the omission.¹⁰ The withheld data showed an elevated risk of neurodevelopmental injury among African American boys who received the injection before thirty-six months of age. Thompson’s disclosure was made under whistleblower protection. Congress has never subpoenaed him to testify. The DeStefano paper remains uncorrected.

Merck faced a parallel qui tam action from two of its own virologists, Stephen Krahling and Joan Wlochowski, who alleged in a federal filing that Merck had falsified mumps vaccine efficacy data submitted to the FDA over the course of a decade.¹¹ The case, filed in 2010, moved slowly through the courts. The Department of Justice declined to intervene. Merck retained its exclusive contract to supply mumps vaccine to the U.S. government. The plaintiffs’ allegations of test manipulation entered the public record and produced no regulatory action.

The shield is a network. Liability preemption from Congress protects the manufacturer. Patent revenue aligns the regulator with the products it approves. Captured research funding directs the studies that might identify harm away from the questions that would find it. Judicial preemption then blocks any citizen who attempts to litigate the design decisions the products embody. Each layer supports the others. The whole structure is invisible to the parent standing in a pediatrician’s office being told the shot is safe.

No Liability, No Studies, No Accountability: The Vaccine System Aaron Siri Exposed in Federal Court

4. The Monitor Becomes the Promoter

The Centers for Disease Control and Prevention operates the Vaccine Adverse Event Reporting System. It also runs the promotional campaigns that place vaccination on the pediatric schedule. The agency responsible for detecting harm from the products is the same agency responsible for driving their uptake.

The conflict is not theoretical. Harvard Pilgrim Health Care, under a grant from the Agency for Healthcare Research and Quality within HHS, conducted an internal study of VAERS reporting rates in a Massachusetts patient population between 2007 and 2010. The study found that fewer than 1% of vaccine adverse events were being captured by the reporting system.¹² When the researchers attempted to communicate their findings to the CDC in order to develop improved reporting mechanisms, the agency stopped responding to their emails. The grant ended. The improved reporting system was never built.

The passive reporting infrastructure that captures under 1% of injuries then becomes the basis for the CDC’s public assurances that adverse events are rare.

The injury table itself has been subject to steady contraction. When the Vaccine Injury Compensation Program began in 1988, the injury table included a broader range of conditions presumed to be caused by vaccination, with corresponding timelines within which onset would qualify a case for compensation.¹³ Over the following decades, categories were removed or narrowed. Sudden Infant Death Syndrome, initially compensable when it followed vaccination within a specified window, was removed. Neurodevelopmental injury, briefly acknowledged as a category during the 1990s when concerns about the MMR product and other injections emerged, was removed. The seizure timelines were narrowed. Encephalopathy definitions were tightened.

The 1995 amendment illustrates the pattern. Residual seizure disorder — a category under which many families of children who had suffered seizures after DPT vaccination had successfully claimed compensation — was removed. Encephalopathy criteria were revised in ways that made the diagnosis nearly impossible to satisfy. The Advisory Commission on Childhood Vaccines, which recommended the changes, drew a majority of its membership from the same medical-institutional networks that administered and defended the vaccine schedule. Petitioners whose cases had been filed under the earlier table found themselves adjudicated under the new one. Cases that would have succeeded were denied.

Each removal reduced the number of compensable claims. The fund benefited. So did the manufacturers whose products would otherwise be more clearly implicated in the injury pattern.

The Institute of Medicine, tasked periodically with reviewing whether specific vaccines cause specific injuries, has repeatedly concluded that the evidence is insufficient to accept or reject a causal relationship for a majority of the injury-outcome pairs it examines.¹⁴ This finding — insufficient evidence — is then used in the injury compensation courtroom to deny claims. The absence of evidence functions as evidence of absence, produced by the very research infrastructure that would have to fund the studies to end the insufficiency.

The industry’s founding figure confirmed the position under oath in the same deposition. Asked directly whether he could make the scientific statement that childhood vaccines do not cause autism, Plotkin answered: “As a scientist, I would say that I do not have evidence one way or the other.” The IOM had found no study establishing that the DTaP or Tdap products do not cause autism. Plotkin acknowledged that no such study existed and that he personally held no evidence to support the claim his industry has spent three decades making.

The parent whose child seized within twelve hours of vaccination, developed encephalopathy, and never recovered enters a system that was prepared for her arrival. The injury table’s timeline for seizure onset has been shortened past the point where her child’s case qualifies. The IOM has declared the evidence insufficient. VAERS captured her report and did nothing with it. The monitor was never separate from the promoter.

5. The Court and the Blame

The Vaccine Injury Compensation Program is administered by the U.S. Court of Federal Claims. It is not a court in the ordinary sense. The proceedings involve no juries, no meaningful discovery, and no Article III judges — no judges appointed for life under the constitutional protections designed to insulate the judiciary from executive influence.

Cases are heard by “Special Masters,” Article I officers appointed by the Chief Judge of the Court of Federal Claims to seven-year terms. The Special Masters are drawn from a pool of attorneys with prior government experience. The Department of Justice provides the attorneys who defend against injury claims. HRSA administers the fund. The petitioner’s attorneys are paid from the same fund out of which awards are made.

Every party in the courtroom — the judge, the government’s defense attorneys, the fund itself, and the petitioner’s legal counsel — is paid by the federal government. The injured child’s family stands before a tribunal in which no independent party has an interest in a finding of injury.

The statistics reflect the structure. The majority of petitions filed with the VICP have been dismissed rather than compensated over the life of the program.¹⁵ Of the cases that succeed, the majority are settled rather than adjudicated on the merits, with no admission that the vaccine caused the injury. The compensation cap for a vaccine-caused death — $250,000 — has not been raised since the statute was passed in 1986.

The excise tax that funds the program is $0.75 per antigen per dose. The fund now holds over $4 billion.¹⁶ The families whose children were injured cannot access it through the ordinary legal system because the ordinary legal system has been closed to them.

This is the structure Leslie Manookian described in her twelve-point summary. Her exact phrasing on the final function is worth returning to: “So, parents who’ve already suffered an unimaginable tragedy are up against a govt court staffed by govt paid special masters and attorneys with no due process defending a govt licensed and govt mandated product for which they blame the victims for harm.”

The final phrase — “they blame the victims for harm” — describes the twelfth function of the machine. When a child collapses after vaccination with the sudden onset of retinal hemorrhages, subdural hematoma, and cerebral edema — the triad — the diagnosis assigned in emergency departments and coroner’s offices is “shaken baby syndrome” or its rebranded successor, “abusive head trauma.” The triad is presumed diagnostic of parental abuse. The parents are arrested.

The vaccine reaction that produces the identical triad — through encephalopathy, elevated intracranial pressure, and hemorrhagic events following injection — is not considered in the differential diagnosis.¹⁷ The diagnostic criteria for “shaken baby syndrome” were developed without accounting for it. The emergency physician, the coroner, and the child protective services investigator have all been trained within an institutional framework in which vaccine injury of this magnitude does not exist.

Alan Yurko’s ten-week-old son died in November 1997 shortly after receiving a round of childhood vaccinations. Yurko was convicted of first-degree murder in 1999 on the basis of the triad diagnosis and sentenced to life plus ten years in Florida state prison. He was released in 2004 after independent medical review of the case demonstrated that the shaking diagnosis could not be sustained and post-conviction proceedings established alternative medical explanations for the child’s injuries.¹⁸ Yurko is one documented case. There are others. The precise number is unknown because the diagnostic framework prevents the question from being asked.

A parent whose child dies after vaccination faces a compound structure. The vaccine that caused the death is licensed by the federal government, recommended by the federal government, purchased by the federal government, and defended in the injury court by the federal government. The manufacturer is shielded from civil liability by federal statute and Supreme Court precedent. The injury table does not recognize the death as vaccine-caused. The state, meanwhile, has assigned the triad diagnosis and turned the case over to the district attorney. The parent must now prove — in a criminal court, against the state — that the child was not shaken.

The Vaccine Court (2014)

The Position

Robalee Bruesewitz spent nearly two decades in litigation on behalf of her daughter. The Supreme Court’s ruling denied her family relief and closed the door behind them for every family that would come after. The 1986 Act had shifted liability from the manufacturer to the taxpayer. Bruesewitz confirmed that the shift was permanent and that no design decision made by the manufacturer could be challenged in any court open to ordinary Americans.

This is the position in which the American parent now stands. Her child’s pediatric visit will produce a recommendation to administer products licensed on the basis of trials that were never controlled against saline. The state will require their administration for school attendance. When injury results, over 99% of adverse events never reach VAERS at all, and the reports that do reach it change nothing. A family that attempts compensation will petition a court in which every party is paid by the federal government to defend the products or administer the fund. And when death occurs with the triad present, the emergency department’s diagnostic framework will not include vaccine reaction in the differential, and the parent enters the criminal jurisdiction as the presumed cause of the child’s death.

There is no exemption from this structure that carries no cost. State legislatures have progressively narrowed medical and religious exemptions; declining vaccines removes a child from school; injury bars a family from ordinary civil courts. And when death is accompanied by the triad, the state prosecutes the parent for the death.

Leslie Manookian described this arrangement, at the close of her twelve-point post, as “crony capitalism at best and pure evil fascism at worst.” The characterization is precise. A private industry produces the product. The state compels its administration, indemnifies the manufacturer against claims of harm, and prosecutes the parent when the harm arrives.

The machine’s design serves the flow of money and the concentration of power. Every safeguard the ordinary citizen might rely on — informed consent, product liability, judicial review, jury trial, prosecutorial restraint — has been removed at the point where the childhood vaccine schedule intersects with the American family. The parent who accepts the recommendation and whose child is injured has no meaningful path to redress. Refusal costs school access. Death with the triad opens the parent to criminal prosecution for a killing they did not commit.

This is the environment in which every American child is now born. The machine was assembled piece by piece across four decades, ratified by every institution that could have prevented it, and defended by the same institutions today. What Leslie Manookian named as crony capitalism at best and fascism at worst describes a working system, operating as designed, in a country that once organised its politics around the presumption that no such system could be permitted to form.

For a Six-Year-Old

There is a big company that makes shots.

The government helps the company make the shots and sell them. The government tells your school that you have to get the shots before you can come to school.

Nobody checks the shots very well. The people who are supposed to check work with the company. So the shots go out into the world before anyone really knows if they are safe.

When a child is hurt by a shot, the family cannot go to a normal judge. There is a special room where a different kind of judge decides. That judge is paid by the government. The lawyers on the other side are paid by the government. The government made the shot rules. The government bought the shots. And the government decides whether the shot hurt you.

Most families are told the shot did not hurt their child, even when it did.

When a shot makes a baby die, the doctors sometimes think the mother or father shook the baby. The parents can be arrested. They can go to prison. For what the shot did.

The company that made the shot never gets in trouble. The company keeps making the shots. Your school keeps requiring them. The next family goes through the same door.

That is the machine.


References

¹ National Childhood Vaccine Injury Act of 1986, Public Law 99-660, 42 U.S.C. § 300aa-1 et seq.

² Bruesewitz v. Wyeth LLC, 562 U.S. 223 (2011).

³ Leslie Manookian, twelve-point summary post, X (@LeslieManookian), July 3, 2026, status/2072712451800625369.

⁴ Deposition of Stanley A. Plotkin, M.D., taken January 11, 2018, in Matheson v. Schmitt, State of Michigan, Circuit Court for the County of Oakland, Family Division, Case No. 2015-831539-DM; transcript published via Informed Consent Action Network.

⁵ ICAN v. HHS, correspondence dated July 9, 2018, in response to FOIA request; HHS acknowledged no biennial reports produced under 42 U.S.C. § 300aa-27(c).

⁶ Vaccines for Children Program expenditure data, Centers for Disease Control and Prevention; annual VFC purchasing figures.

⁷ U.S. Patents 5,437,951 and related — Lowy, Schiller et al., “Self-Assembling Recombinant Papillomavirus Capsid Proteins,” assigned to the United States Department of Health and Human Services; licensed to Merck & Co. for Gardasil.

⁸ Mawson AR et al., “Pilot comparative study on the health of vaccinated and unvaccinated 6- to 12-year-old U.S. children,” Journal of Translational Science, 2017.

⁹ Thomas JL, Lyons-Weiler J, “Relative Incidence of Office Visits and Cumulative Rates of Billed Diagnoses Along the Axis of Vaccination,” International Journal of Environmental Research and Public Health, 2020.

¹⁰ Statement of William W. Thompson, Ph.D., through counsel Rick Morgan, August 27, 2014; documentation regarding DeStefano DA et al., “Age at first measles-mumps-rubella vaccination in children with autism and school-matched control subjects: a population-based study in metropolitan Atlanta,” Pediatrics, 2004.

¹¹ United States ex rel. Krahling and Wlochowski v. Merck & Co., Inc., No. 2:10-cv-04374, U.S. District Court for the Eastern District of Pennsylvania, complaint filed 2010.

¹² Lazarus R et al., “Electronic Support for Public Health–Vaccine Adverse Event Reporting System (ESP:VAERS),” Grant Final Report, Harvard Pilgrim Health Care, Inc., 2011 (AHRQ Grant ID R18 HS 017045).

¹³ Vaccine Injury Table history, Health Resources and Services Administration; successive amendments to 42 C.F.R. § 100.3.

¹⁴ Institute of Medicine (now the National Academy of Medicine), Adverse Effects of Vaccines: Evidence and Causality (2011) and predecessor reports.

¹⁵ Health Resources and Services Administration, VICP claim adjudication statistics.

¹⁶ Vaccine Injury Compensation Trust Fund monthly balance report, U.S. Department of the Treasury.

¹⁷ Michael Innis, “Vaccines, Apparent Life-Threatening Events, Barlow’s Disease, and Questions about ‘Shaken Baby Syndrome,’” Journal of American Physicians and Surgeons, 2006; Harold Buttram and Alan R. Yurko, “Shaken Baby Syndrome or Vaccine-Induced Encephalitis?” Medical Sentinel, subsequent case documentation.

¹⁸ State of Florida v. Alan R. Yurko, Ninth Judicial Circuit, 1999; post-conviction proceedings and release 2004; contemporaneous medical review including Harold E. Buttram, M.D.

¹⁹ Unbekoming, “Interview with Leslie Manookian, Health Freedom Defense Fund,” Lies are Unbekoming, Substack, April 13, 2024.

July 4, 2026 Posted by | Corruption, Deception, Progressive Hypocrite, Science and Pseudo-Science, Timeless or most popular | Comments Off on The Machine

IS YOUR SUNSCREEN CAUSING CANCER?

The HighWire with Del Bigtree | June 25, 2026

The FDA began reevaluating sunscreen chemical safety in 2019, yet millions are still told to apply these products every day. Jefferey looks at what happens when sunscreen chemicals enter the bloodstream, and why new research on sun exposure, vitamin D, and cancer is challenging old assumptions.

June 27, 2026 Posted by | Science and Pseudo-Science, Timeless or most popular, Video | Comments Off on IS YOUR SUNSCREEN CAUSING CANCER?

Vaccines are not a panacea

The shingles vaccine has not cured dementia

By Dr Clare Craig | Health Ethics Advocacy and Resarch Team | June 25, 2026

Over the past month, four studies have been read as showing that the shingles vaccine prevents, or even reverses, dementia. A national newspaper has turned that reading into advice. The advice is to go and get the vaccine now. The evidence tells a different story.

Vinay Prasad has already gone through the four papers and laid the timings side by side, and I will not improve on that account.

●       The Annals paper has a 5% absolute reduction in dementia by three years, with benefit appearing within a year of a nursing-home admission.

●       The JAMA paper has it working by a hundred days.

●       The Welsh study in Nature is a natural experiment, not a simple cohort, and reports a fifth fewer dementia diagnoses over seven years. It declines to publish a time-to-event plot at all.

●       The American paper, in Nature Medicine, includes a hazard function whose curves separate almost immediately.

●       And a meta-analysis concludes that it is not the shingles vaccine alone but every adult vaccine that appears to protect against dementia.

What a dementia diagnosis actually is

A dementia code in a health record is the end of a chain of soft decisions:

●       someone has to notice a change,

●       the patient has to present to the system,

●       a threshold has to be applied,

●       and a clinician has to enter the code.

For each of these steps there is wide variation in the timing between patients. The noticing depends on who is being watched closely and who is not. The same forgetful elderly person can be coded, or not coded, according to how hard anyone happens to be looking. Look harder and you might diagnose more.

Now ask who receives an elective shingles vaccine. It is, overwhelmingly, the people well enough to go and get one. The housebound, the frail, and above all those whose memory is already failing, the people nearest to a diagnosis, are the least likely to present for a non-urgent injection. So the unvaccinated group is enriched, at the very outset and before anyone is followed up, with exactly the people who will later be coded with dementia. This is the “healthy vaccinee effect”, and in this setting it is large enough to produce the whole of the apparent benefit on its own.

Why four studies agreeing is not reassurance

The natural objection is that four datasets agree, and agreement is supposed to be reassuring. But when the cohort studies make the same methodological error it is not reassuring. Every one of them carries the same confounding structure: in each, vaccination marks underlying health, healthcare contact and the simple capacity to turn up, and in each that marker is correlated with the outcome being measured. The errors are not independent results that happen to coincide. They are the same error made several times, because the same bias is built into each design. A biased estimate does not become unbiased by being repeated. These confounded studies in agreement tell you that the confounder is stable, not that the result is real.

Prasad puts this as the earth looking flat whichever way you turn, which is exactly right as an image of the confounded cohorts. The formal version is that replication adds evidence only when the replications are capable of failing in different ways. When they share their confounders, consistency is not corroboration, it is the same confounder doing the same job in each dataset in turn. To break the symmetry you do not add a fifth observational cohort, another flat horizon. You change the design to one whose errors are structured differently. That test already exists.

A regression-discontinuity study exploits the age cut-off for eligibility: people just below and just above the threshold are, for practical purposes, exchangeable, so the baseline-health gap that drives the observational studies is largely removed. The Welsh study is exactly this design, which is why it is the strongest evidence on the table; they claimed dementia was prevented in Wales and in Australia and Ontario. But would an independent team running a study with an identical design find the same thing? George Davey Smith and colleagues have now carried out such a study in England, across some 6.3 million people, roughly twenty times the Welsh sample; the work was presented in June 2026 at a conference and is not yet published. The design worked, in that eligibility produced the expected fall in shingles (although it is not clear what happened in the ‘grace period’ immediately after vaccination) but on dementia it found nothing. The fact that the finding was null in the far larger study should mean the smaller one has been overruled.

The speed matters

Speed is being sold as a measure of how powerful the vaccine is. It is better understood as evidence that the vaccine is doing nothing at all. Dementia is slow. The neuropathology accumulates over years, usually decades, and by the time anyone is coded with the disease the process is long established. Nothing done to a brain can produce a measurable difference in dementia incidence within a hundred days, because the disease does not move on that timescale. So when the curves separate almost immediately, the separation cannot be the vaccine acting. It can only be that the two groups already differed at the moment the vaccine was given.

It is a signature of selection. The rapidity that is being offered as the headline result is the clearest single indicator that what is on the page is the “healthy vaccinee” artefact and not a treatment effect.

From the literature to the newsstand

On 10 June the Daily Telegraph published an interview with John Todd, professor of precision medicine at Oxford, in which he told readers over fifty to get the shingles vaccine now. Jonathan Engler has lodged a complaint with the MHRA about that article.

Professor Todd is, on his own declaration, a paid consultant for GSK, which manufactures Shingrix, and the study he relies on is one he co-authored. When the paper was published, given the issues described above about selection bias the authors wrote that the dementia results “provide a rationale for conducting a randomized control trial aiming to confirm the findings”. Shingrix is not licensed for the prevention of dementia. The advice to buy it for that purpose rests on a paper whose findings, in its authors’ own words, have not been confirmed.

In the United Kingdom, the advertising of prescription-only medicines is governed by Part 14 of the Human Medicines Regulations 2012. Regulation 284 provides that a person may not publish an advertisement likely to lead to the use of a prescription-only medicine. There is proper latitude for journalism, and the MHRA’s guidance for journalists asks only that coverage be balanced, that it not exaggerate benefit, and that it not steer readers towards a named product. It does not require proof of a commercial arrangement before a breach is made out. Engler’s point, which is the right one, is that a piece does not stop being promotional by declining to call itself promotional, and that the absence of the label is itself the difficulty. Where a consultant to the manufacturer urges the purchase of that manufacturer’s unlicensed product, on the strength of his own unconfirmed research, with the consultancy unmentioned, the ordinary description of the result is advertising, whatever else it is also called.

Telegraph readers thinking of taking his advice to rush off and buy a private Shingrix vaccine, might want to take note that (a) Professor Todd has no medical qualification (his FRCP is honorary and his primary qualification is in biochemistry) and (b) the department he heads in Oxford was funded by GSK to the tune of £30 million.

What honest curiosity would do next

The editors of the observational papers could be asked to publish the all-cause mortality curves alongside the dementia curves: if the vaccinated are simply healthier people, their mortality will diverge in the same direction, and the confounding will be visible on the page. This is a control test. Every child at school is taught the importance of using a control group in scientific research but all too often they are left out by actual scientists. The randomised trial that the NIH is said to be considering might need to be run simply to put to bed the excitement around this issue that has been fuelled by media speculation.

The truth is that the studies that kept producing the same answer did so because they shared the same bias, while the largest independent test found nothing and a national newspaper has printed a paid consultant’s promotion of a drug when he will benefit from the sales. Perhaps neither of those stories will sell as many newspapers as the ones that do get printed or clicked on.

June 27, 2026 Posted by | Fake News, Mainstream Media, Warmongering, Science and Pseudo-Science, Timeless or most popular | Comments Off on Vaccines are not a panacea